What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Enzyme
Desmond J. Fitzgerald mainly investigates Platelet, Platelet activation, Internal medicine, Biochemistry and Pharmacology.
A large part of his Platelet studies is devoted to Thromboxane.
His Thromboxane research incorporates elements of Cyclooxygenase and Prostacyclin.
His research integrates issues of Proteome, Proteomics, Thrombin and Thromboxane A2 in his study of Platelet activation.
Desmond J. Fitzgerald combines subjects such as Gastroenterology, Endocrinology and Cardiology with his study of Internal medicine.
His Biochemistry study often links to related topics such as Molecular biology.
His most cited work include:
- Platelet activation in unstable coronary disease. (1054 citations)
- Characterization of the proteins released from activated platelets leads to localization of novel platelet proteins in human atherosclerotic lesions (612 citations)
- Characterization of the proteins released from activated platelets leads to localization of novel platelet proteins in human atherosclerotic lesions (612 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Internal medicine, Platelet, Pharmacology, Molecular biology and Biochemistry.
His work carried out in the field of Internal medicine brings together such families of science as Cyclooxygenase, Endocrinology, Gastroenterology and Cardiology.
His Platelet research is multidisciplinary, incorporating elements of Anesthesia and Aspirin.
His Molecular biology study incorporates themes from Cell culture, Pseudomonas exotoxin and Antibody.
His Biochemistry research is mostly focused on the topic Receptor.
His Thromboxane study combines topics in areas such as Thromboxane B2 and Prostacyclin.
He most often published in these fields:
- Internal medicine (27.82%)
- Platelet (29.20%)
- Pharmacology (19.83%)
What were the highlights of his more recent work (between 2008-2018)?
- Internal medicine (27.82%)
- Cell biology (7.71%)
- Platelet (29.20%)
In recent papers he was focusing on the following fields of study:
Internal medicine, Cell biology, Platelet, Biochemistry and Endocrinology are his primary areas of study.
His Internal medicine research is multidisciplinary, relying on both Immunology, Genotype and Cardiology.
His studies in Platelet integrate themes in fields like Aspirin, Proteomics and Transcription profiling.
His study on Integrin expression, Nuclear protein, Mechanism of action and Turn is often connected to NeutrAvidin as part of broader study in Biochemistry.
He has researched Endocrinology in several fields, including Receptor, Signal transduction, Cyclooxygenase, Apolipoprotein E and Interleukin 10.
His work is dedicated to discovering how Platelet activation, Molecular biology are connected with Immune system, Protein A and Gene expression and other disciplines.
Between 2008 and 2018, his most popular works were:
- Safety and Tolerability of Atopaxar in the Treatment of Patients With Acute Coronary Syndromes: The Lessons From Antagonizing the Cellular Effects of Thrombin-Acute Coronary Syndromes Trial (125 citations)
- Randomized Trial of Atopaxar in the Treatment of Patients With Coronary Artery Disease The Lessons From Antagonizing the Cellular Effect of Thrombin–Coronary Artery Disease Trial (101 citations)
- Transcription profiling in human platelets reveals LRRFIP1 as a novel protein regulating platelet function. (78 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
Desmond J. Fitzgerald spends much of his time researching Cell biology, Internal medicine, Cancer research, Receptor and Biochemistry.
His Internal medicine research integrates issues from Endocrinology and Signal transduction.
His work deals with themes such as Cell growth, Cell cycle, Prostaglandin E2 receptor, EP4 Receptor and Cell surface receptor, which intersect with Cancer research.
His Receptor study also includes
- Nuclear export signal which intersects with area such as Prostaglandin,
- Apolipoprotein E and Peroxisome proliferator-activated receptor most often made with reference to Macrophage.
His Apolipoprotein E study which covers Cyclooxygenase that intersects with Platelet.
His research on Platelet focuses in particular on Platelet activation.
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