D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 85 Citations 24,758 466 World Ranking 9637 National Ranking 276

Overview

What is he best known for?

The fields of study Yasuto Itoyama is best known for:

  • Gene
  • Central nervous system
  • Neuron

Radiology and Lesion are fields of study that intersect with his Magnetic resonance imaging research. By researching both Radiology and Magnetic resonance imaging, he produces research that crosses academic boundaries. His work often combines Lesion and Spinal cord studies. He conducts interdisciplinary study in the fields of Spinal cord and Myelitis through his works. His study on Surgery is mostly dedicated to connecting different topics, such as Corticosteroid and Retrospective cohort study. Many of his studies on Retrospective cohort study apply to Internal medicine as well. Yasuto Itoyama regularly ties together related areas like Aquaporin 4 in his Internal medicine studies. In his papers, he integrates diverse fields, such as Aquaporin 4 and Neuromyelitis optica. Pathology and Glial fibrillary acidic protein are frequently intertwined in his study.

His most cited work include:

  • Loss of aquaporin 4 in lesions of neuromyelitis optica: distinction from multiple sclerosis (531 citations)
  • Diffusion-weighted MRI abnormalities as an early diagnostic marker for Creutzfeldt-Jakob disease (307 citations)
  • Rapid Induction of Vascular Endothelial Growth Factor Gene Expression After Transient Middle Cerebral Artery Occlusion in Rats (297 citations)

What are the main themes of his work throughout his whole career to date

Yasuto Itoyama connects relevant research areas such as Immunohistochemistry and Disease in the realm of Pathology. His Biochemistry research is linked to Gene and Receptor, among other subjects. His Receptor study frequently involves adjacent topics like Internal medicine. His Ischemia research extends to the thematically linked field of Internal medicine. His Genetics research spans across into fields like Gene and Neuroscience. He undertakes multidisciplinary investigations into Neuroscience and Genetics in his work. Yasuto Itoyama integrates several fields in his works, including Endocrinology and Pathology. Borrowing concepts from Antibody, he weaves in ideas under Immunology. His multidisciplinary approach integrates Antibody and Immunology in his work.

Yasuto Itoyama most often published in these fields:

  • Internal medicine (63.81%)
  • Pathology (40.95%)
  • Biochemistry (39.05%)

What were the highlights of his more recent work (between 2008-2016)?

  • Immunology (53.85%)
  • Internal medicine (53.85%)
  • Pathology (46.15%)

In recent works Yasuto Itoyama was focusing on the following fields of study:

Meningeal carcinomatosis and Lumbar puncture are inextricably linked to his Cerebrospinal fluid research. His Cerebrospinal fluid research extends to the thematically linked field of Meningeal carcinomatosis. Yasuto Itoyama performs multidisciplinary study in the fields of Lumbar puncture and Central nervous system via his papers. Borrowing concepts from Astrocyte, Yasuto Itoyama weaves in ideas under Central nervous system. He regularly ties together related areas like Neuroscience in his Astrocyte studies. His studies link Cerebellum with Neuroscience. Yasuto Itoyama undertakes interdisciplinary study in the fields of Cerebellum and White matter through his works. White matter and Voxel-based morphometry are two areas of study in which he engages in interdisciplinary research. Acute motor axonal neuropathy and Polyradiculoneuropathy are the focus of his Guillain-Barre syndrome studies.

Between 2008 and 2016, his most popular works were:

  • Temporal changes and geographical differences in multiple sclerosis phenotypes in Japanese: nationwide survey results over 30 years (145 citations)
  • Marked increase in cerebrospinal fluid glial fibrillar acidic protein in neuromyelitis optica: an astrocytic damage marker (125 citations)
  • A case of NMO seropositive for aquaporin-4 antibody more than 10 years before onset (121 citations)

In his most recent research, the most cited works focused on:

  • Neuromyelitis optica
  • Multiple sclerosis
  • Acute disseminated encephalomyelitis

In his papers, Yasuto Itoyama integrates diverse fields, such as Multiple sclerosis and Expanded Disability Status Scale. Yasuto Itoyama brings together Expanded Disability Status Scale and Multiple sclerosis to produce work in his papers. Yasuto Itoyama connects Psychiatry with Pediatrics in his research. Yasuto Itoyama conducts interdisciplinary study in the fields of Pediatrics and Psychiatry through his research. His Neuromyelitis optica study frequently draws connections between related disciplines such as Antibody. Antibody is closely attributed to Neuromyelitis optica in his research. Yasuto Itoyama combines topics linked to Internal medicine with his work on Aquaporin 4. His study connects Aquaporin 4 and Internal medicine. He performs multidisciplinary study in Immunology and Virology in his work.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Interleukin-1 as a Pathogenetic Mediator of Ischemic Brain Damage in Rats

Yasundo Yamasaki;Naosuke Matsuura;Hidetaka Shozuhara;Hiroshi Onodera.
Stroke (1995)

792 Citations

Anti-aquaporin-4 antibody is involved in the pathogenesis of NMO: a study on antibody titre.

Toshiyuki Takahashi;Kazuo Fujihara;Ichiro Nakashima;Tatsuro Misu.
Brain (2007)

700 Citations

Loss of aquaporin 4 in lesions of neuromyelitis optica: distinction from multiple sclerosis.

T. Misu;K. Fujihara;A. Kakita;H. Konno.
Brain (2007)

669 Citations

Presenilin-1 mutations downregulate the signalling pathway of the unfolded-protein response

Taiichi Katayama;Kazunori Imaizumi;Naoya Sato;Ko Miyoshi.
Nature Cell Biology (1999)

648 Citations

Neuromyelitis optica: pathogenicity of patient immunoglobulin in vivo.

Monika Bradl;Tatsuro Misu;Toshiyuki Takahashi;Mitsutoshi Watanabe.
Annals of Neurology (2009)

564 Citations

Safety and efficacy of edaravone in well defined patients with amyotrophic lateral sclerosis: a randomised, double-blind, placebo-controlled trial

Koji Abe;Masashi Aoki;Shoji Tsuji;Yasuto Itoyama.
Lancet Neurology (2017)

486 Citations

Reduction of ischemic damage by application of vascular endothelial growth factor in rat brain after transient ischemia

Takeshi Hayashi;Koji Abe;Yasuto Itoyama.
Journal of Cerebral Blood Flow and Metabolism (1998)

480 Citations

Rapid induction of vascular endothelial growth factor gene expression after transient middle cerebral artery occlusion in rats.

T. Hayashi;K. Abe;H. Suzuki;Y. Itoyama.
Stroke (1997)

457 Citations

Rats Expressing Human Cytosolic Copper–Zinc Superoxide Dismutase Transgenes with Amyotrophic Lateral Sclerosis: Associated Mutations Develop Motor Neuron Disease

Makiko Nagai;Masashi Aoki;Ichiro Miyoshi;Masaaki Kato.
The Journal of Neuroscience (2001)

455 Citations

Silent Cerebral Microbleeds on T2*-Weighted MRI: Correlation with Stroke Subtype, Stroke Recurrence, and Leukoaraiosis

Hiroyuki Kato;Masahiro Izumiyama;Kimiaki Izumiyama;Akira Takahashi.
Stroke (2002)

430 Citations

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