What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in Molecular biology, RNA, Virology, RNA-dependent RNA polymerase and Hepatitis C virus.
His biological study spans a wide range of topics, including Cell culture, Viral replication, Gene, Cytidine deaminase and RNase P.
The RNA study combines topics in areas such as Replicon and Transcription.
His Virology research is multidisciplinary, relying on both RNA polymerase and Cholesterol.
His RNA-dependent RNA polymerase study integrates concerns from other disciplines, such as Non-coding RNA, RNA editing and RNA silencing.
His Hepatitis C virus research is multidisciplinary, incorporating perspectives in Interferon, Toll-like receptor and Protein biosynthesis.
His most cited work include:
- The molecular biology of coronaviruses (787 citations)
- Inhibition of the interferon-inducible protein kinase PKR by HCV E2 protein (692 citations)
- RNA recombination in animal and plant viruses. (531 citations)
What are the main themes of his work throughout his whole career to date?
Michael M. C. Lai spends much of his time researching Virology, RNA, Molecular biology, Virus and RNA-dependent RNA polymerase.
In general Virology, his work in Viral replication and Hepatitis C virus is often linked to Mouse hepatitis virus and Coronavirus linking many areas of study.
Michael M. C. Lai interconnects Transcription and Intron in the investigation of issues within RNA.
Michael M. C. Lai works mostly in the field of Molecular biology, limiting it down to topics relating to NS5B and, in certain cases, NS5A, as a part of the same area of interest.
His Virus research includes themes of Immune system and Antigen.
His study in RNA-dependent RNA polymerase is interdisciplinary in nature, drawing from both RNA silencing, RNA editing and RNA polymerase.
He most often published in these fields:
- Virology (77.84%)
- RNA (53.64%)
- Molecular biology (48.10%)
What were the highlights of his more recent work (between 2007-2021)?
- Virology (77.84%)
- Virus (48.10%)
- Viral replication (25.95%)
In recent papers he was focusing on the following fields of study:
Michael M. C. Lai mostly deals with Virology, Virus, Viral replication, Hepatitis C virus and Cell biology.
Michael M. C. Lai combines subjects such as RNA, RNA-dependent RNA polymerase and Pathogenesis with his study of Virology.
His studies in Virus integrate themes in fields like Cirrhosis and Antigen.
The concepts of his Hepatitis C virus study are interwoven with issues in Carcinogenesis, Cancer research, Antibody and Gene silencing.
His Cell biology study combines topics in areas such as Autophagy, NS5A and Endosome.
In his study, Molecular biology is strongly linked to MRE11 Homologue Protein, which falls under the umbrella field of DNA repair.
Between 2007 and 2021, his most popular works were:
- Toll-like receptor 4 mediates synergism between alcohol and HCV in hepatic oncogenesis involving stem cell marker Nanog (177 citations)
- Critical Role of Virion-Associated Cholesterol and Sphingolipid in Hepatitis C Virus Infection (160 citations)
- Rab5 and Class III Phosphoinositide 3-Kinase Vps34 Are Involved in Hepatitis C Virus NS4B-Induced Autophagy (116 citations)
In his most recent research, the most cited papers focused on:
Michael M. C. Lai mainly investigates Virology, Cell biology, Viral replication, Viral entry and DNA repair.
His work on RNA-dependent RNA polymerase expands to the thematically related Virology.
Michael M. C. Lai is investigating Transcription and RNA as part of his examination of RNA-dependent RNA polymerase.
His work in Viral replication tackles topics such as NS5A which are related to areas like Cell cycle, RNA interference, Cell growth, Gene Knockdown Techniques and NS5B.
As part of one scientific family, Michael M. C. Lai deals mainly with the area of Viral entry, narrowing it down to issues related to the Viral matrix protein, and often Viral structural protein and Virus maturation.
In DNA, Michael M. C. Lai works on issues like Molecular biology, which are connected to NS3.
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