World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
85
Citations
27573
World Ranking
3135
National Ranking
1585

Overview

Richard A. Lang is affiliated with Cincinnati Children's Hospital Medical Center in the United States. Their research is primarily situated within the fields of Neuroscience, Medicine, and Biochemistry, Genetics and Molecular Biology. Within these fields, their focused subfields include Endocrine and Autonomic Systems, Cellular and Molecular Neuroscience, Molecular Biology, Physiology, and Radiology, Nuclear Medicine and Imaging.

The scientist's work extensively explores several key topics, including Circadian rhythm and melatonin, Photoreceptor and optogenetics research, Retinal Development and Disorders, Adipose Tissue and Metabolism, Sleep and Wakefulness Research, Spaceflight effects on biology, and Neurobiology and Insect Physiology Research.

Among the notable recent publications authored or co-authored by Richard A. Lang are:

  • "Violet-light suppression of thermogenesis by opsin 5 hypothalamic neurons" (2020), published in Nature
  • "Violet light suppresses lens-induced myopia via neuropsin (OPN5) in mice" (2021), published in Proceedings of the National Academy of Sciences
  • "Adaptive Thermogenesis in Mice Is Enhanced by Opsin 3-Dependent Adipocyte Light Sensing" (2020), published in Cell Reports
  • "A dietary change to a high-fat diet initiates a rapid adaptation of the intestine" (2022), published in Cell Reports
  • "An Expanding Role for Nonvisual Opsins in Extraocular Light Sensing Physiology" (2023), published in Annual Review of Vision Science

The frequent co-authors collaborating with Richard A. Lang include Shane P. D'Souza, Shruti Vemaraju, Brian A. Upton, Ethan D. Buhr, and Kevin Zhang.

The primary venues for this scientist's publications are:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Cell Reports
  • Proceedings of the National Academy of Sciences
  • eNeuro
  • Frontiers in Physiology

Best Publications

  • In Vivo Depletion of CD11c+ Dendritic Cells Abrogates Priming of CD8+ T Cells by Exogenous Cell-Associated Antigens

    Steffen Jung;Derya Unutmaz;Phillip Wong;Gen Ichiro Sano

  • Canonical Wnt Signaling in Differentiated Osteoblasts Controls Osteoclast Differentiation

    Donald A. Glass;Peter Bialek;Jong Deok Ahn;Michael Starbuck

  • Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair

    Jeremy S. Duffield;Stuart J. Forbes;Christothea M. Constandinou;Spike Clay

  • Cbfa1-independent decrease in osteoblast proliferation, osteopenia, and persistent embryonic eye vascularization in mice deficient in Lrp5, a Wnt coreceptor

    Masaki Kato;Millan S Patel;Régis Levasseur;Ivan Lobov

  • Angiopoietin-2 displays VEGF-dependent modulation of capillary structure and endothelial cell survival in vivo

    Ivan B. Lobov;Peter C. Brooks;Richard A. Lang

  • Early eye development in vertebrates.

    Robert L. Chow;Richard A. Lang

  • A Distinct Macrophage Population Mediates Metastatic Breast Cancer Cell Extravasation, Establishment and Growth

    Binzhi Qian;Yan Deng;Jae Hong Im;Ruth J. Muschel

  • Pax6 induces ectopic eyes in a vertebrate.

    Robert L. Chow;Curtis R. Altmann;Richard A. Lang;Ali Hemmati-Brivanlou

  • WNT7b mediates macrophage-induced programmed cell death in patterning of the vasculature

    Ivan B. Lobov;Sujata Rao;Thomas J. Carroll;Jefferson E. Vallance

  • Macrophage Wnt7b is critical for kidney repair and regeneration

    Shuei Liong Lin;Bing Li;Sujata Rao;Eun Jin Yeo

  • Transgenic mice expressing a hemopoietic growth factor gene (GM-CSF) develop accumulations of macrophages, blindness, and a fatal syndrome of tissue damage

    Richard A. Lang;Donald Metcalf;R.Andrew Cuthbertson;Ian Lyons

  • Nrarp coordinates endothelial Notch and Wnt signaling to control vessel density in angiogenesis.

    Li Kun Phng;Michael Potente;Jonathan D. Leslie;Jane Babbage

  • Macrophages are required for cell death and tissue remodeling in the developing mouse eye

    Richard A. Lang;J. Michael Bishop

  • Monocyte/Macrophage Suppression in CD11b Diphtheria Toxin Receptor Transgenic Mice Differentially Affects Atherogenesis and Established Plaques

    Victoria Stoneman;Denise Braganza;Nichola Figg;John Mercer

  • Expression of a hemopoietic growth factor cDNA in a factor-dependent cell line results in autonomous growth and tumorigenicity

    Richard A. Lang;Donald Metcalf;Nicholas M. Gough;Ashley R. Dunn

  • Regulation of angiogenesis by a non-canonical Wnt - Flt1 pathway in myeloid cells

    James A. Stefater;James A. Stefater;Ian Lewkowich;Sujata Rao;Sujata Rao;Giovanni Mariggi

  • Distinct functions for Wnt/β-catenin in hair follicle stem cell proliferation and survival and interfollicular epidermal homeostasis

    Yeon Sook Choi;Yuhang Zhang;Yuhang Zhang;Mingang Xu;Yongguang Yang

  • Conditional Macrophage Ablation Demonstrates That Resident Macrophages Initiate Acute Peritoneal Inflammation

    Jean Francois Cailhier;Marina Partolina;Srilatha Vuthoori;Shengji Wu

  • FGF10 is an inducer and Pax6 a competence factor for lacrimal gland development.

    H.P. Makarenkova;M. Ito;V. Govindarajan;S.C. Faber

  • Dermal β-catenin activity in response to epidermal Wnt ligands is required for fibroblast proliferation and hair follicle initiation

    Demeng Chen;Andrew Jarrell;Canting Guo;Richard Lang

Frequent Co-Authors

Yi Zheng
Yi Zheng Cincinnati Children's Hospital Medical Center
Jeffrey W. Pollard
Jeffrey W. Pollard University of Edinburgh
Holger Gerhardt
Holger Gerhardt Max Delbrück Center for Molecular Medicine
Napoleone Ferrara
Napoleone Ferrara University of California, San Diego
Yutaka Yoshida
Yutaka Yoshida Cornell University
David R. Copenhagen
David R. Copenhagen University of California, San Francisco
Makoto Mark Taketo
Makoto Mark Taketo Kyoto University
jeffrey a whitsett
jeffrey a whitsett Cincinnati Children's Hospital Medical Center
Bart O. Williams
Bart O. Williams Van Andel Institute
Lois E. H. Smith
Lois E. H. Smith Boston Children's Hospital

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