D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 43 Citations 8,582 118 World Ranking 4147 National Ranking 1859

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Genetics

Peter Penzes mainly investigates Neuroscience, Dendritic spine, Cell biology, Dendritic spine morphogenesis and Neurotransmission. His work carried out in the field of Neuroscience brings together such families of science as NMDA receptor, Glutamatergic, Disease and Schizophrenia. He interconnects Endophenotype, AMPA receptor, Signal transduction, Alzheimer's disease and Synapse in the investigation of issues within Dendritic spine.

His Cell biology study incorporates themes from Morphogenesis, Actin cytoskeleton and Postsynaptic density. The study incorporates disciplines such as Synaptic plasticity and Receptor in addition to Dendritic spine morphogenesis. In his research, DISC1, Metaplasticity and Synapse maturation is intimately related to Glutamate receptor, which falls under the overarching field of Neurotransmission.

His most cited work include:

  • Dendritic spine pathology in neuropsychiatric disorders. (991 citations)
  • Rapid induction of dendritic spine morphogenesis by trans-synaptic ephrinB-EphB receptor activation of the Rho-GEF kalirin. (422 citations)
  • The neuronal Rho-GEF Kalirin-7 interacts with PDZ domain-containing proteins and regulates dendritic morphogenesis. (336 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Dendritic spine, Neuroscience, Cell biology, Excitatory postsynaptic potential and Synaptic plasticity. He studied Dendritic spine and Small GTPase that intersect with Corticotropin-releasing hormone. Peter Penzes has included themes like Schizophrenia, Signal transduction, Glutamatergic and Disease in his Neuroscience study.

The various areas that Peter Penzes examines in his Schizophrenia study include Alzheimer's disease, Regulation of gene expression, Autism and Auditory cortex. His Glutamatergic research incorporates themes from Neurotransmission and DISC1. His Cell biology research is multidisciplinary, relying on both Ankyrin and Actin cytoskeleton.

He most often published in these fields:

  • Dendritic spine (55.63%)
  • Neuroscience (50.99%)
  • Cell biology (50.99%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (50.99%)
  • Dendritic spine (55.63%)
  • Neuroscience (50.99%)

In recent papers he was focusing on the following fields of study:

Peter Penzes mainly focuses on Cell biology, Dendritic spine, Neuroscience, Excitatory postsynaptic potential and Ankyrin. The Cell biology study combines topics in areas such as CNTNAP2 and Cerebrospinal fluid. His studies deal with areas such as Synaptic plasticity, Excitatory synapse, Guanine nucleotide exchange factor and Small GTPase as well as Dendritic spine.

His work on Synapse as part of general Neuroscience study is frequently linked to Induced pluripotent stem cell, bridging the gap between disciplines. His study in Excitatory postsynaptic potential is interdisciplinary in nature, drawing from both AMPA receptor, Glutamatergic and Balance. His Ankyrin research is multidisciplinary, incorporating elements of HOMER1 and Gene isoform.

Between 2018 and 2021, his most popular works were:

  • Usp9X Controls Ankyrin-Repeat Domain Protein Homeostasis during Dendritic Spine Development (15 citations)
  • A novel role for the late-onset Alzheimer's disease (LOAD)-associated protein Bin1 in regulating postsynaptic trafficking and glutamatergic signaling. (15 citations)
  • Partial Loss of USP9X Function Leads to a Male Neurodevelopmental and Behavioral Disorder Converging on Transforming Growth Factor β Signaling. (14 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Genetics

His primary areas of study are Cell biology, Dendritic spine, Neuroscience, Excitatory postsynaptic potential and AMPA receptor. Peter Penzes focuses mostly in the field of Cell biology, narrowing it down to topics relating to Ankyrin and, in certain cases, Scaffold protein, Dendrite and Axon initial segment. In his study, Vesicular glutamate transporter 1, Guanine nucleotide exchange factor, Dendritic spine morphogenesis, Synapse and Hippocampal formation is strongly linked to Small GTPase, which falls under the umbrella field of Dendritic spine.

The concepts of his Neuroscience study are interwoven with issues in Missense mutation, Amyloid beta and Dysgenesis. Peter Penzes usually deals with Excitatory postsynaptic potential and limits it to topics linked to Glutamatergic and Colocalization, Neurotransmission, Schizophrenia and DISC1. His studies in AMPA receptor integrate themes in fields like Inhibitory postsynaptic potential and Postsynaptic potential.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Dendritic spine pathology in neuropsychiatric disorders

Peter Penzes;Michael E. Cahill;Kelly A. Jones;Jon Eric Vanleeuwen.
Nature Neuroscience (2011)

1375 Citations

Rapid induction of dendritic spine morphogenesis by trans-synaptic ephrinB-EphB receptor activation of the Rho-GEF kalirin.

Peter Penzes;Alexander Beeser;Jonathan Chernoff;Martin R. Schiller.
Neuron (2003)

589 Citations

The neuronal Rho-GEF Kalirin-7 interacts with PDZ domain-containing proteins and regulates dendritic morphogenesis.

Peter Penzes;Richard C. Johnson;Rita Sattler;Xiaoqun Zhang.
Neuron (2001)

454 Citations

Disrupted-in-Schizophrenia 1 (DISC1) regulates spines of the glutamate synapse via Rac1

Akiko Hayashi-Takagi;Manabu Takaki;Nick Graziane;Saurav Seshadri.
Nature Neuroscience (2010)

448 Citations

Kalirin-7 controls activity-dependent structural and functional plasticity of dendritic spines

Zhong Xie;Deepak P. Srivastava;Huzefa Photowala;Li Kai.
Neuron (2007)

385 Citations

Common mechanisms of excitatory and inhibitory imbalance in schizophrenia and autism spectrum disorders.

R. Gao;P. Penzes.
Current Molecular Medicine (2015)

368 Citations

Dendritic Structural Plasticity and Neuropsychiatric Disease

Marc P. Forrest;Euan Parnell;Peter Penzes.
Nature Reviews Neuroscience (2018)

269 Citations

Cloning of a cDNA encoding an aldehyde dehydrogenase and its expression in Escherichia coli. Recognition of retinal as substrate.

Xianshu Wang;Peter Penzes;Joseph L. Napoli.
Journal of Biological Chemistry (1996)

239 Citations

An Isoform of Kalirin, a Brain-specific GDP/GTP Exchange Factor, Is Enriched in the Postsynaptic Density Fraction

Peter Penzes;Richard C. Johnson;M.Rashid Alam;Vikram Kambampati.
Journal of Biological Chemistry (2000)

173 Citations

Kalirin regulates cortical spine morphogenesis and disease-related behavioral phenotypes.

Michael E. Cahill;Zhong Xie;Michelle Day;Huzefa Photowala.
Proceedings of the National Academy of Sciences of the United States of America (2009)

170 Citations

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