Jie Zhu mainly focuses on Immunology, Inflammation, Multiple sclerosis, Proinflammatory cytokine and Pathogenesis. His Immunology research is multidisciplinary, relying on both Neuritis and Myelin. Jie Zhu combines subjects such as Peripheral blood mononuclear cell and Macrophage with his study of Inflammation.
Jie Zhu has researched Multiple sclerosis in several fields, including Meningitis and Central nervous system. His studies deal with areas such as Interleukin 12 and Microglia as well as Proinflammatory cytokine. His Pathogenesis research includes elements of Cerebral cortex, Antibody and Amyloid precursor protein.
His primary areas of investigation include Immunology, Internal medicine, Inflammation, Neuritis and Cytokine. His Immunology study frequently links to related topics such as Myelin. His Internal medicine study deals with Endocrinology intersecting with Kainic acid and Neurodegeneration.
Jie Zhu is interested in Proinflammatory cytokine, which is a field of Inflammation. His Neuritis study combines topics from a wide range of disciplines, such as Peripheral nervous system and Guillain-Barre syndrome. His Cytokine study integrates concerns from other disciplines, such as Tumor necrosis factor alpha, Autoantibody, Autoimmune disease and Autoimmunity.
His primary scientific interests are in Immunology, Multiple sclerosis, Inflammation, Pathogenesis and Experimental autoimmune encephalomyelitis. His study in Immunology concentrates on Immune system, Proinflammatory cytokine, Guillain-Barre syndrome, Autoantibody and Molecular mimicry. The concepts of his Guillain-Barre syndrome study are interwoven with issues in Neuritis, Disease progression, Internal medicine and Gastroenterology.
His research in Multiple sclerosis intersects with topics in Autoimmunity, Rheumatoid arthritis, Bone marrow and Regulatory B cells. His study in Inflammation is interdisciplinary in nature, drawing from both Innate immune system and Disease. His Experimental autoimmune encephalomyelitis study combines topics in areas such as Autoimmune disease, NF-κB, Signal transduction and Antigen presentation.
His scientific interests lie mostly in Immunology, Inflammation, Multiple sclerosis, Experimental autoimmune encephalomyelitis and Pathogenesis. His work in Immunology covers topics such as Macrophage which are related to areas like Microglia. His Multiple sclerosis research is multidisciplinary, incorporating elements of Alzheimer's disease, Disease, Caspase and Neuroscience.
His biological study spans a wide range of topics, including Autoimmune disease, Nod and Innate immune system, AIM2. His work deals with themes such as Aquaporin 4, Leukotriene B4, Neuromyelitis optica and CXCL13, which intersect with Pathogenesis. As part of one scientific family, Jie Zhu deals mainly with the area of Immune system, narrowing it down to issues related to the Inflammatory response, and often Receptor.
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The Antidepressant and Antiinflammatory Effects of Rolipram in the Central Nervous System
Jie Zhu;Eilhard Mix;Bengt Winblad.
Cns Drug Reviews (2006)
The roles of macrophages and microglia in multiple sclerosis and experimental autoimmune encephalomyelitis.
Fengna Chu;Mingchao Shi;Chao Zheng;Donghui Shen.
Journal of Neuroimmunology (2018)
Role of Microglia in Neurological Disorders and Their Potentials as a Therapeutic Target
Li Du;Ying Zhang;Yang Chen;Jie Zhu;Jie Zhu.
Molecular Neurobiology (2017)
Up-regulation of the inflammatory cytokines IFN-γ and IL-12 and down-regulation of IL-4 in cerebral cortex regions of APPSWE transgenic mice
Nagat Abbas;Ivan Bednar;Eilhard Mix;Svedberg Marie.
Journal of Neuroimmunology (2002)
Cytokine production and the pathogenesis of experimental autoimmune neuritis and Guillain–Barré syndrome
Jie Zhu;Eilhard Mix;Hans Link.
Journal of Neuroimmunology (1998)
Kainic acid-induced neurodegenerative model: potentials and limitations.
Xiang-Yu Zheng;Hong-Liang Zhang;Qi Luo;Jie Zhu.
BioMed Research International (2011)
Neutralization of Macrophage Inflammatory Protein 2 (MIP-2) and MIP-1α Attenuates Neutrophil Recruitment in the Central Nervous System during Experimental Bacterial Meningitis
Asim Diab;Hana Abdalla;Hu Lun Li;Fu Dong Shi.
Infection and Immunity (1999)
Suppression of experimental autoimmune neuritis by ABR-215062 is associated with altered Th1/Th2 balance and inhibited migration of inflammatory cells into the peripheral nerve tissue
L.-P. Zou;N. Abbas;I. Volkmann;I. Nennesmo.
Kainic Acid-induced neurotoxicity: targeting glial responses and glia-derived cytokines.
Xing-Mei Zhang;Jie Zhu.
Current Neuropharmacology (2011)
Cytokine Dichotomy in Peripheral Nervous System Influences the Outcome of Experimental Allergic Neuritis: Dynamics of mRNA Expression for IL-1β, IL-6, IL-10, IL-12, TNF-α, TNF-β, and Cytolysin
J. Zhu;X.F. Bai;E. Mix;H. Link.
Clinical Immunology and Immunopathology (1997)
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