D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 43 Citations 6,286 177 World Ranking 14117 National Ranking 1009

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

His primary areas of study are Genetics, Retina, Gene, Molecular biology and Retinitis pigmentosa. His Retina research is multidisciplinary, incorporating perspectives in Retinopathy, Transplantation and Anatomy. His Gene study deals with Macular degeneration intersecting with Locus, Hemicentin 1, Japanese population and Decreased Visual Acuity.

Makoto Tamai has included themes like Cell culture, Northern blot, Microphthalmia-associated transcription factor, Regulation of gene expression and Western blot in his Molecular biology study. Ophthalmology covers Makoto Tamai research in Retinitis pigmentosa. His Ophthalmology research focuses on subjects like Surgery, which are linked to Atrophy.

His most cited work include:

  • A homozygous 1-base pair deletion in the arrestin gene is a frequent cause of Oguchi disease in Japanese. (240 citations)
  • HLF/HIF‐2α is a key factor in retinopathy of prematurity in association with erythropoietin (196 citations)
  • Brain-derived neurotrophic factor prevents axotomized retinal ganglion cell death through MAPK and PI3K signaling pathways. (178 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Retina, Ophthalmology, Retinal, Molecular biology and Genetics. His research integrates issues of Cell biology, Transplantation, Anatomy and Pathology in his study of Retina. His Cell biology research is multidisciplinary, incorporating perspectives in Neurotrophic factors and Retinal ganglion.

His Ophthalmology research focuses on Retinopathy and how it relates to Eye disease, Diabetic retinopathy and Proliferative vitreoretinopathy. His Retinal research is multidisciplinary, relying on both Cell culture, Internal medicine and Endocrinology. His work on Genetics deals in particular with Gene, Retinitis pigmentosa, Mutation, Mutation and Exon.

He most often published in these fields:

  • Retina (21.14%)
  • Ophthalmology (18.91%)
  • Retinal (18.66%)

What were the highlights of his more recent work (between 2005-2018)?

  • Retina (21.14%)
  • Channelrhodopsin (4.48%)
  • Retinal (18.66%)

In recent papers he was focusing on the following fields of study:

Makoto Tamai spends much of his time researching Retina, Channelrhodopsin, Retinal, Cell biology and Retinal ganglion. Retinal pigment epithelium is the focus of his Retina research. His work deals with themes such as Gene, Transduction and Genetic enhancement, which intersect with Channelrhodopsin.

His Gene study is concerned with the field of Genetics as a whole. The various areas that Makoto Tamai examines in his Retinal study include Molecular biology, Neurotrophic factors, Toxicity and Anatomy. His Retinal ganglion study combines topics in areas such as Transgene, Anesthesia, Retinitis pigmentosa and Pathology.

Between 2005 and 2018, his most popular works were:

  • Visual properties of transgenic rats harboring the channelrhodopsin-2 gene regulated by the thy-1.2 promoter. (136 citations)
  • Restoration of visual response in aged dystrophic RCS rats using AAV-mediated channelopsin-2 gene transfer. (109 citations)
  • Channelrhodopsin-2 gene transduced into retinal ganglion cells restores functional vision in genetically blind rats ☆ (106 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

Makoto Tamai mainly investigates Retina, Retinal, Channelrhodopsin, Cell biology and Pathology. The concepts of his Retina study are interwoven with issues in Anatomy, Neural crest, Viability assay, Cornea and Lens. His Retinal study combines topics from a wide range of disciplines, such as Cell culture, Lipofuscin, Molecular biology and Dithiothreitol.

His study in Channelrhodopsin is interdisciplinary in nature, drawing from both Retinal degeneration, Virology, Retinitis pigmentosa and Retinal ganglion. His Retinitis pigmentosa study is related to the wider topic of Ophthalmology. His Pathology study incorporates themes from Cell, Gene expression, Transduction and Transplantation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A homozygous 1-base pair deletion in the arrestin gene is a frequent cause of Oguchi disease in Japanese.

Sigrid Fuchs;Mitsuru Nakazawa;Marion Maw;Makoto Tamai.
Nature Genetics (1995)

366 Citations

Brain-derived neurotrophic factor prevents axotomized retinal ganglion cell death through MAPK and PI3K signaling pathways.

Toru Nakazawa;Makoto Tamai;Nozomu Mori.
Investigative Ophthalmology & Visual Science (2002)

270 Citations

HLF/HIF‐2α is a key factor in retinopathy of prematurity in association with erythropoietin

Masanobu Morita;Osamu Ohneda;Toshiharu Yamashita;Toshiharu Yamashita;Satoru Takahashi.
The EMBO Journal (2003)

257 Citations

Structural organization of the human microphthalmia-associated transcription factor gene containing four alternative promoters.

Tetsuo Udono;Ken Ichi Yasumoto;Kazuhisa Takeda;Shintaro Amae.
Biochimica et Biophysica Acta (2000)

189 Citations

Quantifying alterations of macular thickness before and after panretinal photocoagulation in patients with severe diabetic retinopathy and good vision.

Masahiko Shimura;Kanako Yasuda;Toru Nakazawa;Tetsuya Kano.
Ophthalmology (2003)

184 Citations

Identification of a Novel Isoform of Microphthalmia-Associated Transcription Factor That Is Enriched in Retinal Pigment Epithelium

Shintaro Amae;Nobuo Fuse;Ken-ichi Yasumoto;Shigeru Sato.
Biochemical and Biophysical Research Communications (1998)

166 Citations

Channelrhodopsin-2 gene transduced into retinal ganglion cells restores functional vision in genetically blind rats ☆

Hiroshi Tomita;Eriko Sugano;Hitomi Isago;Teru Hiroi.
Experimental Eye Research (2010)

165 Citations

Restoration of visual response in aged dystrophic RCS rats using AAV-mediated channelopsin-2 gene transfer.

Hiroshi Tomita;Eriko Sugano;Hiromu Yawo;Toru Ishizuka.
Investigative Ophthalmology & Visual Science (2007)

165 Citations

Interleukin-1β and barrier function of retinal pigment epithelial cells (ARPE-19): Aberrant expression of junctional complex molecules

Toshiaki Abe;Eriko Sugano;Yoko Saigo;Makoto Tamai.
Investigative Ophthalmology & Visual Science (2003)

146 Citations

Visual properties of transgenic rats harboring the channelrhodopsin-2 gene regulated by the thy-1.2 promoter.

Hiroshi Tomita;Eriko Sugano;Yugo Fukazawa;Hitomi Isago.
PLOS ONE (2009)

142 Citations

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