What is she best known for?
The fields of study she is best known for:
Ulrike Müller mostly deals with Amyloid precursor protein, Cell biology, APLP1, APLP2 and Biochemistry.
Her Amyloid precursor protein research is multidisciplinary, incorporating elements of Amyloid beta, Sphingomyelin, Immunology, Neuroscience and Amyloid.
While the research belongs to areas of Cell biology, Ulrike Müller spends her time largely on the problem of Genetics, intersecting her research to questions surrounding Commissure, Corpus callosum and Forebrain.
Her APLP1 research incorporates themes from Gene family, Mutant, Long-term potentiation, Molecular biology and Neuromuscular junction.
Her APLP2 study integrates concerns from other disciplines, such as Synaptic plasticity, Neurotransmission and Anatomy.
Her Biochemistry research includes themes of Presenilin and Amyloid precursor protein secretase.
Her most cited work include:
- Functional role of type I and type II interferons in antiviral defense. (2095 citations)
- Aggressive Behavior and Altered Amounts of Brain Serotonin and Norepinephrine in Mice Lacking MAOA (1017 citations)
- GlyR alpha3: an essential target for spinal PGE2-mediated inflammatory pain sensitization. (481 citations)
What are the main themes of her work throughout her whole career to date?
Amyloid precursor protein, Cell biology, Neuroscience, APLP2 and Biochemistry are her primary areas of study.
Ulrike Müller has included themes like Synaptic plasticity, Long-term potentiation and Presenilin in her Amyloid precursor protein study.
Her work carried out in the field of Long-term potentiation brings together such families of science as Hippocampal formation, Hippocampus and Dentate gyrus.
Her research in the fields of Neurogenesis overlaps with other disciplines such as Glycine receptor.
Ulrike Müller has researched Neuroscience in several fields, including Alzheimer's disease and Genetically modified mouse.
Her APLP2 study combines topics in areas such as Genetics, Mutant, APLP1 and Amyloid.
She most often published in these fields:
- Amyloid precursor protein (41.84%)
- Cell biology (32.65%)
- Neuroscience (27.55%)
What were the highlights of her more recent work (between 2013-2021)?
- Amyloid precursor protein (41.84%)
- Neuroscience (27.55%)
- Cell biology (32.65%)
In recent papers she was focusing on the following fields of study:
Her primary areas of investigation include Amyloid precursor protein, Neuroscience, Cell biology, Synaptic plasticity and Long-term potentiation.
APLP2 and Amyloid precursor protein secretase are subfields of Amyloid precursor protein in which her conducts study.
The concepts of her Neuroscience study are interwoven with issues in In situ hybridization, Downregulation and upregulation, Fragile X syndrome and Function.
Her Cell biology research is multidisciplinary, relying on both APLP1, Biochemistry, Neurodegeneration, Presenilin and In vivo.
She focuses mostly in the field of Synaptic plasticity, narrowing it down to topics relating to Neurotrophin and, in certain cases, Microglia, Atrophy and TREM2.
Her work deals with themes such as Dentate gyrus, Forebrain and Ectodomain, which intersect with Long-term potentiation.
Between 2013 and 2021, her most popular works were:
- η-Secretase processing of APP inhibits neuronal activity in the hippocampus (228 citations)
- Not just amyloid: physiological functions of the amyloid precursor protein family (213 citations)
- Tumour-cell-induced endothelial cell necroptosis via death receptor 6 promotes metastasis (172 citations)
In her most recent research, the most cited papers focused on:
Ulrike Müller spends much of her time researching Amyloid precursor protein, Neuroscience, Cell biology, Amyloid precursor protein secretase and APLP2.
Her Amyloid precursor protein research includes elements of Dendritic spine, Parietal lobe, Anatomy, Synapse and Posterior parietal cortex.
Her study in Neuroscience is interdisciplinary in nature, drawing from both Synaptic plasticity, Long-term potentiation, Downregulation and upregulation and Function.
Her research investigates the connection with Cell biology and areas like Gene expression which intersect with concerns in Signal transducing adaptor protein, Neprilysin, Protein subunit and Regulation of gene expression.
Her study focuses on the intersection of Amyloid precursor protein secretase and fields such as Neuroprotection with connections in the field of P3 peptide, Biochemistry of Alzheimer's disease, PI3K/AKT/mTOR pathway, Molecular biology and Neuron death.
The APLP2 study which covers Presenilin that intersects with Gene knockdown, Neurodegeneration, Transcription factor and Mitochondrion.
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