What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Apoptosis
Kaikobad Irani mainly focuses on Cell biology, Internal medicine, Endothelial stem cell, Molecular biology and Endothelium.
Cell biology is closely attributed to Oxidative stress in his research.
His study ties his expertise on Endocrinology together with the subject of Internal medicine.
His Endocrinology research incorporates themes from Downregulation and upregulation, Angiogenesis, microRNA and Small interfering RNA.
His work carried out in the field of Molecular biology brings together such families of science as Homocysteine, Epigenetics, Methylation and CpG site.
The study incorporates disciplines such as Histone methyltransferase, Nitric oxide, EZH2 and Endothelial dysfunction in addition to Endothelium.
His most cited work include:
- MicroRNA miR-27b Rescues Bone Marrow–Derived Angiogenic Cell Function and Accelerates Wound Healing in Type 2 Diabetes Mellitus (86 citations)
- SUPEROXIDE-MEDIATED ACTIN RESPONSE IN POST-HYPOXIC ENDOTHELIAL CELLS (81 citations)
- Homocysteine promotes human endothelial cell dysfunction via site-specific epigenetic regulation of p66shc (75 citations)
What are the main themes of his work throughout his whole career to date?
Kaikobad Irani mostly deals with Cell biology, Internal medicine, Endothelial dysfunction, Endocrinology and Oxidative stress.
His Cell biology study integrates concerns from other disciplines, such as Endothelial stem cell and Molecular biology.
His work deals with themes such as Endothelium, Downregulation and upregulation, microRNA and Cancer research, which intersect with Endothelial dysfunction.
His Endothelium research incorporates elements of Enos and Vasodilation.
His Endocrinology study deals with Patch clamp intersecting with Stimulation, Membrane potential and Cardiomyopathy.
His research in Oxidative stress intersects with topics in Reactive oxygen species, Oxidative phosphorylation, Immunology and Vascular function.
He most often published in these fields:
- Cell biology (46.25%)
- Internal medicine (30.00%)
- Endothelial dysfunction (27.50%)
What were the highlights of his more recent work (between 2017-2020)?
- Cell biology (46.25%)
- Oxidative stress (27.50%)
- Function (6.25%)
In recent papers he was focusing on the following fields of study:
Cell biology, Oxidative stress, Function, Cardiac action potential and microRNA are his primary areas of study.
Mitochondrial ROS is the focus of his Cell biology research.
Kaikobad Irani has researched microRNA in several fields, including Cardiomyopathy, Heart failure, Sudden cardiac death and Endothelial dysfunction.
The concepts of his Endothelial dysfunction study are interwoven with issues in Diabetes mellitus, Cancer research, Endothelium and Aorta.
His studies deal with areas such as CHOP and Internal medicine as well as Diabetes mellitus.
His Tumor necrosis factor alpha research focuses on Endothelial stem cell and how it relates to Downregulation and upregulation.
Between 2017 and 2020, his most popular works were:
- A common variant alters SCN5A–miR-24 interaction and associates with heart failure mortality (17 citations)
- CR6 interacting factor 1 deficiency promotes endothelial inflammation by SIRT1 downregulation. (10 citations)
- Isocitrate dehydrogenase 2 deficiency induces endothelial inflammation via p66sh-mediated mitochondrial oxidative stress. (9 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Apoptosis
His scientific interests lie mostly in Cell biology, Mitochondrial ROS, Tumor necrosis factor alpha, SNP and Genetics.
His research in Cell biology is mostly focused on Reactive oxygen species.
His Mitochondrial ROS study combines topics from a wide range of disciplines, such as Proinflammatory cytokine, Inflammation and Downregulation and upregulation, Sirtuin 1.
His Tumor necrosis factor alpha study typically links adjacent topics like Endothelial stem cell.
His SNP research includes a combination of various areas of study, such as Heart failure, Cardiomyopathy, microRNA and Cardiac action potential.
His work on Genetics is being expanded to include thematically relevant topics such as Sudden cardiac death.
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