D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 77 Citations 17,084 330 World Ranking 13590 National Ranking 7049

Overview

What is she best known for?

The fields of study she is best known for:

  • Internal medicine
  • Gene
  • World War II

Her scientific interests lie mostly in Platelet, Immunology, Internal medicine, Platelet activation and Nitric oxide. Her biological study spans a wide range of topics, including Thrombus, Biochemistry, Pharmacology and Cell biology. The concepts of her Immunology study are interwoven with issues in Hemostasis, Receptor and Framingham Risk Score.

Jane E. Freedman has researched Internal medicine in several fields, including Endocrinology and Cardiology. Within one scientific family, Jane E. Freedman focuses on topics pertaining to Coagulation under Platelet activation, and may sometimes address concerns connected to Flow cytometry, Inner mitochondrial membrane and Apoptosis. Jane E. Freedman interconnects Endothelium, Superoxide and P-selectin in the investigation of issues within Nitric oxide.

Her most cited work include:

  • Homocyst(e)ine Decreases Bioavailable Nitric Oxide by a Mechanism Involving Glutathione Peroxidase (613 citations)
  • Select Flavonoids and Whole Juice From Purple Grapes Inhibit Platelet Function and Enhance Nitric Oxide Release (404 citations)
  • Nitric oxide released from activated platelets inhibits platelet recruitment. (345 citations)

What are the main themes of her work throughout her whole career to date?

Jane E. Freedman mainly investigates Platelet, Internal medicine, Immunology, Platelet activation and Endocrinology. Her Platelet research is multidisciplinary, incorporating perspectives in Thrombosis, Biochemistry, Nitric oxide and Cell biology. In her study, Antioxidant is inextricably linked to Pharmacology, which falls within the broad field of Nitric oxide.

Her work deals with themes such as microRNA and Cardiology, which intersect with Internal medicine. Her Immunology research is multidisciplinary, incorporating elements of Hemostasis and Receptor. Her Platelet activation research incorporates themes from Thrombus and Coagulation.

She most often published in these fields:

  • Platelet (38.39%)
  • Internal medicine (29.34%)
  • Immunology (22.49%)

What were the highlights of her more recent work (between 2014-2021)?

  • Internal medicine (29.34%)
  • microRNA (10.76%)
  • Platelet (38.39%)

In recent papers she was focusing on the following fields of study:

Jane E. Freedman mostly deals with Internal medicine, microRNA, Platelet, Bioinformatics and Cardiology. The study incorporates disciplines such as Endocrinology and Oncology in addition to Internal medicine. Her study looks at the relationship between microRNA and topics such as RNA, which overlap with Computational biology.

Her Platelet research is under the purview of Immunology. Jane E. Freedman usually deals with Immunology and limits it to topics linked to Disease and Cancer. Her Cardiology course of study focuses on Framingham Risk Score and Cohort study.

Between 2014 and 2021, her most popular works were:

  • Circulating Extracellular Vesicles in Human Disease (213 citations)
  • Circulating Platelets as Mediators of Immunity, Inflammation, and Thrombosis. (170 citations)
  • Diverse human extracellular RNAs are widely detected in human plasma (135 citations)

In her most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • World War II

Jane E. Freedman mostly deals with microRNA, Internal medicine, Genetics, Bioinformatics and Platelet. The various areas that she examines in her microRNA study include RNA and Gene expression. Jane E. Freedman has included themes like Endocrinology and Cardiology in her Internal medicine study.

Her research in Bioinformatics intersects with topics in Extracellular, Extracellular RNA, Weight loss and Pathology. Her Platelet study is associated with Immunology. Her Immunology study combines topics in areas such as Receptor and Disease.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Homocyst(e)ine decreases bioavailable nitric oxide by a mechanism involving glutathione peroxidase

Gilbert R. Upchurch;George N. Welch;Attila J. Fabian;Jane E. Freedman.
Journal of Biological Chemistry (1997)

899 Citations

Select Flavonoids and Whole Juice From Purple Grapes Inhibit Platelet Function and Enhance Nitric Oxide Release

Jane E. Freedman;Crawford Parker;Liqing Li;Jacob A. Perlman.
Circulation (2001)

619 Citations

Nitric oxide released from activated platelets inhibits platelet recruitment.

J E Freedman;J Loscalzo;M R Barnard;C Alpert.
Journal of Clinical Investigation (1997)

475 Citations

Circulating Platelets as Mediators of Immunity, Inflammation, and Thrombosis.

Milka Koupenova;Lauren Clancy;Heather A. Corkrey;Jane E. Freedman.
Circulation Research (2018)

438 Citations

Circulating Extracellular Vesicles in Human Disease

Ravi Shah;Tushar C Patel;Jane E. Freedman.
The New England Journal of Medicine (2018)

421 Citations

α-Tocopherol Inhibits Aggregation of Human Platelets by a Protein Kinase C–Dependent Mechanism

Jane E. Freedman;John H. Farhat;Joseph Loscalzo;John F. Keaney.
Circulation (1996)

391 Citations

Oxidative stress and platelets.

Jane E. Freedman.
Arteriosclerosis, Thrombosis, and Vascular Biology (2008)

359 Citations

Platelet–Monocyte Aggregates Bridging Thrombosis and Inflammation

Jane E. Freedman;Joseph Loscalzo.
Circulation (2002)

307 Citations

Deficient platelet-derived nitric oxide and enhanced hemostasis in mice lacking the NOSIII gene.

Jane E. Freedman;Ryan Sauter;Elisabeth M. Battinelli;Kenneth Ault.
Circulation Research (1999)

305 Citations

Aspirin resistance and atherothrombotic disease.

Peter J. Mason;Alice K. Jacobs;Jane E. Freedman.
Journal of the American College of Cardiology (2005)

300 Citations

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