Keith B. Elkon spends much of his time researching Immunology, Apoptosis, Fas receptor, Antigen and Molecular biology. His Immunology study frequently links to related topics such as Systemic lupus erythematosus. He works mostly in the field of Apoptosis, limiting it down to topics relating to Autoimmunity and, in certain cases, Phagocytosis and Immunoglobulin G.
His Fas receptor research includes elements of Phenotype, Fas ligand, CD8 and Mutation. His research in Antigen intersects with topics in Autoantibody and Graft-versus-host disease. His work deals with themes such as Fetus, Reverse transcriptase, Northern blot and Cell biology, which intersect with Molecular biology.
Keith B. Elkon focuses on Immunology, Antibody, Autoantibody, Molecular biology and Apoptosis. Immunology is often connected to Systemic lupus erythematosus in his work. His Antibody study integrates concerns from other disciplines, such as Internal medicine, Gel electrophoresis and Ribosomal protein.
The Autoantibody study combines topics in areas such as Subclass, Counterimmunoelectrophoresis, Epitope, Antigen and Anti-SSA/Ro autoantibodies. His Molecular biology research integrates issues from Complementary DNA, Amino acid and Peptide sequence. Keith B. Elkon has researched Apoptosis in several fields, including Cancer research and Signal transduction.
Keith B. Elkon mostly deals with Apoptosis, Immunology, Fas receptor, Autoimmunity and Cancer research. His study in Apoptosis is interdisciplinary in nature, drawing from both Signal transduction, Cell biology and Complement system. His Immunology and Autoantibody, B cell, Autoimmune disease, Immune system and Adoptive cell transfer investigations all form part of his Immunology research activities.
His Autoantibody study combines topics in areas such as Spleen, Lupus erythematosus and Pathology. His Lupus erythematosus research includes themes of Systemic lupus erythematosus and Endocrinology. His research investigates the link between Fas receptor and topics such as Death domain that cross with problems in FADD.
The scientist’s investigation covers issues in Cytokine, Apoptosis, Cell biology, Innate immune system and Immunology. The various areas that Keith B. Elkon examines in his Apoptosis study include Cancer research and Signal transduction. His work carried out in the field of Signal transduction brings together such families of science as Tumor suppressor gene and Autoimmune disease.
His Cell biology study combines topics from a wide range of disciplines, such as Fas ligand, Complement component 3, Classical complement pathway, Inhibitor of apoptosis and Complement membrane attack complex. His Innate immune system research incorporates elements of Acquired immune system, Tumor necrosis factor alpha, Immunity, Defective virus and Adoptive cell transfer. His research investigates the connection with Immunology and areas like Cytotoxic T cell which intersect with concerns in Autoimmunity.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Systemic vasculitis with asthma and eosinophilia: a clinical approach to the Churg-Strauss syndrome
John G. Lanham;Keith B. Elkon;Charles D. Pusey;Graham R. Hughes.
Complement-dependent Clearance of Apoptotic Cells by Human Macrophages
Dror Mevorach;John O. Mascarenhas;Debra Gershov;Keith B. Elkon.
Journal of Experimental Medicine (1998)
C-Reactive Protein Binds to Apoptotic Cells, Protects the Cells from Assembly of the Terminal Complement Components, and Sustains an Antiinflammatory Innate Immune Response
Debra Gershov;SunJung Kim;Nathan Brot;Keith B. Elkon.
Journal of Experimental Medicine (2000)
SYSTEMIC EXPOSURE TO IRRADIATED APOPTOTIC CELLS INDUCES AUTOANTIBODY PRODUCTION
Dror Mevorach;Jun Liang Zhou;Xin Song;Keith B. Elkon.
Journal of Experimental Medicine (1998)
Fas gene mutations in the Canale-Smith syndrome, an inherited lymphoproliferative disorder associated with autoimmunity.
Jörn Drappa;Akshay K. Vaishnaw;Kathleen E. Sullivan;Jia Li Chu.
The New England Journal of Medicine (1996)
Impaired Fas response and autoimmunity in Pten+/- mice
Antonio Di Cristofano;Paraskevi Kotsi;Yu Feng Peng;Carlos Cordon-Cardo.
The defect in Fas mRNA expression in MRL/lpr mice is associated with insertion of the retrotransposon, ETn.
Jia Li Chu;Jorn Drappa;Andrew Parnassa;Keith B. Elkon.
Journal of Experimental Medicine (1993)
CD40 ligation induces apo-1/fas expression on human B lymphocytes and facilitates apoptosis through the apo-1/fas pathway
Elaine J. Schattner;Keith B. Elkon;Dae Hyun Yoo;Joseph Tumang.
Journal of Experimental Medicine (1995)
The Fas protein is expressed at high levels on CD4+CD8+ thymocytes and activated mature lymphocytes in normal mice but not in the lupus-prone strain, MRL lpr/lpr.
Jorn Drappa;Nathan Brot;Keith B. Elkon.
Proceedings of the National Academy of Sciences of the United States of America (1993)
The apoptosis-1/Fas protein in human systemic lupus erythematosus.
Eduardo Mysler;Paolo Bini;Jörn Drappa;Paula Ramos.
Journal of Clinical Investigation (1994)
Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking d-index is inferred from publications deemed to belong to the considered discipline.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: