James J. Lah

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Biology and Biochemistry D-index 75 Citations 19,793 248 World Ranking 3333 National Ranking 1715

Overview

What are the main themes of his work throughout his whole career to date

His Disease study frequently draws connections between related disciplines such as Neurodegeneration. He conducted interdisciplinary study in his works that combined Alzheimer's disease and Donepezil. While working in this field, he studies both Donepezil and Dementia. His study brings together the fields of Cognitive decline and Dementia. His Neuroscience study frequently draws parallels with other fields, such as Cortex (anatomy). His Cortex (anatomy) study frequently links to other fields, such as Neuroscience. His study deals with a combination of Pathology and Physical medicine and rehabilitation. James J. Lah conducted interdisciplinary study in his works that combined Physical medicine and rehabilitation and Pathology. His Internal medicine study frequently draws parallels with other fields, such as Pathological.

James J. Lah most often published in these fields:

Disease (88.89%)
Alzheimer's disease (66.67%)
Neuroscience (55.56%)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Variant of TREM2 associated with the risk of Alzheimer's disease

Thorlakur Jonsson;Hreinn Stefansson;Stacy Steinberg;Ingileif Jonsdottir.
The New England Journal of Medicine (2013)

2183 Citations

Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.

Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)

1885 Citations

Early role of vascular dysregulation on late-onset Alzheimer’s disease based on multifactorial data-driven analysis

Y. Iturria-Medina;R. C. Sotero;P. J. Toussaint;J. M. Mateos-Pérez.
Nature Communications (2016)

720 Citations

Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease

Rebecca Sims;Sven J. Van Der Lee;Adam C. Naj;Céline Bellenguez;Céline Bellenguez.
Nature Genetics (2017)

683 Citations

Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions

Vivianna M. Van Deerlin;Patrick M A Sleiman;Maria Martinez-Lage;Maria Martinez-Lage;Alice Chen-Plotkin.
Nature Genetics (2010)

484 Citations

Large-scale proteomic analysis of Alzheimer’s disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation

Erik C.B. Johnson;Eric B. Dammer;Duc M. Duong;Lingyan Ping.
Nature Medicine (2020)

350 Citations

Common genetic variants in the CLDN2 and PRSS1-PRSS2 loci alter risk for alcohol-related and sporadic pancreatitis

David C. Whitcomb;Jessica LaRusch;Alyssa M. Krasinskas;Lambertus Klei.
Nature Genetics (2012)

350 Citations

Loss of apolipoprotein E receptor LR11 in Alzheimer disease.

Clemens R. Scherzer;Katrin Offe;Marla Gearing;Howard D. Rees.
JAMA Neurology (2004)

338 Citations

The lipoprotein receptor LR11 regulates amyloid beta production and amyloid precursor protein traffic in endosomal compartments.

Katrin Offe;Sara E. Dodson;James T. Shoemaker;Jason J. Fritz.
The Journal of Neuroscience (2006)

306 Citations

Loss-of-function variants in ABCA7 confer risk of Alzheimer's disease

Stacy Steinberg;Hreinn Stefansson;Thorlakur Jonsson;Hrefna Johannsdottir.
Nature Genetics (2015)

298 Citations

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Frequent Co-Authors

External Links

Personal Website for James J. Lah