His primary scientific interests are in Borrelia burgdorferi, Microbiology, Gene, Cell activation and Treponema. Michael V. Norgard has included themes like Bacterial vaccine, Virulence, Virology, Spirochaete and Signal peptide in his Borrelia burgdorferi study. His Virology research includes themes of Ixodes scapularis and Plasmid.
His Microbiology study incorporates themes from Molecular biology and Macrophage. His Gene research is classified as research in Genetics. His Cell activation study combines topics from a wide range of disciplines, such as Immune system, Immunology and TLR2.
Michael V. Norgard mainly investigates Microbiology, Borrelia burgdorferi, Treponema, Biochemistry and Gene. The study incorporates disciplines such as Bacterial vaccine, Virology, Francisella tularensis, Virulence and Bacterial outer membrane in addition to Microbiology. His research in Virology intersects with topics in Ixodes scapularis, Pathogen, Antibody and Immunity.
His Borrelia burgdorferi study combines topics in areas such as rpoN, Mutant and rpoS. His studies in Treponema integrate themes in fields like Ligand, CD14, Membrane protein and Escherichia coli. His work in Gene addresses issues such as Molecular biology, which are connected to fields such as Peptide sequence, Operon, Southern blot, RNA and Open reading frame.
Michael V. Norgard mostly deals with Biochemistry, Treponema, Microbiology, Borrelia burgdorferi and ATP-binding cassette transporter. His Treponema research is multidisciplinary, incorporating perspectives in Binding protein, D-Glucose, Function and Escherichia coli. Michael V. Norgard works on Microbiology which deals in particular with Pathogen.
He has researched Borrelia burgdorferi in several fields, including Operon, Mutant, CD14, rpoN and rpoS. The various areas that Michael V. Norgard examines in his rpoN study include Regulation of gene expression and Sigma factor. His studies deal with areas such as Phagosome, Receptor, Innate immune system and Protein family as well as Francisella.
The scientist’s investigation covers issues in Biochemistry, Microbiology, Periplasmic space, Flavoprotein and Borrelia burgdorferi. Michael V. Norgard studies Microbiology, namely Tularemia. His research integrates issues of Transport protein, Flavin adenine dinucleotide, Protein structure and Flavin group in his study of Periplasmic space.
His Flavoprotein study deals with Treponema intersecting with Escherichia coli, Secondary syphilis, Bacterial pathogenesis, Virology and Pathogenicity. He has included themes like Pathogen, Regulation of gene expression, Gene, rpoN and rpoS in his Borrelia burgdorferi study. His rpoS research is multidisciplinary, relying on both Electrophoretic mobility shift assay, Lyme disease microbiology, Sigma factor and Regulatory Pathway.
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Host defense mechanisms triggered by microbial lipoproteins through toll-like receptors.
Hans D. Brightbill;Daniel H. Libraty;Stephan R. Krutzik;Ruey Bing Yang.
Induction of Direct Antimicrobial Activity Through Mammalian Toll-Like Receptors
Sybille Thoma-Uszynski;Steffen Stenger;Osamu Takeuchi;Maria Teresa Ochoa.
The Lyme disease agent exploits a tick protein to infect the mammalian host
Nandhini Ramamoorthi;Sukanya Narasimhan;Utpal Pal;Fukai Bao.
Microbial lipopeptides stimulate dendritic cell maturation via Toll-like receptor 2.
Cheryl J. Hertz;Sylvia M. Kiertscher;Paul J. Godowski;Deborah A. Bouis.
Journal of Immunology (2001)
DNA microarray analysis of differential gene expression in Borrelia burgdorferi, the Lyme disease spirochete
Andrew T. Revel;Adel M. Talaat;Michael V. Norgard.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Activation and regulation of Toll-like receptors 2 and 1 in human leprosy.
Stephan R. Krutzik;Maria Teresa Ochoa;Peter A. Sieling;Satoshi Uematsu.
Nature Medicine (2003)
OspC facilitates Borrelia burgdorferi invasion of Ixodes scapularis salivary glands
Utpal Pal;Xiaofeng Yang;Manchuan Chen;Linda K. Bockenstedt.
Journal of Clinical Investigation (2004)
Expression of Borrelia burgdorferi OspC and DbpA is controlled by a RpoN–RpoS regulatory pathway
Anette Hübner;Xiaofeng Yang;Dena M. Nolen;Taissia G. Popova.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Essential Role for OspA/B in the Life Cycle of the Lyme Disease Spirochete
Xiaofeng F. Yang;Utpal Pal;Sophie M. Alani;Erol Fikrig.
Journal of Experimental Medicine (2004)
Activation of Toll-Like Receptor 2 on Human Dendritic Cells Triggers Induction of IL-12, But Not IL-10
Sybille Thoma-Uszynski;Sylvia M. Kiertscher;Maria Teresa Ochoa;Deborah A. Bouis.
Journal of Immunology (2000)
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