Desmond J. Fitzgerald mainly investigates Platelet, Platelet activation, Internal medicine, Biochemistry and Pharmacology. A large part of his Platelet studies is devoted to Thromboxane. His Thromboxane research incorporates elements of Cyclooxygenase and Prostacyclin.
His research integrates issues of Proteome, Proteomics, Thrombin and Thromboxane A2 in his study of Platelet activation. Desmond J. Fitzgerald combines subjects such as Gastroenterology, Endocrinology and Cardiology with his study of Internal medicine. His Biochemistry study often links to related topics such as Molecular biology.
His primary areas of investigation include Internal medicine, Platelet, Pharmacology, Molecular biology and Biochemistry. His work carried out in the field of Internal medicine brings together such families of science as Cyclooxygenase, Endocrinology, Gastroenterology and Cardiology. His Platelet research is multidisciplinary, incorporating elements of Anesthesia and Aspirin.
His Molecular biology study incorporates themes from Cell culture, Pseudomonas exotoxin and Antibody. His Biochemistry research is mostly focused on the topic Receptor. His Thromboxane study combines topics in areas such as Thromboxane B2 and Prostacyclin.
Internal medicine, Cell biology, Platelet, Biochemistry and Endocrinology are his primary areas of study. His Internal medicine research is multidisciplinary, relying on both Immunology, Genotype and Cardiology. His studies in Platelet integrate themes in fields like Aspirin, Proteomics and Transcription profiling.
His study on Integrin expression, Nuclear protein, Mechanism of action and Turn is often connected to NeutrAvidin as part of broader study in Biochemistry. He has researched Endocrinology in several fields, including Receptor, Signal transduction, Cyclooxygenase, Apolipoprotein E and Interleukin 10. His work is dedicated to discovering how Platelet activation, Molecular biology are connected with Immune system, Protein A and Gene expression and other disciplines.
Desmond J. Fitzgerald spends much of his time researching Cell biology, Internal medicine, Cancer research, Receptor and Biochemistry. His Internal medicine research integrates issues from Endocrinology and Signal transduction. His work deals with themes such as Cell growth, Cell cycle, Prostaglandin E2 receptor, EP4 Receptor and Cell surface receptor, which intersect with Cancer research.
His Receptor study also includes
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Platelet activation in unstable coronary disease.
Desmond J. Fitzgerald;Louis Roy;Francesca Catella;Garret A. FitzGerald.
The New England Journal of Medicine (1986)
Characterization of the proteins released from activated platelets leads to localization of novel platelet proteins in human atherosclerotic lesions
Judith A. Coppinger;Gerard Cagney;Gerard Cagney;Sinead Toomey;Sinead Toomey;Thomas Kislinger;Thomas Kislinger.
Ticlopidine and clopidogrel
Martin J. Quinn;Desmond J. Fitzgerald.
The Relationship Between Cyclooxygenase-2 Expression and Colorectal Cancer
Katherine M. Sheehan;Kieran Sheahan;Diarmuid P. O'Donoghue;Fergus MacSweeney.
The alpha 2-macroglobulin receptor/low density lipoprotein receptor-related protein binds and internalizes Pseudomonas exotoxin A
M Z Kounnas;R E Morris;M R Thompson;D J FitzGerald.
Journal of Biological Chemistry (1992)
Marked platelet activation in vivo after intravenous streptokinase in patients with acute myocardial infarction.
D J Fitzgerald;F Catella;L Roy;G A FitzGerald.
OXIDATIVE DAMAGE OF CARDIOMYOCYTES IS LIMITED BY EXTRACELLULAR REGULATED KINASES 1/2-MEDIATED INDUCTION OF CYCLOOXYGENASE-2
Sharon R. Adderley;Desmond J. Fitzgerald.
Journal of Biological Chemistry (1999)
Expert Consensus Document on the Use of Antiplatelet Agents The Task Force on the Use of Antiplatelet Agents in Patients with Atherosclerotic Cardiovascular Disease of the European Society of Cardiology
C Patrono;F Bachmann;C Baigent;C Bode.
European Heart Journal (2004)
Regulation of connexin 43-mediated gap junctional intercellular communication by Ca2+ in mouse epidermal cells is controlled by E-cadherin.
W. M. F. Jongen;D. J. Fitzgerald;M. Asamoto;C. Piccoli.
Journal of Cell Biology (1991)
Cyclooxygenase-1 and -2–Dependent Prostacyclin Formation in Patients With Atherosclerosis
Orina Belton;Dara Byrne;Dermot Kearney;Austin Leahy.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: