His primary areas of investigation include Synovial fluid, Pathology, Cytokine, Molecular biology and Synovial membrane. His work carried out in the field of Pathology brings together such families of science as Inflammation, Endothelium, Growth factor and Antigen. His Cytokine research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha, Endothelial stem cell and Angiogenesis.
The various areas that he examines in his Synovial membrane study include Chemokine, Peripheral blood mononuclear cell, Macrophage and Interleukin 8. His Peripheral blood mononuclear cell research includes themes of Alpha and Macrophage inflammatory protein. His Arthritis research entails a greater understanding of Immunology.
His primary areas of study are Immunology, Synovial fluid, Rheumatoid arthritis, Molecular biology and Arthritis. He frequently studies issues relating to Apoptosis and Immunology. The concepts of his Synovial fluid study are interwoven with issues in Cytokine, T cell, Antigen, Pathology and Peripheral blood mononuclear cell.
His Pathology study integrates concerns from other disciplines, such as Angiogenesis and Growth factor. His biological study spans a wide range of topics, including Tumor necrosis factor alpha, Cytotoxic T cell, NFKB1 and Transfection. His work focuses on many connections between Arthritis and other disciplines, such as Macrophage, that overlap with his field of interest in CD14.
Richard M. Pope mainly focuses on Immunology, Rheumatoid arthritis, Flip, Arthritis and Cancer research. His research in Immunology intersects with topics in Synovial fluid, Hematopoietic stem cell transplantation and Macrophage. His studies in Rheumatoid arthritis integrate themes in fields like Dermatology and TLR2.
His Flip research incorporates elements of Signal transduction and Pathogenesis. His research combines Tumor necrosis factor alpha and Arthritis. His Cancer research study combines topics in areas such as T cell, Antigen and Small interfering RNA.
His main research concerns Immunology, Cell biology, Apoptosis, CD14 and Macrophage. Richard M. Pope integrates Immunology with Purine in his study. His work in the fields of Signal transduction overlaps with other areas such as Adenosine diphosphate.
His Apoptosis research is multidisciplinary, relying on both Synovial fluid, Cancer research and Transfection, Small interfering RNA. His CD14 study combines topics from a wide range of disciplines, such as Tumor necrosis factor alpha, Toll-like receptor and Inflammation, Arthritis, Synovial membrane. His Macrophage research incorporates themes from STAT protein and Inflammatory arthritis.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Vascular endothelial growth factor. A cytokine modulating endothelial function in rheumatoid arthritis.
Alisa E. Koch;Alisa E. Koch;Alisa E. Koch;Lisa A. Harlow;George K. Haines;Edward P. Amento.
Journal of Immunology (1994)
Enhanced production of monocyte chemoattractant protein-1 in rheumatoid arthritis.
A E Koch;S L Kunkel;S L Kunkel;L A Harlow;L A Harlow;B Johnson;B Johnson.
Journal of Clinical Investigation (1992)
Human abdominal aortic aneurysms. Immunophenotypic analysis suggesting an immune-mediated response.
A E Koch;G K Haines;R J Rizzo;J A Radosevich.
American Journal of Pathology (1990)
Apoptosis as a therapeutic tool in rheumatoid arthritis.
Richard M. Pope.
Nature Reviews Immunology (2002)
Macrophage inflammatory protein-1 alpha. A novel chemotactic cytokine for macrophages in rheumatoid arthritis.
A E Koch;S L Kunkel;S L Kunkel;L A Harlow;D D Mazarakis.
Journal of Clinical Investigation (1994)
The CD95 receptor: apoptosis revisited.
Marcus E. Peter;Ralph C. Budd;Julie Desbarats;Stephen M. Hedrick.
A genomewide screen in multiplex rheumatoid arthritis families suggests genetic overlap with other autoimmune diseases.
Damini Jawaheer;Michael F. Seldin;Christopher I. Amos;Wei V. Chen.
American Journal of Human Genetics (2001)
Synovial tissue macrophage as a source of the chemotactic cytokine IL-8.
A. E. Koch;S. L. Kunkel;J. C. Burrows;H. L. Evanoff.
Journal of Immunology (1991)
Expression of matrix metalloproteinase 9 (96-kd gelatinase B) in human rheumatoid arthritis
Diane Ahrens;Alisa E. Koch;Richard M. Pope;Monica Stein-Picarella.
Arthritis & Rheumatism (1996)
FLICE-inhibitory protein expression during macrophage differentiation confers resistance to fas-mediated apoptosis.
Harris Perlman;Harris Perlman;Lisa J. Pagliari;Lisa J. Pagliari;Constantinos Georganas;Constantinos Georganas;Toshiaki Mano.
Journal of Experimental Medicine (1999)
Profile was last updated on December 6th, 2021.
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