Her work on Immunology as part of general Immunohistochemistry research is frequently linked to Neuroscience, thereby connecting diverse disciplines of science. Immunology is closely attributed to Immunohistochemistry in her research. She performs integrative Neuroscience and Cell biology research in her work. Her Cell biology study frequently draws connections to adjacent fields such as Intracellular. Anne Messer integrates many fields, such as Intracellular and Gene, in her works. Anne Messer undertakes multidisciplinary studies into Gene and Neuronal ceroid lipofuscinosis in her work. She integrates many fields, such as Genetics and Cell, in her works. Her Disease study frequently draws connections between related disciplines such as Huntingtin Protein. Her Huntingtin Protein study typically links adjacent topics like Huntington's disease.
Anne Messer conducts interdisciplinary study in the fields of Gene and Molecular biology through her research. Anne Messer conducts interdisciplinary study in the fields of Molecular biology and Gene through her research. Her research combines In vitro and Genetics. Her study brings together the fields of Genetics and In vitro. Her study ties her expertise on Motor neuron together with the subject of Neuroscience. She integrates Motor neuron with Spinal cord in her study. Many of her studies involve connections with topics such as Neuroscience and Spinal cord. Her studies link Intracellular with Cell biology. In her papers, she integrates diverse fields, such as Biochemistry and Enzyme.
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Msh2 deficiency prevents in vivo somatic instability of the CAG repeat in Huntington disease transgenic mice.
Kevin Manley;Thomas L. Shirley;Thomas L. Shirley;Lorraine Flaherty;Lorraine Flaherty;Anne Messer;Anne Messer.
Nature Genetics (1999)
The neuronal ceroid lipofuscinoses in human EPMR and mnd mutant mice are associated with mutations in CLN8.
Susanna Ranta;Yonghui Zhang;Barbara Ross;Liina Lonka.
Nature Genetics (1999)
Severe deficiencies in dopamine signaling in presymptomatic Huntington's disease mice
James A. Bibb;Zhen Yan;Per Svenningsson;Gretchen L. Snyder.
Proceedings of the National Academy of Sciences of the United States of America (2000)
The maintenance and identification of mouse cerebellar granule cells in monolayer culture
Anne Messer;Anne Messer.
Brain Research (1977)
Human single-chain Fv intrabodies counteract in situ huntingtin aggregation in cellular models of Huntington's disease.
Jean-Michel Lecerf;Thomas L. Shirley;Quan Zhu;Aleksey Kazantsev.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Potent inhibition of huntingtin aggregation and cytotoxicity by a disulfide bond-free single-domain intracellular antibody.
David W. Colby;YiJia Chu;John P. Cassady;Martin Duennwald.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Suppression of Huntington's disease pathology in Drosophila by human single-chain Fv antibodies
William J. Wolfgang;Todd W. Miller;Jack M. Webster;James S. Huston.
Proceedings of the National Academy of Sciences of the United States of America (2005)
A human single-chain Fv intrabody blocks aberrant cellular effects of overexpressed α-synuclein
Chun Zhou;Sharareh Emadi;Michael R. Sierks;Anne Messer;Anne Messer.
Molecular Therapy (2004)
Development of a Human Light Chain Variable Domain (VL) Intracellular Antibody Specific for the Amino Terminus of Huntingtin via Yeast Surface Display
David W. Colby;Payal Garg;Tina Holden;Ginger Chao.
Journal of Molecular Biology (2004)
Dystonin is essential for maintaining neuronal cytoskeleton organization.
Gratien Dalpé;Nicole Leclerc;Annie Vallée;Anne Messer.
Molecular and Cellular Neuroscience (1998)
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