2018 - Member of the National Academy of Sciences
His work often combines Genetics and Mutant studies. Borrowing concepts from Transcription factor, Ying-Hui Fu weaves in ideas under Gene. He frequently studies issues relating to Genetics and Transcription factor. He merges many fields, such as Mutation and Locus (genetics), in his writings. His work often combines Locus (genetics) and Mutation studies. Internal medicine is frequently linked to Dyskinesia in his study. His research on Dyskinesia often connects related areas such as Internal medicine. He carries out multidisciplinary research, doing studies in Missense mutation and Phenotype. His multidisciplinary approach integrates Phenotype and Missense mutation in his work.
In his work, Internal medicine is strongly intertwined with Disease, which is a subfield of Pathology. His Internal medicine study often links to related topics such as Disease. In his articles, Ying-Hui Fu combines various disciplines, including Genetics and Mutation. He merges many fields, such as Gene and Molecular biology, in his writings. He combines Molecular biology and Gene in his research. He connects Neuroscience with Cell biology in his study. His work blends Cell biology and Neuroscience studies together. In his works, he conducts interdisciplinary research on Circadian rhythm and Circadian clock. Ying-Hui Fu applies his multidisciplinary studies on Circadian clock and Circadian rhythm in his research.
Ying-Hui Fu incorporates Operating system and Sleep (system call) in his studies. In his study, he carries out multidisciplinary Sleep (system call) and Operating system research. He combines Neuroscience and Behavioural genetics in his research. In his works, he performs multidisciplinary study on Behavioural genetics and Molecular genetics. He connects Molecular genetics with Human genetics in his research. He combines Genetics and Cell biology in his studies. Ying-Hui Fu combines Cell biology and Gene in his studies. He performs integrative study on Gene and Evolutionary biology. He combines Evolutionary biology and Genetics in his research.
Ying-Hui Fu integrates many fields, such as Operating system and Sleep (system call), in his works. In his works, he performs multidisciplinary study on Sleep (system call) and Operating system. His study connects Adrenergic receptor and Genetics. As part of his studies on Adrenergic receptor, he often connects relevant areas like Receptor. His Genetics research extends to the thematically linked field of Receptor. He regularly ties together related areas like Circadian rhythm in his Neuroscience studies. He integrates several fields in his works, including Circadian rhythm and Chronotype. His research is interdisciplinary, bridging the disciplines of Neuroscience and Chronotype. Ying-Hui Fu brings together Circadian clock and Cryptochrome to produce work in his papers.
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Identification of a gene (FMR-1) containing a CGG repeat coincident with a breakpoint cluster region exhibiting length variation in fragile X syndrome
Annemiske J.M.H. Verkerk;Maura Pieretti;James S. Sutcliffe;Ying-Hui Fu.
Cell (1991)
Candidate gene for the chromosome 1 familial Alzheimer's disease locus
Ephrat Levy-Lahad;Wilma Wasco;Parvoneh Poorkaj;Donna M. Romano.
Science (1995)
Variation of the CGG repeat at the fragile X site results in genetic instability: resolution of the Sherman paradox.
Ying-Hui Fu;Derek P.A. Kuhl;Antonio Pizzuti;Maura Pieretti.
Cell (1991)
Positional Cloning of the Werner's Syndrome Gene
Chang En Yu;Junko Oshima;Ying Hui Fu;Ellen M. Wijsman.
Science (1996)
An unstable triplet repeat in a gene related to myotonic muscular dystrophy.
Y. H. Fu;A. Pizzuti;R. G. Fenwick;J. King.
Science (1992)
An hPer2 Phosphorylation Site Mutation in Familial Advanced Sleep Phase Syndrome
Kong L. Toh;Christopher R. Jones;Yan He;Erik J. Eide.
Science (2001)
Absence of expression of the FMR-1 gene in fragile X syndrome.
Maura Pieretti;Fuping Zhang;Ying-Hui Fu;Stephen T. Warren.
Cell (1991)
Mutations in Kir2.1 cause the developmental and episodic electrical phenotypes of Andersen's syndrome
Nikki M. Plaster;Rabi Tawil;Martin Tristani-Firouzi;Sonia Canún.
Cell (2001)
Bone dysplasia sclerosteosis results from loss of the SOST gene product, a novel cystine knot-containing protein
Mary E. Brunkow;Jessica C. Gardner;Jeff Van Ness;Bryan W. Paeper.
American Journal of Human Genetics (2001)
Functional consequences of a CKIδ mutation causing familial advanced sleep phase syndrome
Ying Xu;Quasar S. Padiath;Robert E. Shapiro;Christopher R. Jones.
Nature (2005)
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