What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Cancer
- Gene
His scientific interests lie mostly in Immunology, Chemokine, Immune system, CCL27 and Cell biology.
All of his Immunology and Inflammation, T cell, Cytokine, CXCL16 and C-C chemokine receptor type 6 investigations are sub-components of the entire Immunology study.
Bernhard Homey interconnects SUPERFAMILY, Cancer and Cancer research in the investigation of issues within Chemokine.
His research investigates the connection with Cancer research and areas like CCR10 which intersect with concerns in Lymphocyte and Tumor necrosis factor alpha.
His research integrates issues of Dermatology, Interferon, Surgery and MEDLINE in his study of Immune system.
His Chemokine receptor study combines topics from a wide range of disciplines, such as Cancer cell, Angiogenesis and Metastasis.
His most cited work include:
- Involvement of chemokine receptors in breast cancer metastasis. (4370 citations)
- Plasmacytoid dendritic cells sense self-DNA coupled with antimicrobial peptide. (1302 citations)
- Plasmacytoid predendritic cells initiate psoriasis through interferon-α production (821 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Immunology, Dermatology, Chemokine, Cancer research and Gynecology.
Inflammation, Atopic dermatitis, Cytokine, Immune system and Allergy are the core of his Immunology study.
His research is interdisciplinary, bridging the disciplines of Psoriasis and Atopic dermatitis.
Epidermal growth factor receptor is closely connected to Adverse effect in his research, which is encompassed under the umbrella topic of Dermatology.
Bernhard Homey combines topics linked to Cell biology with his work on Chemokine.
His study in Cancer research is interdisciplinary in nature, drawing from both Metastasis and Pathology.
He most often published in these fields:
- Immunology (32.13%)
- Dermatology (24.26%)
- Chemokine (14.75%)
What were the highlights of his more recent work (between 2016-2021)?
- Immunology (32.13%)
- Cancer research (12.46%)
- Chemokine (14.75%)
In recent papers he was focusing on the following fields of study:
Bernhard Homey mainly investigates Immunology, Cancer research, Chemokine, Atopic dermatitis and Dermatology.
His research ties Treatment outcome and Immunology together.
When carried out as part of a general Cancer research research project, his work on Melanoma is frequently linked to work in Nucleotide excision repair, therefore connecting diverse disciplines of study.
Bernhard Homey has researched Melanoma in several fields, including Breast cancer, Carcinoma and Metastasis.
His study looks at the intersection of Chemokine and topics like Cell biology with CXCR4, Endothelium and Platelet.
His research in Atopic dermatitis intersects with topics in Psoriasis, SCORAD, Quality of life, Interleukin and Allergic contact dermatitis.
Between 2016 and 2021, his most popular works were:
- TSLP-activated dendritic cells induce human T follicular helper cell differentiation through OX40-ligand (59 citations)
- Microbe-host interplay in atopic dermatitis and psoriasis. (52 citations)
- Human "TH9" cells are a subpopulation of PPAR-γ+ TH2 cells. (28 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Cancer
- Gene
His primary areas of investigation include Immunology, Chemokine, Cancer research, Inflammation and Immunoglobulin G.
The concepts of his Immunology study are interwoven with issues in Genetic variation and BCL6.
His research investigates the connection between Chemokine and topics such as Wound healing that intersect with problems in CCR3, CCR10, C-C chemokine receptor type 6, CXC chemokine receptors and Interferon.
Bernhard Homey works in the field of Cancer research, focusing on Melanoma in particular.
His biological study spans a wide range of topics, including Cell, DNA and Function, Cell biology.
His Immunoglobulin G research includes elements of Pemphigus vulgaris, Autoimmunity and Interleukin 4.
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