The scientist’s investigation covers issues in Immunology, Virus, Virology, Pathology and Viral replication. His work deals with themes such as Intestinal mucosa and Microbiology, which intersect with Immunology. His Virus study combines topics from a wide range of disciplines, such as Budding, Lymphatic system, Disease and Mutant.
The study incorporates disciplines such as Open reading frame, Antibody, Genetic predisposition and Genotype in addition to Virology. The various areas that Jan M. Orenstein examines in his Pathology study include Microsporidia, Encephalitozoon intestinalis, Enterocytozoon bieneusi and In situ hybridization. His Viral replication research is multidisciplinary, incorporating perspectives in Interferon alfa, Alpha interferon and Murine leukemia virus.
Jan M. Orenstein mainly investigates Immunology, Virology, Pathology, Virus and Viral replication. His studies deal with areas such as Acquired immunodeficiency syndrome and Microbiology as well as Immunology. Jan M. Orenstein has researched Virology in several fields, including Cell culture, Macrophage, Enterocytozoon bieneusi and Microsporidia.
In the subject of general Pathology, his work in Immunohistochemistry, Autopsy and Ultrastructure is often linked to Myofibroblast, thereby combining diverse domains of study. His research integrates issues of Antibody and Signal transduction in his study of Virus. The concepts of his Viral disease study are interwoven with issues in Microsporidiosis and Diarrhea.
His primary areas of investigation include Immunology, Pathology, Virus, Virology and Immune system. His Immunology research incorporates themes from Heart disease, Gene expression, Arteritis and Asymptomatic. Jan M. Orenstein combines subjects such as Antibody and Cytoplasmic inclusion with his study of Pathology.
In the field of Virus, his study on Viral replication, Viral life cycle and Viral matrix protein overlaps with subjects such as Coronavirus. Many of his studies on Virology involve topics that are commonly interrelated, such as Reverse transcriptase. His Immune system research is multidisciplinary, incorporating elements of Inflammation, Viremia and Viral load.
His primary areas of study are Immunology, Cytoplasmic inclusion, Inclusion bodies, Pathology and Virus. In the subject of general Immunology, his work in Pathogenesis is often linked to Coronary artery aneurysm, thereby combining diverse domains of study. The Pathology study combines topics in areas such as Aneurysm, Lung and Myocardial infarction.
His study connects Alpha interferon and Virus. As part of his research on Viral replication, studies on Virology and Cell culture are part of the effort. His Virology research incorporates elements of Mitochondrial toxicity, Mitochondrion and Reverse transcriptase.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
HIV infection is active and progressive in lymphoid tissue during the clinically latent stage of disease
Giuseppe Pantaleo;Cecilia Graziosi;James F. Demarest;Luca Butini.
Nature (1993)
Human intestinal macrophages display profound inflammatory anergy despite avid phagocytic and bacteriocidal activity
Lesley E. Smythies;Marty Sellers;Ronald H. Clements;Meg Mosteller-Barnum.
Journal of Clinical Investigation (2005)
Studies in subjects with long-term nonprogressive human immunodeficiency virus infection.
Giuseppe Pantaleo;Stefano Menzo;Mauro Vaccarezza;Cecilia Graziosi.
The New England Journal of Medicine (1995)
Macrophages as a Source of HIV During Opportunistic Infections
Jan M. Orenstein;Cecil Fox;Sharon M. Wahl.
Science (1997)
Secretory leukocyte protease inhibitor: a human saliva protein exhibiting anti-human immunodeficiency virus 1 activity in vitro.
T B McNeely;M Dealy;D J Dripps;J M Orenstein.
Journal of Clinical Investigation (1995)
A role for tumor necrosis factor receptor-2 and receptor-interacting protein in programmed necrosis and antiviral responses.
Francis Ka Ming Chan;Francis Ka Ming Chan;Joanna L. Shisler;Jacqueline G. Bixby;Martin Felices.
Journal of Biological Chemistry (2003)
Intestinal Macrophages Lack CD14 and CD89 and Consequently Are Down-Regulated for LPS- and IgA-Mediated Activities
Phillip D. Smith;Lesley E. Smythies;Meg Mosteller-Barnum;Don A. Sibley.
Journal of Immunology (2001)
Overexpression of the N-terminal domain of TSG101 inhibits HIV-1 budding by blocking late domain function
Dimiter G. Demirov;Akira Ono;Jan M. Orenstein;Eric O. Freed.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Interferon-alpha but not AZT suppresses HIV expression in chronically infected cell lines
Guido Poli;Jan M. Orenstein;Audrey Kinter;Thomas M. Folks.
Science (1989)
High-level variability in the ORF-K1 membrane protein gene at the left end of the Kaposi's sarcoma-associated herpesvirus genome defines four major virus subtypes and multiple variants or clades in different human populations.
Jian Chao Zong;Dolores M. Ciufo;Donald J. Alcendor;Xiaoyu Wan.
Journal of Virology (1999)
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