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Takashi Kobayashi

Takashi Kobayashi

D-Index & Metrics

Biology and Biochemistry

D-Index
54
Citations
11507
World Ranking
15549
National Ranking
1104

Overview

Takashi Kobayashi is a researcher affiliated with Oita University in Japan. Their scientific contributions primarily span the fields of Medicine, Biochemistry, Genetics and Molecular Biology, and Immunology and Microbiology. The researcher's subfields of focus include Immunology, Molecular Biology, Oncology, Infectious Diseases, and Surgery.

The scope of Kobayashi's research topics encompasses several areas of immunology and molecular biology, with particular attention to T-cell and B-cell Immunology, Immune Cell Function and Interaction, RNA modifications and cancer, and pathways involving IL-33, ST2, and innate lymphoid cells (ILC). Additionally, Kobayashi's work addresses COVID-19 Clinical Research Studies, Gut microbiota and health, and NF-κB Signaling Pathways.

Throughout their career, Kobayashi has published papers in various scientific venues. They have frequently published in:

  • Biochemical and Biophysical Research Communications
  • Genes to Cells
  • BioNanoScience
  • Internal Medicine
  • International Immunology

Recent notable papers by Kobayashi include:

  • The Roles of IL-17, IL-21, and IL-23 in the Helicobacter pylori Infection and Gastrointestinal Inflammation: A Review, 2021, Toxins
  • DUSP4 is involved in the enhanced proliferation and survival of DUSP4-overexpressing cancer cells, 2020, Biochemical and Biophysical Research Communications
  • The Lethal(2)-Essential-for-Life [L(2)EFL] Gene Family Modulates Dengue Virus Infection in Aedes aegypti, 2020, International Journal of Molecular Sciences
  • Excess mortality in COVID-19-affected solid organ transplant recipients across the pandemic, 2024, American Journal of Transplantation
  • Development of a portable reverse transcription loop-mediated isothermal amplification system to detect the E1 region of Chikungunya virus in a cost-effective manner, 2020, Genes to Cells

Kobayashi has collaborated extensively with several coauthors. Frequent collaborators include:

  • Sotaro Ozaka
  • Naganori Kamiyama
  • Nozomi Sachi
  • Kazunari Murakami
  • Yasuhiro Soga

Best Publications

  • Distinct molecular mechanism for initiating TRAF6 signalling.

    Hong Ye;Joseph R. Arron;Joseph R. Arron;Betty Lamothe;Maurizio Cirilli

  • Histamine regulates T-cell and antibody responses by differential expression of H1 and H2 receptors

    Marek Jutel;Takeshi Watanabe;Sven Klunker;Mübeccel Akdis

  • Foxp3 Inhibits RORγt-mediated IL-17A mRNA Transcription through Direct Interaction with RORγt

    Kenji Ichiyama;Hideyuki Yoshida;Yu Wakabayashi;Takatoshi Chinen

  • Osteoclast differentiation independent of the TRANCE-RANK-TRAF6 axis.

    Nacksung Kim;Yuho Kadono;Masamichi Takami;Junwon Lee

  • IL-1β and TNFα-initiated IL-6–STAT3 pathway is critical in mediating inflammatory cytokines and RANKL expression in inflammatory arthritis

    Tomoaki Mori;Takeshi Miyamoto;Hideyuki Yoshida;Mayoko Asakawa

  • Impaired locomotor activity and exploratory behavior in mice lacking histamine H1 receptors

    Isao Inoue;Kazuhiko Yanai;Daisuke Kitamura;Ichiro Taniuchi

  • TRAF6 is a critical factor for dendritic cell maturation and development.

    Takashi Kobayashi;Patrick T Walsh;Matthew C Walsh;Kendra M Speirs

  • Loss of SOCS3 in the liver promotes fibrosis by enhancing STAT3-mediated TGF-β1 production

    H Ogata;T Chinen;T Yoshida;T Yoshida;I Kinjyo

  • Medullary thymic epithelial cells expressing Aire represent a unique lineage derived from cells expressing claudin.

    Yoko Hamazaki;Harumi Fujita;Takashi Kobayashi;Yongwon Choi

  • Involvement of Th17 cells and the effect of anti-IL-6 therapy in autoimmune uveitis

    Takeru Yoshimura;Koh Hei Sonoda;Nobuyuki Ohguro;Yoshiyuki Ohsugi

  • Loss of Suppressor of Cytokine Signaling 1 in Helper T Cells Leads to Defective Th17 Differentiation by Enhancing Antagonistic Effects of IFN-γ on STAT3 and Smads

    Kentaro Tanaka;Kenji Ichiyama;Masayuki Hashimoto;Hideyuki Yoshida

  • Deletion of the SOCS3 gene in liver parenchymal cells promotes hepatitis-induced hepatocarcinogenesis.

    Hisanobu Ogata;Takashi Kobayashi;Takatoshi Chinen;Hiromi Takaki

  • Loss of SOCS3 in T helper cells resulted in reduced immune responses and hyperproduction of interleukin 10 and transforming growth factor–β1

    Ichiko Kinjyo;Hiromasa Inoue;Shinjiro Hamano;Satoru Fukuyama

  • IFNγ-dependent, spontaneous development of colorectal carcinomas in SOCS1-deficient mice

    Toshikatsu Hanada;Toshikatsu Hanada;Takashi Kobayashi;Takatoshi Chinen;Kazuko Saeki

  • TRAF6 is a T cell-intrinsic negative regulator required for the maintenance of immune homeostasis.

    Carolyn G King;Takashi Kobayashi;Pedro J Cejas;Taesoo Kim

  • Abnormal functional and morphological regulation of the gastric mucosa in histamine H2 receptor–deficient mice

    Takashi Kobayashi;Shunsuke Tonai;Yasunobu Ishihara;Ritsuko Koga

  • STAT6 Inhibits TGF-β1-mediated Foxp3 Induction through Direct Binding to the Foxp3 Promoter, Which Is Reverted by Retinoic Acid Receptor

    Hiromi Takaki;Kenji Ichiyama;Keiko Koga;Takatoshi Chinen

  • Regulation of the Subcellular Localization of Tumor Necrosis Factor Receptor–associated Factor (TRAF)2 by TRAF1 Reveals Mechanisms of TRAF2 Signaling

    Joseph R. Arron;Yael Pewzner-Jung;Matthew C. Walsh;Takashi Kobayashi

  • The role of TRAF6 in signal transduction and the immune response.

    Takashi Kobayashi;Matthew C. Walsh;Yongwon Choi

  • Loss of mammalian Sprouty2 leads to enteric neuronal hyperplasia and esophageal achalasia

    Takaharu Taketomi;Daigo Yoshiga;Koji Taniguchi;Takashi Kobayashi

Frequent Co-Authors

Akihiko Yoshimura
Akihiko Yoshimura Keio University
Osamu Ogawa
Osamu Ogawa Kyoto University
Yongwon Choi
Yongwon Choi University of Pennsylvania
Mikio Shoji
Mikio Shoji Hirosaki University
Hideo Takeuchi
Hideo Takeuchi Tohoku University
Christopher A. Hunter
Christopher A. Hunter University of Pennsylvania
Laurence A. Turka
Laurence A. Turka Harvard University
Hiroyuki Aburatani
Hiroyuki Aburatani University of Tokyo
Takeshi Miyamoto
Takeshi Miyamoto Kumamoto University
Lothar Hennighausen
Lothar Hennighausen National Institutes of Health

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