Bart O. Williams focuses on Wnt signaling pathway, Cell biology, Internal medicine, Endocrinology and Molecular biology. His Wnt signaling pathway research is multidisciplinary, incorporating elements of Cell signaling, PI3K/AKT/mTOR pathway and Stem cell. The concepts of his PI3K/AKT/mTOR pathway study are interwoven with issues in Cancer research and Phosphorylation.
His research integrates issues of Genetics, Cell growth, Cellular differentiation and Stromal cell in his study of Cell biology. The various areas that Bart O. Williams examines in his Internal medicine study include Beta-catenin, Retina and LRP5. Bart O. Williams has researched Molecular biology in several fields, including Embryonic stem cell, Retinoblastoma protein, Mutant and WNT3A.
Wnt signaling pathway, Cell biology, Cancer research, LRP5 and Internal medicine are his primary areas of study. His Wnt signaling pathway study is focused on Signal transduction in general. His Signal transduction research is multidisciplinary, incorporating perspectives in Molecular biology and Mutant.
Within one scientific family, Bart O. Williams focuses on topics pertaining to Cellular differentiation under Cell biology, and may sometimes address concerns connected to Cell type and Mesenchymal stem cell. His studies in Cancer research integrate themes in fields like Carcinogenesis, Cancer, Tumor suppressor gene, Protein kinase B and Pathology. His work in Internal medicine addresses issues such as Endocrinology, which are connected to fields such as Osteoblast and Cortical bone.
The scientist’s investigation covers issues in Wnt signaling pathway, Cell biology, Cancer research, Cortical bone and Endocrinology. He combines subjects such as Receptor and Homeostasis with his study of Wnt signaling pathway. His Cell biology research incorporates themes from SUMO protein, Osteoclast and Osteoblast.
His work carried out in the field of Cancer research brings together such families of science as Cancer, Pediatric cancer, Chromatin, SWI/SNF and Epigenetics. His Cortical bone study integrates concerns from other disciplines, such as Bone resorption, Hormone, Signal transduction, Bone remodeling and Null allele. His work deals with themes such as Osteocalcin, Internal medicine and Allele, which intersect with Endocrinology.
His main research concerns Cell biology, Wnt signaling pathway, Bone marrow, Cortical bone and Osteoclast. His Adipogenesis and LRP5 study in the realm of Cell biology connects with subjects such as Premature aging. He is studying LRP6, which is a component of Wnt signaling pathway.
His biological study spans a wide range of topics, including Cancer research, PORCN, Osteoblast, Toxicity and Fibrosis. His studies deal with areas such as Internal medicine, Endocrinology and Bone resorption as well as Cortical bone. His Internal medicine study incorporates themes from Messenger RNA, Gene and Allele.
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Tumor spectrum analysis in p53-mutant mice.
Tyler Jacks;Lee Remington;Bart O. Williams;Earlene M. Schmitt.
Current Biology (1994)
TSC2 Integrates Wnt and Energy Signals via a Coordinated Phosphorylation by AMPK and GSK3 to Regulate Cell Growth
Ken Inoki;Hongjiao Ouyang;Tianqing Zhu;Charlotta Lindvall.
Prostaglandin E2 promotes colon cancer cell growth through a Gs-axin-beta-catenin signaling axis.
Maria Domenica Castellone;Hidemi Teramoto;Bart O. Williams;Kirk M. Druey.
p53 -dependent apoptosis produced by Rb -deficiency in the developing mouse lens
Sharon D. Morgenbesser;Bart O. Williams;Tyler Jacks;Ronald A. DePinho.
A subset of p53-deficient embryos exhibit exencephaly.
Valerie P. Sah;Laura D. Attardi;George J. Mulligan;Bart O. Williams.
Nature Genetics (1995)
Targeted disruption of the three Rb-related genes leads to loss of G1 control and immortalization
Julien Sage;George J. Mulligan;Laura D. Attardi;Abigail Miller.
Genes & Development (2000)
Essential Role of β-Catenin in Postnatal Bone Acquisition
Sheri L. Holmen;Cassandra R. Zylstra;Aditi Mukherjee;Robert E. Sigler.
Journal of Biological Chemistry (2005)
p53-dependent G1 arrest involves pRB-related proteins and is disrupted by the human papillomavirus 16 E7 oncoprotein.
Robbert J. C. Slebos;Mann H. Lee;Beverly S. Plunkett;Theodore D. Kessis.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Cooperative tumorigenic effects of germline mutations in Rb and p53.
Bart O. Williams;Lee Remington;Daniel M. Albert;Shizuo Mukai.
Nature Genetics (1994)
Lrp5 functions in bone to regulate bone mass
Yajun Cui;Paul J Niziolek;Paul J Niziolek;Bryan T MacDonald;Cassandra R Zylstra.
Nature Medicine (2011)
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