His main research concerns Tumor necrosis factor alpha, Cell biology, Receptor, Signal transduction and Apoptosis. His Tumor necrosis factor alpha research incorporates elements of Cytokine, Cell culture, Immune system and Programmed cell death. His study looks at the relationship between Programmed cell death and fields such as NFKB1, as well as how they intersect with chemical problems.
His work on NF-κB is typically connected to Context as part of general Cell biology study, connecting several disciplines of science. The study incorporates disciplines such as Molecular biology, Stimulation and Antigen in addition to Receptor. He studied Signal transduction and Cancer research that intersect with Caspase 8.
Peter Scheurich mainly investigates Tumor necrosis factor alpha, Cell biology, Receptor, Apoptosis and Signal transduction. The concepts of his Tumor necrosis factor alpha study are interwoven with issues in Molecular biology, Cancer research, Antibody and Cytokine. He combines subjects such as Cell, Fas ligand, Fas receptor, FADD and Death domain with his study of Cell biology.
His Endocrinology research extends to Receptor, which is thematically connected. His work carried out in the field of Apoptosis brings together such families of science as Crosstalk and Signalling. His Signal transduction research is multidisciplinary, incorporating perspectives in Kinase, Protein kinase A, Downregulation and upregulation, Transcription factor and Tumor Necrosis Factor Receptor-Associated Factors.
Peter Scheurich mostly deals with Cell biology, Tumor necrosis factor alpha, Apoptosis, Signal transduction and Receptor. His Cell biology study combines topics from a wide range of disciplines, such as Fas ligand, Programmed cell death, Death domain and Immune system. His Tumor necrosis factor alpha study improves the overall literature in Immunology.
His Apoptosis study incorporates themes from Cell, Downregulation and upregulation and Crosstalk. His Signal transduction research includes elements of Transcription factor, Neurodegeneration, Molecular biology, FADD and Internalization. His Receptor research entails a greater understanding of Biochemistry.
His primary areas of investigation include Tumor necrosis factor alpha, Cell biology, Signal transduction, Immunology and Receptor. His Tumor necrosis factor alpha research is multidisciplinary, incorporating elements of Humanized antibody, Tissue homeostasis, Cytokine and Fragment crystallizable region. His studies in Cell biology integrate themes in fields like Inhibitor of apoptosis, Apoptosis, Programmed cell death and TRADD.
He has researched Apoptosis in several fields, including Transcription factor and Neuroscience. His Signal transduction study is concerned with Biochemistry in general. The various areas that Peter Scheurich examines in his Receptor study include Extracellular, Cell membrane and Binding site.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Tumor necrosis factor signaling.
H Wajant;K Pfizenmaier;P Scheurich.
Cell Death & Differentiation (2003)
THE TRANSMEMBRANE FORM OF TUMOR NECROSIS FACTOR IS THE PRIME ACTIVATING LIGAND OF THE 80 KDA TUMOR NECROSIS FACTOR RECEPTOR
Matthias Grell;Eleni Douni;Harald Wajant;Matthias Löhden.
NF-kappaB inducers upregulate cFLIP, a cycloheximide-sensitive inhibitor of death receptor signaling
Sebastian Kreuz;Daniela Siegmund;Peter Scheurich;Harald Wajant.
Molecular and Cellular Biology (2001)
Immunoregulatory activity of recombinant human tumor necrosis factor (TNF)-alpha: induction of TNF receptors on human T cells and TNF-alpha-mediated enhancement of T cell responses
P Scheurich;B Thoma;U Ucer;K Pfizenmaier.
Journal of Immunology (1987)
The type 1 receptor (CD120a) is the high-affinity receptor for soluble tumor necrosis factor
Matthias Grell;Harald Wajant;Gudrun Zimmermann;Peter Scheurich.
Proceedings of the National Academy of Sciences of the United States of America (1998)
The TNF-receptor-associated factor family: scaffold molecules for cytokine receptors, kinases and their regulators.
Harald Wajant;Frank Henkler;Peter Scheurich.
Cellular Signalling (2001)
Bistability Analyses of a Caspase Activation Model for Receptor-induced Apoptosis
Thomas Eissing;Holger Conzelmann;Ernst Dieter Gilles;Ernst Dieter Gilles;Frank Allgöwer.
Journal of Biological Chemistry (2004)
Induction of cell death by tumour necrosis factor (TNF) receptor 2, CD40 and CD30: a role for TNF‐R1 activation by endogenous membrane‐anchored TNF
Matthias Grell;Gudrun Zimmermann;Eva Gottfried;Chun Ming Chen;Chun Ming Chen.
The EMBO Journal (1999)
Ligands working as receptors: reverse signaling by members of the TNF superfamily enhance the plasticity of the immune system.
Günther Eissner;Walter Kolch;Peter Scheurich.
Cytokine & Growth Factor Reviews (2004)
Tumor necrosis factor enhances HLA-A,B,C and HLA-DR gene expression in human tumor cells.
K Pfizenmaier;P Scheurich;C Schlüter;M Krönke.
Journal of Immunology (1987)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: