What is he best known for?
The fields of study he is best known for:
Sander M. Houten spends much of his time researching Internal medicine, Endocrinology, Biochemistry, Insulin resistance and Mitochondrion.
His research links Farnesoid X receptor with Internal medicine.
His studies in Farnesoid X receptor integrate themes in fields like Receptor and Small heterodimer partner.
His Endocrinology research is multidisciplinary, incorporating elements of Nuclear receptor and Cytokine.
The concepts of his Insulin resistance study are interwoven with issues in FOXO1, Metabolic syndrome and NAD+ kinase.
His research integrates issues of Acetyl-CoA, Carnitine and Homeostasis in his study of Mitochondrion.
His most cited work include:
- Bile acids induce energy expenditure by promoting intracellular thyroid hormone activation (1451 citations)
- Bile acids lower triglyceride levels via a pathway involving FXR, SHP, and SREBP-1c. (899 citations)
- Specific SIRT1 Activation Mimics Low Energy Levels and Protects against Diet-Induced Metabolic Disorders by Enhancing Fat Oxidation (611 citations)
What are the main themes of his work throughout his whole career to date?
Internal medicine, Endocrinology, Biochemistry, Beta oxidation and Insulin resistance are his primary areas of study.
Adipose tissue, Lipotoxicity, Knockout mouse, Myopathy and Carbohydrate metabolism are among the areas of Internal medicine where he concentrates his study.
His work on Fatty acid expands to the thematically related Endocrinology.
His Biochemistry and Enzyme, Peroxisome, Mitochondrion, Metabolism and Dehydrogenase investigations all form part of his Biochemistry research activities.
His Beta oxidation study frequently links to other fields, such as Acyl CoA dehydrogenase.
Sander M. Houten studies Glucose homeostasis which is a part of Insulin resistance.
He most often published in these fields:
- Internal medicine (52.11%)
- Endocrinology (49.47%)
- Biochemistry (38.95%)
What were the highlights of his more recent work (between 2018-2021)?
- Biochemistry (38.95%)
- Internal medicine (52.11%)
- Endocrinology (49.47%)
In recent papers he was focusing on the following fields of study:
Biochemistry, Internal medicine, Endocrinology, DHTKD1 and Mitochondrion are his primary areas of study.
His work in the fields of Biochemistry, such as Enzyme, Dehydrogenase, Lysine and Metabolism, intersects with other areas such as Saccharopine.
The study incorporates disciplines such as Haploinsufficiency and Glucose lowering in addition to Internal medicine.
His Endocrinology research incorporates themes from Glutamine and Long-chain acyl-CoA dehydrogenase.
His studies deal with areas such as Genetics, High-throughput screening, BCKDHB, Messenger RNA and Exon as well as DHTKD1.
Sander M. Houten studied Mitochondrion and Peroxisome that intersect with Carnitine, Orchiectomy, Acute kidney injury and Transcriptome.
Between 2018 and 2021, his most popular works were:
- Peroxisomes can oxidize medium- and long-chain fatty acids through a pathway involving ABCD3 and HSD17B4 (24 citations)
- PLPHP deficiency: clinical, genetic, biochemical, and mechanistic insights (22 citations)
- Intestinal Inflammation Modulates the Expression of ACE2 and TMPRSS2 and Potentially Overlaps With the Pathogenesis of SARS-CoV-2-related Disease. (16 citations)
In his most recent research, the most cited papers focused on:
Sander M. Houten mostly deals with Mitochondrion, Biochemistry, Peroxisome, Carnitine and Internal medicine.
His work on Dehydrogenase, Dihydrolipoamide dehydrogenase, Oxidoreductase and OGDH is typically connected to Glutaric Acidemia Type 1 as part of general Biochemistry study, connecting several disciplines of science.
His Peroxisome study incorporates themes from Metabolite, Beta oxidation, Metabolism and Inner mitochondrial membrane.
Sander M. Houten interconnects Gene, Long-chain acyl-CoA dehydrogenase and Carnitine biosynthesis in the investigation of issues within Carnitine.
His Internal medicine research includes elements of Endocrinology and Allele.
His Endocrinology research is multidisciplinary, relying on both Haploinsufficiency and Toxicity.
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