What is she best known for?
The fields of study she is best known for:
Her primary areas of investigation include Poly ADP ribose polymerase, Molecular biology, DNA damage, DNA repair and Base excision repair.
Her Poly ADP ribose polymerase research focuses on Poly glycohydrolase in particular.
Her DNA damage research includes themes of DNA ligase and Homologous recombination.
When carried out as part of a general DNA repair research project, her work on Nucleotide excision repair is frequently linked to work in XRCC1, therefore connecting diverse disciplines of study.
Her research in the fields of DNA repair protein XRCC4 overlaps with other disciplines such as DNA polymerase delta.
The concepts of her NAD+ kinase study are interwoven with issues in FOXO1, Insulin, Insulin resistance and Cell biology.
Her most cited work include:
- Poly(ADP-ribose): novel functions for an old molecule. (1477 citations)
- XRCC1 Is Specifically Associated with Poly(ADP-Ribose) Polymerase and Negatively Regulates Its Activity following DNA Damage (813 citations)
- PARP-2, A Novel Mammalian DNA Damage-dependent Poly(ADP-ribose) Polymerase (597 citations)
What are the main themes of her work throughout her whole career to date?
Poly ADP ribose polymerase, Molecular biology, DNA damage, DNA repair and Cell biology are her primary areas of study.
In most of her Poly ADP ribose polymerase studies, her work intersects topics such as Chromatin.
In her research on the topic of Molecular biology, Poly glycohydrolase is strongly related with PARG.
Valérie Schreiber has included themes like Replication protein A and DNA ligase in her DNA damage study.
Valérie Schreiber has researched DNA repair in several fields, including PARP1 and Proliferating cell nuclear antigen.
Her research investigates the connection between Cell biology and topics such as NAD+ kinase that intersect with issues in Sirtuin 1.
She most often published in these fields:
- Poly ADP ribose polymerase (65.00%)
- Molecular biology (41.25%)
- DNA damage (37.50%)
What were the highlights of her more recent work (between 2012-2019)?
- Poly ADP ribose polymerase (65.00%)
- DNA repair (33.75%)
- DNA damage (37.50%)
In recent papers she was focusing on the following fields of study:
Valérie Schreiber mostly deals with Poly ADP ribose polymerase, DNA repair, DNA damage, Cell biology and Molecular biology.
The study incorporates disciplines such as Cancer research, Thymocyte and Synthetic lethality in addition to Poly ADP ribose polymerase.
Her DNA repair research entails a greater understanding of Biochemistry.
The DNA damage study combines topics in areas such as Interleukin 21 and Homeostasis.
Her study in Cell biology is interdisciplinary in nature, drawing from both Programmed cell death and Homologous recombination.
Her Molecular biology research is multidisciplinary, incorporating elements of Chromatin, Pericentric heterochromatin, PARG and DNA methylation.
Between 2012 and 2019, her most popular works were:
- Poly(ADP-ribose) polymerases in double-strand break repair: focus on PARP1, PARP2 and PARP3 (175 citations)
- PARP3 affects the relative contribution of homologous recombination and nonhomologous end-joining pathways (63 citations)
- Parp-2 is required to maintain hematopoiesis following sublethal γ-irradiation in mice (48 citations)
In her most recent research, the most cited papers focused on:
Valérie Schreiber mainly investigates Cell biology, Homologous recombination, Poly ADP ribose polymerase, DNA repair and DNA damage.
Her Cell biology research includes elements of Hemolysis and Downregulation and upregulation.
The various areas that Valérie Schreiber examines in her Homologous recombination study include Molecular biology, PARP1 and Replication protein A.
Her Molecular biology research incorporates elements of Ku70, Genotoxic Stress, PARG, DNA replication and DNA End-Joining Repair.
Her DNA repair study combines topics from a wide range of disciplines, such as Cancer research, Bioinformatics, Progenitor cell, Apoptosis and Blood cell.
Her work deals with themes such as Haematopoiesis, Stem cell and Programmed cell death, which intersect with DNA damage.
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