His scientific interests lie mostly in Candida albicans, Corpus albicans, Gene, Microbiology and Genetics. His studies in Candida albicans integrate themes in fields like Gene expression, Transcription factor, Efflux, Agar and Drug resistance. In his study, Aequorea victoria is strongly linked to Regulation of gene expression, which falls under the umbrella field of Corpus albicans.
His work in the fields of Mutant, Mitotic crossover and Fungal protein overlaps with other areas such as Site-specific recombination. As a part of the same scientific study, he usually deals with the Microbiology, concentrating on Virulence and frequently concerns with Gene family. His study on Genome, Phenotype and Saccharomyces cerevisiae is often connected to Comparative genomics as part of broader study in Genetics.
Joachim Morschhäuser mostly deals with Candida albicans, Microbiology, Corpus albicans, Gene and Genetics. His Candida albicans study integrates concerns from other disciplines, such as Transcription factor, Mutant, Fluconazole, Efflux and Drug resistance. His Mutant study deals with Mutation intersecting with Downregulation and upregulation.
His work on Microbiology is being expanded to include thematically relevant topics such as Promoter. His Corpus albicans research includes themes of Phenotype and ATP-binding cassette transporter. In his study, Reporter gene and URA3 is inextricably linked to Molecular biology, which falls within the broad field of Gene.
Joachim Morschhäuser spends much of his time researching Candida albicans, Corpus albicans, Microbiology, Gene and Transcription factor. Joachim Morschhäuser combines subjects such as Fluconazole, Mutant, Efflux, Cell biology and Drug resistance with his study of Candida albicans. His Corpus albicans course of study focuses on Regulation of gene expression and Kinase.
His research in Microbiology intersects with topics in Epithelial Damage and Biofilm. His primary area of study in Gene is in the field of Allele. His work investigates the relationship between Transcription factor and topics such as Saccharomyces cerevisiae that intersect with problems in Proline, Chromatin immunoprecipitation, Urate oxidase and Catabolism.
Candida albicans, Corpus albicans, Microbiology, Drug resistance and Gene are his primary areas of study. Joachim Morschhäuser interconnects Efflux, Fluconazole, Mutant and Transcription factor in the investigation of issues within Candida albicans. His Fluconazole research incorporates elements of Gene duplication, Genetics, Gene expression and Loss of heterozygosity.
His Transcription factor research is multidisciplinary, relying on both Molecular biology, P-glycoprotein and Downregulation and upregulation. His Corpus albicans study combines topics in areas such as Regulation of gene expression and Chromosomal translocation. The study incorporates disciplines such as Secretion, Transcellular and Epithelial Damage in addition to Microbiology.
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The SAT1 flipper, an optimized tool for gene disruption in Candida albicans.
Oliver Reuss;Ashild Vik;Roberto Kolter;Joachim Morschhäuser.
Multiple Molecular Mechanisms Contribute to a Stepwise Development of Fluconazole Resistance in Clinical Candida albicans Strains
Renate Franz;Steven L. Kelly;David C. Lamb;Diane E. Kelly.
Antimicrobial Agents and Chemotherapy (1998)
A Mutation in Tac1p, a Transcription Factor Regulating CDR1 and CDR2, Is Coupled With Loss of Heterozygosity at Chromosome 5 to Mediate Antifungal Resistance in Candida albicans
Alix T. Coste;Vincent Turner;Françoise Ischer;Joachim Morschhäuser.
A Human-Curated Annotation of the Candida albicans Genome
Burkhard R Braun;Marco van het Hoog;Christophe d'Enfert;Mikhail Martchenko.
PLOS Genetics (2005)
Regulation of multidrug resistance in pathogenic fungi
Fungal Genetics and Biology (2010)
The transcription factor Mrr1p controls expression of the MDR1 efflux pump and mediates multidrug resistance in Candida albicans.
Joachim Morschhäuser;Katherine S Barker;Teresa T Liu;Julia Blaß-Warmuth.
PLOS Pathogens (2007)
Mutations in the multi-drug resistance regulator MRR1, followed by loss of heterozygosity, are the main cause of MDR1 overexpression in fluconazole-resistant Candida albicans strains.
Nico Dunkel;Julia Blaß;P. David Rogers;Joachim Morschhäuser.
Molecular Microbiology (2008)
The genetic basis of fluconazole resistance development in Candida albicans.
Biochimica et Biophysica Acta (2002)
A gain-of-function mutation in the transcription factor Upc2p causes upregulation of ergosterol biosynthesis genes and increased fluconazole resistance in a clinical Candida albicans isolate.
Nico Dunkel;Teresa T. Liu;Katherine S. Barker;Ramin Homayouni.
Eukaryotic Cell (2008)
Tetracycline-Inducible Gene Expression and Gene Deletion in Candida albicans
Yang-Nim Park;Joachim Morschhäuser.
Eukaryotic Cell (2005)
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