Malcolm G. Dunlop spends much of his time researching Genetics, Colorectal cancer, Internal medicine, Cancer and Genome-wide association study. As a member of one scientific family, Malcolm G. Dunlop mostly works in the field of Genetics, focusing on Disease and, on occasion, Polymorphism and Early onset. He interconnects Randomized controlled trial, Surgery, Case-control study, Aspirin and Genetic testing in the investigation of issues within Colorectal cancer.
He has included themes like Gastroenterology, Immunology and Oncology in his Internal medicine study. His Cancer research incorporates elements of Odds ratio, Genetic variation, Family history and Risk factor. His Genome-wide association study research includes themes of Candidate gene, Uric acid, Gout, Genetic association and Genetic architecture.
His primary areas of study are Colorectal cancer, Internal medicine, Genetics, Oncology and Genome-wide association study. His Colorectal cancer research focuses on Cancer research and how it connects with Familial adenomatous polyposis. The various areas that Malcolm G. Dunlop examines in his Internal medicine study include Gastroenterology and Surgery.
His studies in Oncology integrate themes in fields like Pathology, Genetic variation, Family history and Genetic predisposition. His research investigates the connection with Genome-wide association study and areas like Genetic association which intersect with concerns in Genotyping and Linkage disequilibrium. His Cancer study incorporates themes from Penetrance and Cohort.
Malcolm G. Dunlop mainly focuses on Colorectal cancer, Internal medicine, Oncology, Cancer and Genome-wide association study. His study in Colorectal cancer is interdisciplinary in nature, drawing from both Observational study, Genetic variation, Genetic association and MEDLINE. His work in Internal medicine addresses subjects such as Biobank, which are connected to disciplines such as Mendelian inheritance.
The study incorporates disciplines such as Genetic model and Surgical resection in addition to Oncology. His Cancer research integrates issues from Adverse effect, Triage and Medical diagnosis. Malcolm G. Dunlop is researching Genome-wide association study as part of the investigation of Single-nucleotide polymorphism, Genetics and Gene.
The scientist’s investigation covers issues in Internal medicine, Colorectal cancer, Vitamin D and neurology, Gastroenterology and Genome-wide association study. His biological study spans a wide range of topics, including Epidemiology, Oncology, MEDLINE, Meta-analysis and Risk factor. His research integrates issues of Surgical resection and Disease severity in his study of Oncology.
His work carried out in the field of Vitamin D and neurology brings together such families of science as Perioperative, Systematic review, Survival analysis and Area under the curve. His research investigates the link between Gastroenterology and topics such as Guideline that cross with problems in Disease management, Pouchitis, Vedolizumab, Ulcerative colitis and Mesalazine. His work deals with themes such as Cancer and Medical genetics, which intersect with Genome-wide association study.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Telomere reduction in human colorectal carcinoma and with ageing
Nicholas D. Hastie;Maureen Dempster;Malcolm G. Dunlop;Alastair M. Thompson.
Nature (1990)
Mutations of two PMS homologues in hereditary nonpolyposis colon cancer.
Nicholas C. Nicolaides;Nickolas Papadopoulos;Bo Liu;Ying Fei Weit.
Nature (1994)
Analysis of mismatch repair genes in hereditary non–polyposis colorectal cancer patients
Bo Liu;Ramon Parsons;Nickolas Papadopoulos;Nicholas C. Nicolaides.
Nature Medicine (1996)
Guidelines for colorectal cancer screening and surveillance in moderate and high risk groups (update from 2002)
Stuart R Cairns;John H Scholefield;Robert J Steele;Malcolm G Dunlop.
Gut (2010)
Long-term effect of aspirin on cancer risk in carriers of hereditary colorectal cancer: an analysis from the CAPP2 randomised controlled trial
John Burn;Anne Marie Gerdes;Finlay MacRae;Jukka Pekka Mecklin.
The Lancet (2011)
Causal relationship between obesity and vitamin D status: bi-directional Mendelian randomization analysis of multiple cohorts.
Karani S. Vimaleswaran;Diane J. Berry;Emmi Tikkanen;Emmi Tikkanen;Stefan Pilz;Stefan Pilz.
PLOS Medicine (2013)
Runs of Homozygosity in European Populations
Ruth McQuillan;Anne-Louise Leutenegger;Rehab Abdel-Rahman;Rehab Abdel-Rahman;Christopher S. Franklin.
American Journal of Human Genetics (2008)
Cancer Risk Associated with Germline DNA Mismatch Repair Gene Mutations
Malcolm G. Dunlop;Malcolm G. Dunlop;Susan M. Farrington;Susan M. Farrington;Andrew D. Carothers;Andrew H. Wyllie.
Human Molecular Genetics (1997)
Genome-wide association scan identifies a colorectal cancer susceptibility locus on chromosome 8q24
Brent W. Zanke;Celia M.T. Greenwood;Celia M.T. Greenwood;Jagadish Rangrej;Rafal Kustra;Rafal Kustra.
Nature Genetics (2007)
Modulation of genetic associations with serum urate levels by body-mass-index in humans
Jennifer E. Huffman;Eva Albrecht;Alexander Teumer;Massimo Mangino.
PLOS ONE (2015)
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