Prostate cancer, Cancer research, Cancer, Prostate and Internal medicine are his primary areas of study. His research integrates issues of Single-nucleotide polymorphism and Pathology in his study of Prostate cancer. He has researched Cancer research in several fields, including DNA methylation, Carcinogenesis, Gene, Molecular biology and Chromoplexy.
His Cancer research is multidisciplinary, relying on both Genome-wide association study and PTEN. He usually deals with Prostate and limits it to topics linked to Risk factor and Epidemiology. His Internal medicine study combines topics in areas such as Endocrinology and Oncology.
William B. Isaacs mainly focuses on Prostate cancer, Internal medicine, Prostate, Oncology and Cancer research. He interconnects Single-nucleotide polymorphism, Genotype and Pathology in the investigation of issues within Prostate cancer. His Internal medicine research includes themes of Endocrinology and Gynecology.
As a member of one scientific family, William B. Isaacs mostly works in the field of Prostate, focusing on Tumor suppressor gene and, on occasion, Chromosome. His Oncology study combines topics from a wide range of disciplines, such as Prostatectomy, Germline mutation, Germline, Disease and Risk factor. His biological study spans a wide range of topics, including DNA methylation, Carcinogenesis, Gene, Chromoplexy and Androgen receptor.
His primary areas of study are Prostate cancer, Internal medicine, Oncology, Prostate and Cancer research. His Prostate cancer study deals with the bigger picture of Cancer. William B. Isaacs has researched Oncology in several fields, including Odds ratio, Prostatectomy, Disease and Genetic testing.
William B. Isaacs combines subjects such as Gastroenterology, Metastasis, Immunology and Pathology with his study of Prostate. His Cancer research research is multidisciplinary, incorporating elements of DNA methylation, Oncogene, Lymph node, Receptor and Androgen receptor. In his study, Genetic predisposition is inextricably linked to Gynecology, which falls within the broad field of Single-nucleotide polymorphism.
William B. Isaacs mostly deals with Prostate cancer, Oncology, Internal medicine, Prostate and Cancer research. His Prostate cancer study introduces a deeper knowledge of Cancer. His work on Primary Gleason Pattern as part of general Cancer research is often related to MSH2, thus linking different fields of science.
His Oncology research is multidisciplinary, relying on both Germline mutation, Case-control study, Confidence interval, Proportional hazards model and Genetic testing. His Prostate research includes themes of Tumor microenvironment, Metastasis and Transplantation. His Cancer research research includes elements of Missense mutation, MiRNA binding, Microsatellite instability and Androgen receptor.
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Overexpression of Hypoxia-inducible Factor 1α in Common Human Cancers and Their Metastases
Hua Zhong;Angelo M. De Marzo;Erik Laughner;Michael Lim.
Cancer Research (1999)
AR-V7 and resistance to enzalutamide and abiraterone in prostate cancer.
Emmanuel S. Antonarakis;Changxue Lu;Hao Wang;Brandon Luber.
The New England Journal of Medicine (2014)
Mutations of the VHL tumour suppressor gene in renal carcinoma
J.R. Gnarra;K. Tory;Y. Weng;L. Schmidt.
Nature Genetics (1994)
Inflammation in prostate carcinogenesis
Angelo M. De Marzo;Angelo M. De Marzo;Elizabeth A. Platz;Siobhan Sutcliffe;Jianfeng Xu.
Nature Reviews Cancer (2007)
DD3::A New Prostate-specific Gene, Highly Overexpressed in Prostate Cancer
M.J.G. Bussemakers;A. van Bokhoven;G.W.C.T. Verhaegh;F.P. Smit.
Cancer Research (1999)
Pan-cancer analysis of whole genomes
Peter J. Campbell;Gad Getz;Jan O. Korbel;Joshua M. Stuart.
The evolutionary history of lethal metastatic prostate cancer.
Gunes Gundem;Peter Van Loo;Peter Van Loo;Peter Van Loo;Barbara Kremeyer;Ludmil B. Alexandrov.
Ligand-Independent Androgen Receptor Variants Derived from Splicing of Cryptic Exons Signify Hormone-Refractory Prostate Cancer
Rong Hu;Thomas A. Dunn;Shuanzeng Wei;Sumit Isharwal.
Cancer Research (2009)
E-Cadherin Expression Is Silenced by DNA Hypermethylation in Human Breast and Prostate Carcinomas
Jeremy R. Graff;James G. Herman;Rena G. Lapidus;Hemi Chopra.
Cancer Research (1995)
REVEL: An Ensemble Method for Predicting the Pathogenicity of Rare Missense Variants
Nilah M M. Ioannidis;Joseph H H. Rothstein;Joseph H H. Rothstein;Vikas Pejaver;Sumit Middha.
American Journal of Human Genetics (2016)
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