2008 - Fellow of the American Association for the Advancement of Science (AAAS)
Virology, Virus, Simian immunodeficiency virus, Viral envelope and Viral replication are her primary areas of study. Her Virology research incorporates elements of Molecular biology, Glycoprotein, CD4 antigen and Immunology. Her Virus research incorporates themes from Macrophage, Antibody and Gene.
As a part of the same scientific study, Cecilia Cheng-Mayer usually deals with the Simian immunodeficiency virus, concentrating on Simian and frequently concerns with Viremia. Her work deals with themes such as Coreceptor activity and Tropism, which intersect with Viral envelope. The Viral replication study combines topics in areas such as Glioma, Cell type and Antigen.
Her scientific interests lie mostly in Virology, Virus, Simian immunodeficiency virus, Immunology and Viral replication. Her Virology research includes elements of V3 loop and Antibody. Her work carried out in the field of V3 loop brings together such families of science as Amino acid, Molecular biology, Conserved sequence and Monoclonal antibody.
As a part of the same scientific family, Cecilia Cheng-Mayer mostly works in the field of Virus, focusing on Clone and, on occasion, Simian AIDS and Cell culture. As part of the same scientific family, Cecilia Cheng-Mayer usually focuses on Simian immunodeficiency virus, concentrating on Immunodeficiency and intersecting with Recombinant DNA. Her Viral replication research integrates issues from Viral culture, Glycosylation, Oncovirus, Viral transformation and Antiviral antibody.
Cecilia Cheng-Mayer mainly investigates Virology, Simian immunodeficiency virus, Virus, Immunology and Viral load. Virology is often connected to Antibody in her work. In general Simian immunodeficiency virus study, her work on Simian Acquired Immunodeficiency Syndrome often relates to the realm of Transmissibility, Cabotegravir and Integrase inhibitor, thereby connecting several areas of interest.
Her work on Simian and Viral transformation as part of her general Virus study is frequently connected to Resistance mutation, thereby bridging the divide between different branches of science. Her study looks at the intersection of Immunology and topics like Transmission with Sexual transmission and Genetic variation. Her Viral replication research incorporates elements of Viral culture, Recombinant virus, Molecular biology and Oncovirus.
Cecilia Cheng-Mayer mostly deals with Simian immunodeficiency virus, Virology, Viral load, Macaque and Rhesus macaque. Her Simian immunodeficiency virus study improves the overall literature in Virus. Her study looks at the relationship between Virology and fields such as AIDS Vaccines, as well as how they intersect with chemical problems.
Her study in V3 loop is interdisciplinary in nature, drawing from both Epitope mapping and Monoclonal antibody. Her Tissue tropism research is multidisciplinary, relying on both Viral envelope, Encephalitis and Viral replication. The study incorporates disciplines such as Plasma protein binding, Cell biology and Binding site in addition to Tropism.
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CD4-dependent, antibody-sensitive interactions between HIV-1 and its co-receptor CCR-5.
Alexandra Trkola;Tatjana Dragic;James Arthos;James M. Binley.
Nature (1996)
Hepatitis C virus glycoproteins mediate pH-dependent cell entry of pseudotyped retroviral particles.
Mayla Hsu;Jie Zhang;Mike Flint;Carine Logvinoff.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Biologic features of HIV-1 that correlate with virulence in the host
Cecilia Cheng-Mayer;Deborah Seto;Masatoshi Tateno;Jay A. Levy.
Science (1988)
Antibody Protects Macaques against Vaginal Challenge with a Pathogenic R5 Simian/Human Immunodeficiency Virus at Serum Levels Giving Complete Neutralization In Vitro
Paul W. H. I. Parren;Preston A. Marx;Preston A. Marx;Ann J. Hessell;Amara Luckay.
Journal of Virology (2001)
Macrophage and T cell-line tropisms of HIV-1 are determined by specific regions of the envelope gp120 gene.
Tatsuo Shioda;Jay A. Levy;Cecilia Cheng-Mayer.
Nature (1991)
Small amino acid changes in the V3 hypervariable region of gp120 can affect the T-cell-line and macrophage tropism of human immunodeficiency virus type 1.
Tatsuo Shioda;Jay A. Levy;Cecilia Cheng-Mayer.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Distinct pathogenic sequela in rhesus macaques infected with CCR5 or CXCR4 utilizing SHIVs
Janet M. Harouse;Agegnehu Gettie;Rei Chin How Tan;James Blanchard.
Science (1999)
Human immunodeficiency virus can productively infect cultured human glial cells.
Cecilia Cheng-Mayer;James T. Rutka;Mark L. Rosenblum;Thomas McHugh.
Proceedings of the National Academy of Sciences of the United States of America (1987)
Mutational analysis of the human immunodeficiency virus: the orf-B region down-regulates virus replication
Paul A. Luciw;Cecilia Cheng-Mayer;Jay A. Levy.
Proceedings of the National Academy of Sciences of the United States of America (1987)
HIV-1 nef leads to inhibition or activation of T cells depending on its intracellular localization
Andreas S. Baur;Earl T. Sawai;Paul Dazin;Wendy J. Fantl.
Immunity (1994)
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