Steven J. Collins mostly deals with Disease, Retinoic acid, Retinoic acid receptor, Pathology and Cancer research. Steven J. Collins is interested in Acute promyelocytic leukemia, which is a branch of Retinoic acid. His Acute promyelocytic leukemia study integrates concerns from other disciplines, such as Leukemia and Mutant.
In his work, Cellular differentiation, Receptor, Tretinoin, Proteomics and RNA is strongly intertwined with Cell biology, which is a subfield of Retinoic acid receptor. Steven J. Collins has included themes like Myeloid, Immunology and Promyelocyte in his Cellular differentiation study. His Pathology research focuses on Central nervous system disease and how it relates to Magnetic resonance imaging, Fluid-attenuated inversion recovery and Electroencephalography.
His primary scientific interests are in Disease, Pathology, Biochemistry, Cell biology and Retinoic acid. His study in Disease is interdisciplinary in nature, drawing from both Pediatrics and Virology. As part of his studies on Pathology, he often connects relevant areas like Immunology.
Steven J. Collins combines topics linked to Receptor with his work on Cell biology. Steven J. Collins interconnects Cancer research and Cellular differentiation in the investigation of issues within Retinoic acid. His study looks at the relationship between Cellular differentiation and topics such as Cell culture, which overlap with Molecular biology.
Steven J. Collins mainly focuses on Disease, Cerebrospinal fluid, Internal medicine, Biomarker and Alzheimer's disease. To a larger extent, he studies Pathology with the aim of understanding Disease. His study explores the link between Cerebrospinal fluid and topics such as Csf biomarkers that cross with problems in β amyloid.
His Internal medicine research includes themes of Endocrinology and Oncology. His Biomarker study combines topics in areas such as Clinical endpoint, Neurodegeneration and Bioinformatics. The Alzheimer's disease study combines topics in areas such as Neuropathology and Dementia, Frontotemporal dementia, Cognitive decline.
His primary areas of investigation include Alzheimer's disease, Biomarker, Cerebrospinal fluid, PRNP and Pathology. His Biomarker study combines topics from a wide range of disciplines, such as Neurodegeneration, Oncology and Cognitive decline. Steven J. Collins usually deals with Cerebrospinal fluid and limits it to topics linked to Dementia and Schizophrenia, C-reactive protein and Bioinformatics.
His research integrates issues of Cell, Cancer research, Cell culture and Plasma protein binding in his study of PRNP. His work in the fields of Pathology, such as Amyloid, Amyloid beta and PrPSc Proteins, intersects with other areas such as Angiopathy. Steven J. Collins has researched Disease in several fields, including Penetrance, Asymptomatic and Surrogate endpoint.
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The HL-60 promyelocytic leukemia cell line: proliferation, differentiation, and cellular oncogene expression
Steven J. Collins.
Integrated Genomic and Proteomic Analyses of Gene Expression in Mammalian Cells
Qiang Tian;Serguei B. Stepaniants;Mao Mao;Lee Weng.
Molecular & Cellular Proteomics (2004)
Updated clinical diagnostic criteria for sporadic Creutzfeldt-Jakob disease
I. Zerr;K. Kallenberg;D. M. Summers;C. Romero.
Normal functional characteristics of cultured human promyelocytic leukemia cells (HL-60) after induction of differentiation by dimethylsulfoxide.
S J Collins;F W Ruscetti;R E Gallagher;R C Gallo.
Journal of Experimental Medicine (1979)
Terminal differentiation of human promyelocytic leukemic cells in primary culture in response to retinoic acid.
T. R. Breitman;Steven J. Collins;B. R. Keene.
An exceptionally conserved transcriptional repressor, CTCF, employs different combinations of zinc fingers to bind diverged promoter sequences of avian and mammalian c-myc oncogenes.
G N Filippova;S Fagerlie;E M Klenova;C Myers.
Molecular and Cellular Biology (1996)
Iatrogenic Creutzfeldt-Jakob disease at the millennium.
P. Brown;M. Preece;J.-P. Brandel;T. Sato.
Analysis of EEG and CSF 14-3-3 proteins as aids to the diagnosis of Creutzfeldt-Jakob disease.
I. Zerr;M. Pocchiari;S. Collins;J.P. Brandel.
Ultrasensitive human prion detection in cerebrospinal fluid by real-time quaking-induced conversion
Ryuichiro Atarashi;Katsuya Satoh;Kazunori Sano;Takayuki Fuse.
Nature Medicine (2011)
Transmissible spongiform encephalopathies.
Steven J Collins;Victoria A Lawson;Colin L Masters;Colin L Masters.
The Lancet (2004)
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