Christine Chaponnier spends much of her time researching Cell biology, Actin, Myofibroblast, Granulation tissue and Wound healing. Her study in Cell biology focuses on Cytoplasm in particular. Her Actin study combines topics in areas such as Profilin, Cell, Immunology and Microfilament.
Her Myofibroblast study combines topics from a wide range of disciplines, such as Myocyte, Vascular smooth muscle and Anatomy. Her research investigates the connection between Wound healing and topics such as Pathology that intersect with issues in Fibronectin. Her Myofibroblast contraction research is multidisciplinary, incorporating elements of Molecular biology and Contractility.
Her scientific interests lie mostly in Actin, Cell biology, Molecular biology, Cytoskeleton and Pathology. The concepts of her Actin study are interwoven with issues in Cell, Cytoplasm, Myofibroblast, Myocyte and Myosin. Her study in Myofibroblast is interdisciplinary in nature, drawing from both Wound healing, Granulation tissue, Vascular smooth muscle and Anatomy.
MDia1 is closely connected to Actin remodeling in her research, which is encompassed under the umbrella topic of Cell biology. Her work in Molecular biology covers topics such as In vitro which are related to areas like Antibody. She interconnects Fibroblast and Cancer in the investigation of issues within Pathology.
The scientist’s investigation covers issues in Actin, Cell biology, Cytoskeleton, Myocyte and Cytoplasm. Her work carried out in the field of Actin brings together such families of science as Skeletal muscle, Molecular biology, Contraction and Myosin. Christine Chaponnier undertakes interdisciplinary study in the fields of Cell biology and Mitotic spindle organization through her works.
Her Cytoskeleton study incorporates themes from Virus, Myofibroblast and Cell polarity. Her Myofibroblast research integrates issues from Transdifferentiation and Arp2/3 complex. As part of one scientific family, she deals mainly with the area of Myocyte, narrowing it down to issues related to the Pathology, and often Actinin.
Christine Chaponnier mostly deals with Actin, Cell biology, Myocyte, Cytoplasm and Cytoskeleton. Her Actin research incorporates themes from Sarcomere and Heart failure. Her Cell biology research incorporates elements of Myopathy, Contraction and Skeletal muscle.
Her Myocyte research includes themes of ACTA2 and Growth factor receptor. Her Cytoplasm research is multidisciplinary, relying on both Cell, Motility, Actin cytoskeleton and Monoclonal antibody. The various areas that Christine Chaponnier examines in her Cytoskeleton study include Flow cytometry and Cell polarity.
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Myofibroblasts and mechano-regulation of connective tissue remodelling
James J. Tomasek;Giulio Gabbiani;Boris Hinz;Christine Chaponnier.
Nature Reviews Molecular Cell Biology (2002)
Alpha-Smooth Muscle Actin Expression Upregulates Fibroblast Contractile Activity
Boris Hinz;Giuseppe Celetta;James J. Tomasek;Giulio Gabbiani.
Molecular Biology of the Cell (2001)
Tissue repair, contraction, and the myofibroblast.
Alexis Desmoulière;Christine Chaponnier;Giulio Gabbiani.
Wound Repair and Regeneration (2005)
Mechanical tension controls granulation tissue contractile activity and myofibroblast differentiation
Boris Hinz;Dominique Mastrangelo;Christophe E. Iselin;Christine Chaponnier.
American Journal of Pathology (2001)
Vascular smooth muscle cells differ from other smooth muscle cells: predominance of vimentin filaments and a specific alpha-type actin.
G. Gabbiani;E. Schmid;S. Winter;C. Chaponnier.
Proceedings of the National Academy of Sciences of the United States of America (1981)
Cytoplasmic filaments and gap junctions in epithelial cells and myofibroblasts during wound healing
Giulio Gabbiani;Christine Chaponnier;I. Huttner.
Journal of Cell Biology (1978)
Focal adhesion features during myofibroblastic differentiation are controlled by intracellular and extracellular factors
Vera Dugina;Lionel Fontao;Christine Chaponnier;Jury Vasiliev.
Journal of Cell Science (2001)
Evidence of an intracellular reservoir in the nasal mucosa of patients with recurrent Staphylococcus aureus rhinosinusitis.
Sophie Clement;Pierre Vaudaux;Patrice Francois;Jacques Schrenzel.
The Journal of Infectious Diseases (2005)
The NH2-terminal peptide of α–smooth muscle actin inhibits force generation by the myofibroblast in vitro and in vivo
Boris Hinz;Giulio Gabbiani;Christine Chaponnier.
Journal of Cell Biology (2002)
Alpha-smooth muscle actin is crucial for focal adhesion maturation in myofibroblasts.
Boris Hinz;Vera Dugina;Christoph Ballestrem;Bernhard Wehrle-Haller.
Molecular Biology of the Cell (2003)
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