His primary scientific interests are in Cell biology, Biochemistry, Disease, Ubiquitin and Proteome. His Cell biology research also works with subjects such as
His work in Disease addresses issues such as Transcriptome, which are connected to fields such as Exome sequencing. His work on Ubiquitin-conjugating enzyme and Ubiquitin ligase as part of general Ubiquitin research is frequently linked to Structural diversity, thereby connecting diverse disciplines of science. In his research, Computational biology is intimately related to Proteomics, which falls under the overarching field of Proteome.
Cell biology, Proteome, Disease, Proteomics and Neuroscience are his primary areas of study. His research in Cell biology intersects with topics in RNA splicing, Neuroinflammation, Neurodegeneration and Microglia. Within the field of Biochemistry and Genetics Nicholas T. Seyfried studies Proteome.
His Disease study incorporates themes from Cancer research, Cerebrospinal fluid, Immunology, Pathogenesis and Asymptomatic. His study in Proteomics is interdisciplinary in nature, drawing from both Pathological, Pathology, Ubiquitin and RNA-binding protein. His biological study spans a wide range of topics, including Amyloid and Cognitive decline.
His scientific interests lie mostly in Proteome, Disease, Computational biology, Cell biology and Neuroscience. Nicholas T. Seyfried interconnects Proteomics and Multiplex in the investigation of issues within Proteome. The various areas that Nicholas T. Seyfried examines in his Disease study include Humanized mouse and Cancer research.
His studies in Computational biology integrate themes in fields like Neuropathology, Transcriptome, Gene and Human brain. His Cell biology study integrates concerns from other disciplines, such as Neuroinflammation, Acetylation and Neurodegeneration. His Neuroscience research includes themes of Quantitative proteomics and Cognitive decline.
The scientist’s investigation covers issues in Proteome, Proteomics, Cell biology, Disease and Quantitative proteomics. His Proteome research is multidisciplinary, incorporating perspectives in Multiplex, Computational biology and Human brain. His work deals with themes such as Arginine, Phosphorylation, Mass spectrometry, Biomarker and RNA-binding protein, which intersect with Proteomics.
In Cell biology, Nicholas T. Seyfried works on issues like Neuroinflammation, which are connected to Cell sorting and GPNMB. His Disease research incorporates themes from Cerebrospinal fluid and Pathophysiology. In his research on the topic of Quantitative proteomics, Synucleinopathies, Synuclein, Downregulation and upregulation and Alpha-synuclein is strongly related with Microglia.
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Quantitative proteomics reveals the function of unconventional ubiquitin chains in proteasomal degradation.
Ping Xu;Duc M. Duong;Nicholas T. Seyfried;Dongmei Cheng.
Continuous release of endostatin from microencapsulated engineered cells for tumor therapy.
Tatsuhiro Joki;Tatsuhiro Joki;Tatsuhiro Joki;Marcelle Machluf;Anthony Atala;Jianhong Zhu;Jianhong Zhu.
Nature Biotechnology (2001)
Expression of Cyclooxygenase 2 (COX-2) in Human Glioma and in Vitro Inhibition by a Specific COX-2 Inhibitor, NS-398
Tatsuhiro Joki;Oliver Heese;Demetrios C. Nikas;Lorenzo Bello.
Cancer Research (2000)
TDP-43 pathology disrupts nuclear pore complexes and nucleocytoplasmic transport in ALS/FTD
Ching Chieh Chou;Yi Zhang;Yi Zhang;Mfon E. Umoh;Spencer W. Vaughan.
Nature Neuroscience (2018)
Cleavage of tau by asparagine endopeptidase mediates the neurofibrillary pathology in Alzheimer’s disease
Zhentao Zhang;Mingke Song;Xia Liu;Seong Su Kang.
Nature Medicine (2014)
A Multi-network Approach Identifies Protein-Specific Co-expression in Asymptomatic and Symptomatic Alzheimer’s Disease
Nicholas T. Seyfried;Eric B. Dammer;Vivek Swarup;Divya Nandakumar.
Cell systems (2017)
U1 small nuclear ribonucleoprotein complex and RNA splicing alterations in Alzheimer's disease
Bing Bai;Chadwick M. Hales;Ping Chung Chen;Yair Gozal.
Proceedings of the National Academy of Sciences of the United States of America (2013)
Evidence for brain glucose dysregulation in Alzheimer's disease
Yang An;Vijay R. Varma;Sudhir Varma;Ramon Casanova.
Alzheimers & Dementia (2018)
Polyubiquitin Linkage Profiles in Three Models of Proteolytic Stress Suggest the Etiology of Alzheimer Disease
Eric B. Dammer;Chan Hyun Na;Ping Xu;Nicholas T. Seyfried.
Journal of Biological Chemistry (2011)
Glutamate Dehydrogenase 1 Signals through Antioxidant Glutathione Peroxidase 1 to Regulate Redox Homeostasis and Tumor Growth
Lingtao Jin;Dan Li;Gina N. Alesi;Jun Fan.
Cancer Cell (2015)
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