D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 50 Citations 12,454 70 World Ranking 2732 National Ranking 1307

Overview

What is he best known for?

The fields of study he is best known for:

  • Immune system
  • Cytokine
  • DNA

His primary areas of investigation include Synovial fluid, Pathology, Cytokine, Molecular biology and Synovial membrane. His work carried out in the field of Pathology brings together such families of science as Inflammation, Endothelium, Growth factor and Antigen. His Cytokine research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha, Endothelial stem cell and Angiogenesis.

The various areas that he examines in his Synovial membrane study include Chemokine, Peripheral blood mononuclear cell, Macrophage and Interleukin 8. His Peripheral blood mononuclear cell research includes themes of Alpha and Macrophage inflammatory protein. His Arthritis research entails a greater understanding of Immunology.

His most cited work include:

  • Enhanced production of monocyte chemoattractant protein-1 in rheumatoid arthritis. (582 citations)
  • Vascular endothelial growth factor. A cytokine modulating endothelial function in rheumatoid arthritis. (518 citations)
  • Vascular endothelial growth factor. A cytokine modulating endothelial function in rheumatoid arthritis. (518 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Immunology, Synovial fluid, Rheumatoid arthritis, Molecular biology and Arthritis. He frequently studies issues relating to Apoptosis and Immunology. The concepts of his Synovial fluid study are interwoven with issues in Cytokine, T cell, Antigen, Pathology and Peripheral blood mononuclear cell.

His Pathology study integrates concerns from other disciplines, such as Angiogenesis and Growth factor. His biological study spans a wide range of topics, including Tumor necrosis factor alpha, Cytotoxic T cell, NFKB1 and Transfection. His work focuses on many connections between Arthritis and other disciplines, such as Macrophage, that overlap with his field of interest in CD14.

He most often published in these fields:

  • Immunology (65.42%)
  • Synovial fluid (42.99%)
  • Rheumatoid arthritis (36.45%)

What were the highlights of his more recent work (between 2005-2021)?

  • Immunology (65.42%)
  • Rheumatoid arthritis (36.45%)
  • Flip (15.89%)

In recent papers he was focusing on the following fields of study:

Richard M. Pope mainly focuses on Immunology, Rheumatoid arthritis, Flip, Arthritis and Cancer research. His research in Immunology intersects with topics in Synovial fluid, Hematopoietic stem cell transplantation and Macrophage. His studies in Rheumatoid arthritis integrate themes in fields like Dermatology and TLR2.

His Flip research incorporates elements of Signal transduction and Pathogenesis. His research combines Tumor necrosis factor alpha and Arthritis. His Cancer research study combines topics in areas such as T cell, Antigen and Small interfering RNA.

Between 2005 and 2021, his most popular works were:

  • The CD95 receptor: apoptosis revisited. (345 citations)
  • Inhibition of ADP/ATP exchange in receptor-interacting protein-mediated necrosis. (176 citations)
  • The role of interleukin-1 and the inflammasome in gout: implications for therapy. (150 citations)

In his most recent research, the most cited papers focused on:

  • Immune system
  • DNA
  • Internal medicine

His main research concerns Immunology, Cell biology, Apoptosis, CD14 and Macrophage. Richard M. Pope integrates Immunology with Purine in his study. His work in the fields of Signal transduction overlaps with other areas such as Adenosine diphosphate.

His Apoptosis research is multidisciplinary, relying on both Synovial fluid, Cancer research and Transfection, Small interfering RNA. His CD14 study combines topics from a wide range of disciplines, such as Tumor necrosis factor alpha, Toll-like receptor and Inflammation, Arthritis, Synovial membrane. His Macrophage research incorporates themes from STAT protein and Inflammatory arthritis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Vascular endothelial growth factor. A cytokine modulating endothelial function in rheumatoid arthritis.

Alisa E. Koch;Alisa E. Koch;Alisa E. Koch;Lisa A. Harlow;George K. Haines;Edward P. Amento.
Journal of Immunology (1994)

799 Citations

Enhanced production of monocyte chemoattractant protein-1 in rheumatoid arthritis.

A E Koch;S L Kunkel;S L Kunkel;L A Harlow;L A Harlow;B Johnson;B Johnson.
Journal of Clinical Investigation (1992)

791 Citations

Human abdominal aortic aneurysms. Immunophenotypic analysis suggesting an immune-mediated response.

A E Koch;G K Haines;R J Rizzo;J A Radosevich.
American Journal of Pathology (1990)

546 Citations

Apoptosis as a therapeutic tool in rheumatoid arthritis.

Richard M. Pope.
Nature Reviews Immunology (2002)

483 Citations

Macrophage inflammatory protein-1 alpha. A novel chemotactic cytokine for macrophages in rheumatoid arthritis.

A E Koch;S L Kunkel;S L Kunkel;L A Harlow;D D Mazarakis.
Journal of Clinical Investigation (1994)

443 Citations

The CD95 receptor: apoptosis revisited.

Marcus E. Peter;Ralph C. Budd;Julie Desbarats;Stephen M. Hedrick.
Cell (2007)

441 Citations

A genomewide screen in multiplex rheumatoid arthritis families suggests genetic overlap with other autoimmune diseases.

Damini Jawaheer;Michael F. Seldin;Christopher I. Amos;Wei V. Chen.
American Journal of Human Genetics (2001)

435 Citations

Synovial tissue macrophage as a source of the chemotactic cytokine IL-8.

A. E. Koch;S. L. Kunkel;J. C. Burrows;H. L. Evanoff.
Journal of Immunology (1991)

431 Citations

Expression of matrix metalloproteinase 9 (96-kd gelatinase B) in human rheumatoid arthritis

Diane Ahrens;Alisa E. Koch;Richard M. Pope;Monica Stein-Picarella.
Arthritis & Rheumatism (1996)

372 Citations

FLICE-inhibitory protein expression during macrophage differentiation confers resistance to fas-mediated apoptosis.

Harris Perlman;Harris Perlman;Lisa J. Pagliari;Lisa J. Pagliari;Constantinos Georganas;Constantinos Georganas;Toshiaki Mano.
Journal of Experimental Medicine (1999)

345 Citations

Best Scientists Citing Richard M. Pope

Alisa E. Koch

Alisa E. Koch

University of Michigan–Ann Arbor

Publications: 112

Paul P. Tak

Paul P. Tak

GlaxoSmithKline (United Kingdom)

Publications: 39

Peter K. Gregersen

Peter K. Gregersen

Feinstein Institute for Medical Research

Publications: 37

Zoltán Szekanecz

Zoltán Szekanecz

University of Debrecen

Publications: 37

Harris Perlman

Harris Perlman

Northwestern University

Publications: 31

Marcus E. Peter

Marcus E. Peter

Northwestern University

Publications: 29

Christopher D. Buckley

Christopher D. Buckley

University of Oxford

Publications: 26

Marc Feldmann

Marc Feldmann

University of Oxford

Publications: 25

Robert M. Strieter

Robert M. Strieter

University of Virginia

Publications: 25

Steven L. Kunkel

Steven L. Kunkel

University of Michigan–Ann Arbor

Publications: 24

Robert M. Plenge

Robert M. Plenge

Bristol Myers Squibb

Publications: 22

Christopher I. Amos

Christopher I. Amos

Baylor College of Medicine

Publications: 22

Wan-Uk Kim

Wan-Uk Kim

Catholic University of Korea

Publications: 21

Cornelia M. Weyand

Cornelia M. Weyand

Stanford University

Publications: 21

Guido Kroemer

Guido Kroemer

Sorbonne University

Publications: 20

Peter Vandenabeele

Peter Vandenabeele

Ghent University

Publications: 20

Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking d-index is inferred from publications deemed to belong to the considered discipline.

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