2016 - Fellow of the Royal Society of Edinburgh
In his works, Stuart J. Forbes undertakes multidisciplinary study on Cell biology and Neuroscience. He performs integrative study on Neuroscience and Cell biology. The study of Immunology is intertwined with the study of Galectin-3 in a number of ways. His Immunology research extends to the thematically linked field of Galectin-3. He merges Pathology with Immunohistochemistry in his study. In his research, he performs multidisciplinary study on Immunohistochemistry and Pathology. His Extracellular matrix research extends to Biochemistry, which is thematically connected. His Extracellular matrix study typically links adjacent topics like Biochemistry. He performs multidisciplinary study in Fibrosis and Gene in his work.
His Immunology research is linked to Immune system and Inflammation, among other subjects. He integrates many fields in his works, including Immune system and Immunology. He is involved in relevant fields of research such as Transplantation, Fibrosis and Cirrhosis in the realm of Internal medicine. Much of his study explores Fibrosis relationship to Pathology. His study in Bone marrow extends to Pathology with its themes. He applies his multidisciplinary studies on Bone marrow and Stem cell in his research. In his study, Stuart J. Forbes carries out multidisciplinary Stem cell and Progenitor cell research. Stuart J. Forbes is investigating Cellular differentiation and Embryonic stem cell as part of his examination of Gene. Stuart J. Forbes performs multidisciplinary study in the fields of Embryonic stem cell and Gene via his papers.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair
Jeremy S. Duffield;Stuart J. Forbes;Christothea M. Constandinou;Spike Clay.
Journal of Clinical Investigation (2005)
Bone marrow contributes to renal parenchymal turnover and regeneration
Richard Poulsom;Stuart J. Forbes;Kairbaan Hodivala-Dilke;Eoin Ryan.
The Journal of Pathology (2001)
Expert consensus document: Cholangiocarcinoma: current knowledge and future perspectives consensus statement from the European Network for the Study of Cholangiocarcinoma (ENS-CCA)
Jesus M. Banales;Vincenzo Cardinale;Guido Carpino;Marco Marzioni.
Nature Reviews Gastroenterology & Hepatology (2016)
Differential Ly-6C expression identifies the recruited macrophage phenotype, which orchestrates the regression of murine liver fibrosis
Prakash Ramachandran;Antonella Pellicoro;Madeleine A. Vernon;Luke Boulter.
Proceedings of the National Academy of Sciences of the United States of America (2012)
Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver disease.
Luke Boulter;Olivier Govaere;Tom G Bird;Sorina Radulescu.
Nature Medicine (2012)
The Bone Marrow Functionally Contributes to Liver Fibrosis
Francesco P Russo;Malcolm R Alison;Brian W Bigger;Eunice Amofah.
Preparing the ground for tissue regeneration: from mechanism to therapy
Stuart J Forbes;Nadia Alicia Rosenthal.
Nature Medicine (2014)
A significant proportion of myofibroblasts are of bone marrow origin in human liver fibrosis
Stuart J. Forbes;Francesco P. Russo;Virginia Rey;Patrizia Burra.
Regulation of Alternative Macrophage Activation by Galectin-3
Alison C. MacKinnon;Sarah L. Farnworth;Philip S. Hodkinson;Neil C. Henderson.
Journal of Immunology (2008)
Matrix Stiffness Modulates Proliferation, Chemotherapeutic Response and Dormancy in Hepatocellular Carcinoma Cells
Joerg Schrader;Joerg Schrader;Timothy T. Gordon-Walker;Rebecca L. Aucott;Marielle van Deemter.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: