His main research concerns Cell biology, Cell cycle, Cyclin-dependent kinase 1, Cyclin-dependent kinase and Kinase. He has researched Cell biology in several fields, including CHEK1, Biochemistry and Proliferating cell nuclear antigen. His biological study spans a wide range of topics, including Molecular biology and Cell growth.
His Cyclin-dependent kinase study integrates concerns from other disciplines, such as Phosphatase, CDC25A and Cyclin-dependent kinase 2. He combines subjects such as Phosphorylation and Cyclin with his study of Kinase. In his work, G1/S transition and Cancer research is strongly intertwined with Centrosome, which is a subfield of Phosphorylation.
Bernard Ducommun focuses on Cell biology, Cell cycle, Biochemistry, Mitosis and Cdc25. His studies deal with areas such as Cyclin-dependent kinase 1, Cyclin-dependent kinase and CHEK1 as well as Cell biology. His study looks at the relationship between Cyclin-dependent kinase 1 and topics such as Cyclin-dependent kinase 7, which overlap with Cyclin-dependent kinase complex.
His Cell cycle study incorporates themes from Multicellular organism, Cancer research, Tumor spheroid and Cell growth. His research in Mitosis focuses on subjects like DNA damage, which are connected to Downregulation and upregulation. His Cdc25 research incorporates themes from HeLa and CDC25A.
Cell biology, Spheroid, Tumor spheroid, Cell and Cancer research are his primary areas of study. When carried out as part of a general Cell biology research project, his work on Phosphorylation is frequently linked to work in Micronucleus test, therefore connecting diverse disciplines of study. The Spheroid study combines topics in areas such as Biophysics, Cell growth and Biomedical engineering.
His Cytoskeleton study in the realm of Cell connects with subjects such as Dynamics. His studies in Cancer research integrate themes in fields like Cancer, Cell cycle, Cell cycle checkpoint, Breast cancer cells and Cluster analysis. His research in Cell cycle intersects with topics in Probabilistic logic and Simulation.
Bernard Ducommun mostly deals with Cell biology, Spheroid, Cell, Cell growth and Phosphorylation. He conducts interdisciplinary study in the fields of Cell biology and Histone code through his research. Within one scientific family, Bernard Ducommun focuses on topics pertaining to Biophysics under Spheroid, and may sometimes address concerns connected to Cancer, In vitro, Characterization and Cancer cell.
His work in Cell growth covers topics such as Cell culture which are related to areas like Proliferation index and Catalase. Bernard Ducommun has included themes like Tumor spheroid, Nanotechnology, DNA damage, Reactive oxygen species and Growth inhibition in his Phosphorylation study. His work deals with themes such as Apoptosis, Programmed cell death, Toxicology and Cysteine, which intersect with DNA damage.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Nuclear gene OPA1, encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophy.
C Delettre;G Lenaers;J-M Griffoin;N Gigarel.
Nature Genetics (2000)
CDC25 phosphatases in cancer cells: key players? Good targets?
Rose Boutros;Valérie Lobjois;Bernard Ducommun.
Nature Reviews Cancer (2007)
The when and wheres of CDC25 phosphatases.
Rose Boutros;Christine Dozier;Bernard Ducommun.
Current Opinion in Cell Biology (2006)
The human dynamin-related protein OPA1 is anchored to the mitochondrial inner membrane facing the inter-membrane space.
Aurélien Olichon;Laurent J Emorine;Eric Descoins;Laetitia Pelloquin.
FEBS Letters (2002)
cdc2 phosphorylation is required for its interaction with cyclin.
B. Ducommun;P. Brambilla;M.-A. Felix;B.R. Franza.
The EMBO Journal (1991)
Histone acetyltransferase activity of CBP is controlled by cycle-dependent kinases and oncoprotein E1A
S. Ait-Si-Ali;S. Ramirez;F.-X. Barre;F. Dkhissi.
p21 binding to PCNA causes G1 and G2 cell cycle arrest in p53-deficient cells
Corinne Cayrol;Martine Knibiehler;Bernard Ducommun.
Phosphorylation of CDC25B by Aurora-A at the centrosome contributes to the G2–M transition
Stéphanie Dutertre;Martine Cazales;Muriel Quaranta;Carine Froment.
Journal of Cell Science (2004)
Distinct nuclear and spindle pole body populations of cyclin–cdc2 in fission yeast
Caroline E. Alfa;Bernard Ducommun;Bernard Ducommun;David Beach;Jeremy S. Hyams.
Involvement of the Interaction between p21 and Proliferating Cell Nuclear Antigen for the Maintenance of G2/M Arrest after DNA Damage
Tomoaki Ando;Takumi Kawabe;Hirotaka Ohara;Bernard Ducommun.
Journal of Biological Chemistry (2001)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: