His primary areas of study are Cell biology, Apoptosis, Programmed cell death, Caspase and Neuroscience. As part of his studies on Cell biology, Christoph Borner often connects relevant subjects like Biochemistry. His Apoptosis study integrates concerns from other disciplines, such as Cancer research and Cell growth.
His Programmed cell death research includes elements of Cancer cell, Mutant and Model organism. His studies deal with areas such as Caspase 3, Proteases, Cysteine protease, Immunology and Molecular biology as well as Caspase. His work focuses on many connections between Neuroscience and other disciplines, such as Signal transduction, that overlap with his field of interest in Membrane protein.
The scientist’s investigation covers issues in Cell biology, Apoptosis, Programmed cell death, Molecular biology and Protein kinase C. His research investigates the connection between Cell biology and topics such as Bcl-2-associated X protein that intersect with issues in Bcl-2 Homologous Antagonist-Killer Protein. His research in Apoptosis intersects with topics in Tumor necrosis factor alpha and Cancer research.
He usually deals with Programmed cell death and limits it to topics linked to Neuroscience and Necroptosis. His research investigates the connection between Molecular biology and topics such as Mitogen-activated protein kinase kinase that intersect with problems in MAP2K7 and Cyclin-dependent kinase 2. His study looks at the relationship between Protein kinase C and topics such as Gene isoform, which overlap with Carcinogenesis.
Christoph Borner mainly investigates Cell biology, Apoptosis, Programmed cell death, Tumor necrosis factor alpha and Signal transduction. His Cell biology study combines topics in areas such as Semliki Forest virus and Nuclear protein. His Apoptosis research includes themes of RNA interference and Viral replication.
The concepts of his Programmed cell death study are interwoven with issues in Autophagy, Glutathione, Neuroscience and Intracellular. Christoph Borner combines subjects such as Pyroptosis, Immunogenic cell death, Intrinsic apoptosis and Necroptosis with his study of Neuroscience. His Tumor necrosis factor alpha research incorporates themes from NFKB1 and NF-κB.
Christoph Borner focuses on Programmed cell death, Apoptosis, Cell biology, Signal transduction and Neuroscience. In Programmed cell death, Christoph Borner works on issues like Autophagy, which are connected to Multicellular organism. His Apoptosis research is multidisciplinary, incorporating perspectives in Jurkat cells and Cell signaling.
His study deals with a combination of Cell biology and Mechanism. The various areas that he examines in his Signal transduction study include Ubiquitin, Cytokine, NFKB1, Deubiquitinating Enzyme CYLD and Allosteric regulation. His Neuroscience research is multidisciplinary, relying on both Pyroptosis, Entosis, Immunogenic cell death, Intrinsic apoptosis and Necroptosis.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.
Lorenzo Galluzzi;Ilio Vitale;Stuart A. Aaronson;John M. Abrams.
Cell Death & Differentiation (2018)
Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c
Thierry Rossé;Reynald Olivier;Laurent Monney;Monika Rager.
Nature (1998)
The Bcl-2 protein family: sensors and checkpoints for life-or-death decisions.
Christoph Borner.
Molecular Immunology (2003)
Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
L. Galluzzi;J. M. Bravo-San Pedro;I. Vitale;S. A. Aaronson.
Cell Death & Differentiation (2015)
Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME
Patricio Godoy;Nicola J. Hewitt;Ute Albrecht;Melvin E. Andersen.
Archives of Toxicology (2013)
Guidelines for the use and interpretation of assays for monitoring cell death in higher eukaryotes
L. Galluzzi;L. Galluzzi;L. Galluzzi;S. A. Aaronson;J. Abrams;E. S. Alnemri.
Cell Death & Differentiation (2009)
Bcl-2 decreases the free Ca2+ concentration within the endoplasmic reticulum
Reyhaneh Foyouzi-Youssefi;Serge Arnaudeau;Christoph Borner;William L. Kelley.
Proceedings of the National Academy of Sciences of the United States of America (2000)
XIAP discriminates between type I and type II FAS-induced apoptosis
Philipp. Jost;Stephanie. Grabow;Stephanie. Grabow;Daniel. Gray;Mark D. McKenzie;Mark D. McKenzie.
Nature (2009)
Nephrin and CD2AP associate with phosphoinositide 3-OH kinase and stimulate AKT-dependent signaling.
Tobias B. Huber;Björn Hartleben;Jeong Kim;Miriam Schmidts.
Molecular and Cellular Biology (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Freiburg
Novartis (Switzerland)
Institut Gustave Roussy
Sorbonne University
Cornell University
Walter and Eliza Hall Institute of Medical Research
The University of Texas Southwestern Medical Center
University of Rome Tor Vergata
Karolinska Institute
Inserm : Institut national de la santé et de la recherche médicale
Georgia Institute of Technology
Pennsylvania State University
Johnson & Johnson
University of Michigan–Ann Arbor
University of Queensland
Royal Belgian Institute of Natural Sciences
Addis Ababa University
University of California, Santa Cruz
West Virginia University
Sungkyunkwan University
University of Cologne
Boston University
Duke University
University of Memphis
University of York
Duke University