His main research concerns Cancer research, Pathology, DNA methylation, Molecular biology and Carcinogenesis. His Cancer research study integrates concerns from other disciplines, such as Promoter, Gene silencing, Epigenetics and Cadherin. The various areas that Yae Kanai examines in his Pathology study include Mutation and Pancreatic disease, Pancreas.
His DNA methylation study combines topics in areas such as HCCS and Methylation. The Molecular biology study combines topics in areas such as Messenger RNA, Gene expression, Gene and Methyltransferase. His work carried out in the field of Carcinoma brings together such families of science as Cancer, Cell and Lymphatic system.
Yae Kanai mostly deals with Pathology, Cancer research, DNA methylation, Cancer and Internal medicine. His Pathology study incorporates themes from Pancreatic disease, Pancreas and Adenocarcinoma. His work deals with themes such as Gene expression, Gene and DNA, which intersect with Cancer research.
His DNA methylation research incorporates themes from Carcinogenesis, Molecular biology, Methylation and Epigenetics. Yae Kanai focuses mostly in the field of Molecular biology, narrowing it down to matters related to DNA methyltransferase and, in some cases, DNMT1. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology, Endocrinology and Oncology.
His primary areas of study are Cancer research, Pathology, DNA methylation, Internal medicine and Epigenetics. His study in Cancer research is interdisciplinary in nature, drawing from both Intrahepatic Cholangiocarcinoma, Immunology, Organoid, Epigenomics and microRNA. His Pathology research is multidisciplinary, relying on both Tumor microenvironment and Cancer.
His DNA methylation research is multidisciplinary, incorporating perspectives in Carcinogenesis and Molecular biology. His biological study spans a wide range of topics, including Gastroenterology, Endocrinology, Oncology and Mutation. Yae Kanai has included themes like Bisulfite sequencing and Methylation in his Epigenetics study.
His main research concerns Cancer research, Pathology, DNA methylation, Carcinogenesis and Gene. His Cancer research research includes themes of Cell culture, Intrahepatic Cholangiocarcinoma, Immunology, Organoid and Promoter. His research integrates issues of Mutation, KRAS, Gastroenterology and GNAS complex locus in his study of Pathology.
His DNA methylation research is multidisciplinary, relying on both Molecular biology and Carcinoma. His Carcinogenesis research focuses on Epigenetics and how it relates to Glycolysis. His CpG site research also works with subjects such as
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Silencing of the E-cadherin invasion-suppressor gene by CpG methylation in human carcinomas.
Kenta Yoshiura;Yae Kanai;Atsushi Ochiai;Yutaka Shimoyama.
Proceedings of the National Academy of Sciences of the United States of America (1995)
Cell adhesion system and human cancer morphogenesis
Setsuo Hirohashi;Yae Kanai.
Cancer Science (2003)
Immune cell infiltration as an indicator of the immune microenvironment of pancreatic cancer.
Y Ino;R Yamazaki-Itoh;K Shimada;M Iwasaki.
British Journal of Cancer (2013)
FOXP3+ regulatory T cells affect the development and progression of hepatocarcinogenesis
Noritoshi Kobayashi;Nobuyoshi Hiraoka;Wataru Yamagami;Hidenori Ojima.
Clinical Cancer Research (2007)
E-cadherin gene mutations in human gastric carcinoma cell lines.
Tatsuya Oda;Yae Kanai;Tsukasa Oyama;Kenta Yoshiura.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Increased DNA methyltransferase 1 (DNMT1) protein expression correlates significantly with poorer tumor differentiation and frequent DNA hypermethylation of multiple CpG islands in gastric cancers.
Tsuyoshi Etoh;Yae Kanai;Saori Ushijima;Tohru Nakagawa.
American Journal of Pathology (2004)
A Truncated β-Catenin Disrupts the Interaction between E-Cadherin and α-Catenin: A Cause of Loss of Intercellular Adhesiveness in Human Cancer Cell Lines
Tsukasa Oyama;Yae Kanai;Atsushi Ochiai;Shingo Akimoto.
Cancer Research (1994)
Genetic instability and aberrant DNA methylation in chronic hepatitis and cirrhosis--A comprehensive study of loss of heterozygosity and microsatellite instability at 39 loci and DNA hypermethylation on 8 CpG islands in microdissected specimens from patients with hepatocellular carcinoma.
Yutaka Kondo;Y. A. E. Kanai;Michiie Sakamoto;Masashi Mizokami.
Hepatology (2000)
Expression of mRNA for DNA methyltransferases and methyl-CpG-binding proteins and DNA methylation status on CpG islands and pericentromeric satellite regions during human hepatocarcinogenesis.
Yoshimasa Saito;Yoshimasa Saito;Yae Kanai;Michiie Sakamoto;Hidetsugu Saito.
Hepatology (2001)
The E-cadherin gene is silenced by CpG methylation in human hepatocellular carcinomas
Yae Kanai;Saori Ushijima;Ai Min Hui;Atsushi Ochiai.
International Journal of Cancer (1997)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
National Cancer Research Institute, UK
Keio University
National Cancer Center
University of Tokyo
National Cancer Research Institute, UK
University of Tokyo
MIT
Kyoto University
National Defense Medical College
Tokyo Medical and Dental University
University of Kent
Peking University
Samsung (South Korea)
Virginia Tech
Tsinghua University
Peking University
University of Michigan–Ann Arbor
University of Helsinki
University of Massachusetts Medical School
University of Manchester
Met Office
University of California, San Francisco
Vanderbilt University
University of California, San Diego
Columbia University
University of Lausanne