D-Index & Metrics Best Publications
Jacques P. Tremblay

Jacques P. Tremblay

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 52 Citations 6,410 220 World Ranking 1712 National Ranking 51

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

Jacques P. Tremblay spends much of his time researching Transplantation, Myocyte, Duchenne muscular dystrophy, Dystrophin and Immunology. His Transplantation study combines topics in areas such as Cell, Genetic enhancement, Bioinformatics and Pathology. The subject of his Myocyte research is within the realm of Cell biology.

The various areas that Jacques P. Tremblay examines in his Duchenne muscular dystrophy study include Mutation, Endocrinology, Transgene and Cancer research. His work deals with themes such as Biceps, Surgery, Molecular biology and Autologous transplantation, which intersect with Dystrophin. Jacques P. Tremblay combines subjects such as Distribution and Myopathy with his study of Immunology.

His most cited work include:

  • First test of a ''high-density injection'' protocol for myogenic cell transplantation throughout large volumes of muscles in a Duchenne muscular dystrophy patient: eighteen months follow-up (153 citations)
  • Efficacy of myoblast transplantation in nonhuman primates following simple intramuscular cell injections: toward defining strategies applicable to humans. (111 citations)
  • Evidence of mdx mouse skeletal muscle fragility in vivo by eccentric running exercise. (108 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Transplantation, Myocyte, Duchenne muscular dystrophy, Dystrophin and Molecular biology. His Transplantation research integrates issues from Immunosuppression, Immunology, Precursor cell and Anatomy. His Myocyte research incorporates themes from Skeletal muscle, Cancer research and Pathology.

His Duchenne muscular dystrophy study incorporates themes from Cell therapy, Autologous transplantation, Transgene and Myostatin. His biological study spans a wide range of topics, including Myopathy and Exon. In his research, Plasmid is intimately related to Transfection, which falls under the overarching field of Molecular biology.

He most often published in these fields:

  • Transplantation (47.41%)
  • Myocyte (44.81%)
  • Duchenne muscular dystrophy (32.59%)

What were the highlights of his more recent work (between 2013-2021)?

  • Gene (9.26%)
  • CRISPR (5.93%)
  • Molecular biology (24.44%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Gene, CRISPR, Molecular biology, Duchenne muscular dystrophy and Myocyte. He interconnects Plasmid, Cell culture, Transfection and Expression vector in the investigation of issues within Molecular biology. His Duchenne muscular dystrophy research is mostly focused on the topic Dystrophin.

While the research belongs to areas of Dystrophin, Jacques P. Tremblay spends his time largely on the problem of Exon, intersecting his research to questions surrounding Muscular dystrophy and Cell biology. His Myocyte study combines topics from a wide range of disciplines, such as Immunosuppression, Pathology, Cell therapy, Skeletal muscle and Transplantation. His studies in Transplantation integrate themes in fields like Precursor cell and Flow cytometry, Immunology.

Between 2013 and 2021, his most popular works were:

  • Efficient Restoration of the Dystrophin Gene Reading Frame and Protein Structure in DMD Myoblasts Using the CinDel Method (52 citations)
  • Crispr-based methods and products for increasing frataxin levels and uses thereof (36 citations)
  • An AAV9 coding for frataxin clearly improved the symptoms and prolonged the life of Friedreich ataxia mouse models. (35 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

His main research concerns Duchenne muscular dystrophy, Dystrophin, Myocyte, Pathology and Molecular biology. His study in Duchenne muscular dystrophy is interdisciplinary in nature, drawing from both Spinal muscular atrophy, Strimvelis, Retinitis pigmentosa and Choroideremia. As a part of the same scientific family, he mostly works in the field of Dystrophin, focusing on Exon and, on occasion, CRISPR, Intron, Muscular dystrophy and Donor derived.

Jacques P. Tremblay has researched Myocyte in several fields, including Histology, Cell therapy, Transplantation and Skeletal muscle. His Transplantation study integrates concerns from other disciplines, such as Cell, Stem cell and Centimeter. His biological study deals with issues like Gene, which deal with fields such as Effector.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

First test of a ''high-density injection'' protocol for myogenic cell transplantation throughout large volumes of muscles in a Duchenne muscular dystrophy patient: eighteen months follow-up

Daniel Skuk;Marlyne Goulet;Brigitte Roy;Vincent Piette.
Neuromuscular Disorders (2007)

246 Citations

Efficacy of myoblast transplantation in nonhuman primates following simple intramuscular cell injections: toward defining strategies applicable to humans.

Daniel Skuk;Marlyne Goulet;Brigitte Roy;Jacques P. Tremblay.
Experimental Neurology (2002)

171 Citations

Myoblast transplantation: the current status of a potential therapeutic tool for myopathies.

Daniel Skuk;Jacques P. Tremblay.
Journal of Muscle Research and Cell Motility (2003)

163 Citations

Evidence of mdx mouse skeletal muscle fragility in vivo by eccentric running exercise.

J.-T. Vilquin;V. Brussee;I. Asselin;I. Kinoshita.
Muscle & Nerve (1998)

163 Citations

Electrotransfer of naked DNA in the skeletal muscles of animal models of muscular dystrophies

J T Vilquin;P F Kennel;M Paturneau-Jouas;P Chapdelaine.
Gene Therapy (2001)

155 Citations

Human myoblast transplantation in immunodeficient and immunosuppressed mice: Evidence of rejection

Johnny Huard;Raynald Roy;Benoît Guérette;Steeve Verreault.
Muscle & Nerve (1994)

148 Citations

Resetting the problem of cell death following muscle-derived cell transplantation: detection, dynamics and mechanisms.

Daniel Skuk;Nicolas J. Caron;Marlyne Goulet;Brigitte Roy.
Journal of Neuropathology and Experimental Neurology (2003)

136 Citations

Successful Myoblast Transplantation in Primates Depends on Appropriate Cell Delivery and Induction of Regeneration in the Host Muscle

Daniel Skuk;Brigitte Roy;Marlyne Goulet;Jacques P. Tremblay.
Experimental Neurology (1999)

135 Citations

Autologous transplantation of muscle precursor cells modified with a lentivirus for muscular dystrophy: human cells and primate models.

Simon P Quenneville;Pierre Chapdelaine;Daniel Skuk;Matin Paradis.
Molecular Therapy (2007)

130 Citations

Successful histocompatible myoblast transplantation in dystrophin-deficient mdx mouse despite the production of antibodies against dystrophin.

J T Vilquin;E Wagner;I Kinoshita;R Roy.
Journal of Cell Biology (1995)

123 Citations

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