What is he best known for?
The fields of study he is best known for:
- Gene
- Biochemistry
- Genetics
Chad A. Dickey mainly investigates Tau protein, Molecular biology, Hsp90, Alzheimer's disease and Cell biology.
His work in Tau protein addresses issues such as Neurodegeneration, which are connected to fields such as Neuroscience, Progressive supranuclear palsy and Brain tissue.
The Hsp90 study combines topics in areas such as Ubiquitin ligase and Phosphorylation.
His Ubiquitin ligase study integrates concerns from other disciplines, such as Heat shock factor, Mediator and HSF1.
His studies deal with areas such as Genetically modified mouse and Tauopathy as well as Alzheimer's disease.
His Genetically modified mouse research includes elements of BECN1 and Immunology.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Aβ peptide vaccination prevents memory loss in an animal model of Alzheimer's disease (1486 citations)
- Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17 (1484 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Cell biology, Tau protein, Chaperone, Hsp90 and Neuroscience.
His Cell biology research is multidisciplinary, incorporating elements of Heat shock protein, Hsp70 and Tauopathy.
Alzheimer's disease covers Chad A. Dickey research in Tau protein.
His research integrates issues of Dementia and Amyloid in his study of Alzheimer's disease.
The concepts of his Hsp90 study are interwoven with issues in Receptor, Small molecule and Proteasome.
His Neuroscience research incorporates themes from Disease and Neurodegeneration.
He most often published in these fields:
- Cell biology (38.10%)
- Tau protein (23.81%)
- Chaperone (20.24%)
What were the highlights of his more recent work (between 2016-2021)?
- Hsp90 (20.83%)
- Cell biology (38.10%)
- Chaperone (20.24%)
In recent papers he was focusing on the following fields of study:
Chad A. Dickey focuses on Hsp90, Cell biology, Chaperone, Biochemistry and Protein folding.
His Hsp90 research is multidisciplinary, incorporating elements of Receptor, Biophysics and Genetically modified mouse.
His Cell biology study incorporates themes from Phenotype, Mutant, Binding site and Tauopathy.
His research in Chaperone intersects with topics in Tau protein, Neuroscience and Gene knockdown.
Biochemistry is closely attributed to Amyloid in his work.
His Protein folding research includes themes of Regulator, Myocilin, Computational biology and Co-chaperone.
Between 2016 and 2021, his most popular works were:
- Mapping interactions with the chaperone network reveals factors that protect against tau aggregation. (47 citations)
- Hsp90 activator Aha1 drives production of pathological tau aggregates. (43 citations)
- Structure and pro-toxic mechanism of the human Hsp90/PPIase/Tau complex. (31 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Biochemistry
- Genetics
The scientist’s investigation covers issues in Hsp90, Chaperone, Tau protein, Heat shock protein and Cell biology.
Chad A. Dickey has researched Hsp90 in several fields, including Receptor, Genetically modified mouse and FKBP5.
His research integrates issues of Glutamate receptor, AMPA receptor, Cognition, Neuroscience and Dementia in his study of Genetically modified mouse.
His work carried out in the field of Chaperone brings together such families of science as Biophysics and Cellular homeostasis.
In Tau protein, he works on issues like Biochemistry, which are connected to Amyloid.
His study in Heat shock protein is interdisciplinary in nature, drawing from both Activator and Gene isoform.
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