Member of the Association of American Physicians
Cell biology, Nerve growth factor, Receptor, Neurotrophin and Brain-derived neurotrophic factor are her primary areas of study. Her Cell biology study integrates concerns from other disciplines, such as Plasmin, Immunology and Nervous system. The study incorporates disciplines such as Apoptosis and Cell surface receptor in addition to Nerve growth factor.
Her Neurotrophin study incorporates themes from Receptor tyrosine kinase and Programmed cell death. Her work in Trk receptor addresses subjects such as Low-affinity nerve growth factor receptor, which are connected to disciplines such as Molecular biology. As part of the same scientific family, Barbara L. Hempstead usually focuses on Neurotrophic factors, concentrating on Neuroscience and intersecting with Synaptic plasticity.
Barbara L. Hempstead mainly investigates Cell biology, Neurotrophin, Receptor, Neuroscience and Trk receptor. Her research in Cell biology intersects with topics in Neurotrophic factors, Endocrinology, Programmed cell death and Nerve growth factor. Her study in Neurotrophin is interdisciplinary in nature, drawing from both Apoptosis and Receptor complex.
Her research investigates the connection with Receptor and areas like Immunology which intersect with concerns in Endothelial stem cell. Her studies deal with areas such as Brain-derived neurotrophic factor and Synaptic plasticity as well as Neuroscience. Her work carried out in the field of Trk receptor brings together such families of science as Ligand, Tyrosine kinase, Tropomyosin receptor kinase C, Molecular biology and Receptor tyrosine kinase.
Her main research concerns Neuroscience, Cell biology, Neurotrophic factors, Brain-derived neurotrophic factor and Receptor. In her work, Neurotrophin-3 is strongly intertwined with Nerve growth factor, which is a subfield of Neuroscience. Her work deals with themes such as Cell culture, Neurotrophin, Programmed cell death and HIF1A, which intersect with Cell biology.
She has included themes like Synaptic plasticity and Neuroprotection in her Neurotrophin study. She has researched Brain-derived neurotrophic factor in several fields, including Postsynaptic potential, Tropomyosin receptor kinase A, Immunology, Signal transduction and Tropomyosin receptor kinase B. Barbara L. Hempstead combines subjects such as Hippocampal formation and Growth cone with her study of Receptor.
Her primary areas of study are Brain-derived neurotrophic factor, Cell biology, Neuroscience, Neurotrophic factors and Tropomyosin receptor kinase B. As a part of the same scientific family, Barbara L. Hempstead mostly works in the field of Brain-derived neurotrophic factor, focusing on Hippocampal formation and, on occasion, Neurogenesis and HEK 293 cells. Her Cell biology study deals with Cell culture intersecting with HIF1A.
Barbara L. Hempstead works mostly in the field of Neuroscience, limiting it down to concerns involving Synaptic plasticity and, occasionally, Immunology, Microglia and Signal transduction. Her Neurotrophic factors research is multidisciplinary, incorporating elements of Tropomyosin receptor kinase A, Nerve growth factor, Neuroplasticity and Endocrinology. Her Tropomyosin receptor kinase B research is multidisciplinary, relying on both Long-term potentiation, Low-affinity nerve growth factor receptor, Neurotrophin and Anatomy.
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Regulation of cell survival by secreted proneurotrophins.
Ramee Lee;Pouneh Kermani;Kenneth K. Teng;Barbara L. Hempstead.
Science (2001)
Microglia Promote Learning-Dependent Synapse Formation through Brain-Derived Neurotrophic Factor
Christopher N. Parkhurst;Guang Yang;Ipe Ninan;Jeffrey N. Savas.
Cell (2013)
The trk proto-oncogene product: a signal transducing receptor for nerve growth factor
David R. Kaplan;Barbara L. Hempstead;Dionisio Martin-Zanca;Moses V. Chao.
Science (1991)
High-affinity NGF binding requires coexpression of the trk proto-oncogene and the low-affinity NGF receptor.
Barbara L. Hempstead;Dionisio Martin-Zanca;David R. Kaplan;Luis F. Parada.
Nature (1991)
Genetic variant BDNF (Val66Met) polymorphism alters anxiety-related behavior
Zhe-Yu Chen;Deqiang Jing;Kevin G. Bath;Alessandro Ieraci.
Science (2006)
Variant Brain-Derived Neurotrophic Factor (BDNF) (Met66) Alters the Intracellular Trafficking and Activity-Dependent Secretion of Wild-Type BDNF in Neurosecretory Cells and Cortical Neurons
Zhe Yu Chen;Paresh D. Patel;Gayatree Sant;Chui Xiang Meng.
The Journal of Neuroscience (2004)
Cleavage of proBDNF by tPA/plasmin is essential for long-term hippocampal plasticity.
Petti T. Pang;Petti T. Pang;Henry K. Teng;Eygene Zaitsev;Newton T. Woo.
Science (2004)
Sortilin is essential for proNGF-induced neuronal cell death
Anders Nykjaer;Ramee Lee;Kenneth K. Teng;Pernille Jansen;Pernille Jansen.
Nature (2004)
p75 and Trk: A two-receptor system
Moses V. Chao;Barbara L. Hempstead.
Trends in Neurosciences (1995)
ProBDNF Induces Neuronal Apoptosis via Activation of a Receptor Complex of p75NTR and Sortilin
Henry K. Teng;Kenneth K. Teng;Ramee Lee;Saundrene Wright.
The Journal of Neuroscience (2005)
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