His primary areas of study are Internal medicine, Endocrinology, Pulmonary hypertension, Hypoxia and Vasoconstriction. Many of his studies on Internal medicine apply to Cardiology as well. Ivan F. McMurtry focuses mostly in the field of Endocrinology, narrowing it down to matters related to Pulmonary artery and, in some cases, Hemodynamics, Pathophysiology of hypertension and Bronchiectasis.
His Pulmonary hypertension study combines topics from a wide range of disciplines, such as Respiratory disease, Lung, Anesthesia and Nitric oxide synthase, Nitric oxide. His Hypoxia study incorporates themes from Vascular remodelling in the embryo, Fasudil, Rho-associated protein kinase and Bioinformatics. His Vasoconstriction research incorporates elements of Endothelial stem cell, Arachidonic acid and Cell biology.
Ivan F. McMurtry spends much of his time researching Internal medicine, Endocrinology, Pulmonary hypertension, Hypoxia and Cardiology. His work is connected to Vasoconstriction, Hypoxic pulmonary vasoconstriction, Vasodilation, Lung and Pulmonary artery, as a part of Internal medicine. Ivan F. McMurtry works mostly in the field of Hypoxic pulmonary vasoconstriction, limiting it down to topics relating to RHOA and, in certain cases, Y-27632, as a part of the same area of interest.
In Endocrinology, Ivan F. McMurtry works on issues like Endothelin 1, which are connected to Endothelin receptor. His research in Pulmonary hypertension intersects with topics in Muscle hypertrophy, Anesthesia, Pharmacology and Rho-associated protein kinase. His Hypoxia research includes elements of Vascular smooth muscle, Blood pressure, Immunology, Fasudil and Perfusion.
Ivan F. McMurtry mainly investigates Internal medicine, Pulmonary hypertension, Hypoxia, Endocrinology and Cardiology. Ivan F. McMurtry has included themes like Knockout rat and Pathology in his Internal medicine study. His Pulmonary hypertension research is multidisciplinary, relying on both Stem cell, Lung, Pulmonary artery, Cell type and Pharmacology.
His work carried out in the field of Hypoxia brings together such families of science as Ventricle, Vasoconstriction, Fibrosis and Gene knockdown. His studies deal with areas such as Sphingosine and Kinase as well as Endocrinology. His work on Right ventricular hypertrophy, Perfusion and Retrograde perfusion is typically connected to Right ventricular structure as part of general Cardiology study, connecting several disciplines of science.
His primary areas of investigation include Pulmonary hypertension, Internal medicine, Hypoxia, Endocrinology and Pharmacology. Many of his studies involve connections with topics such as Progenitor cell and Pulmonary hypertension. As a part of the same scientific study, Ivan F. McMurtry usually deals with the Internal medicine, concentrating on Pathology and frequently concerns with Hemodynamics.
He studied Hypoxia and Fibrosis that intersect with Vasoconstriction, Sphingosine kinase 1 and SPHK2. His Endocrinology research is multidisciplinary, incorporating elements of Cell cycle, Heart failure and Immunology. His studies deal with areas such as Vasodilation, Permeability, Sildenafil, Hydrogen peroxide and PLGA as well as Pharmacology.
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Cellular and molecular basis of pulmonary arterial hypertension.
Nicholas W. Morrell;Serge Adnot;Stephen L. Archer;Jocelyn Dupuis.
Journal of the American College of Cardiology (2009)
Animal models of pulmonary arterial hypertension: the hope for etiological discovery and pharmacological cure
Kurt R. Stenmark;Barbara Meyrick;Nazzareno Galie;Wolter J. Mooi.
American Journal of Physiology-lung Cellular and Molecular Physiology (2009)
Role of endothelium-derived relaxing factor during transition of pulmonary circulation at birth.
S. H. Abman;B. A. Chatfield;S. L. Hall;I. F. McMurtry.
American Journal of Physiology-heart and Circulatory Physiology (1990)
Reperfusion after acute coronary occlusion in dogs impairs endothelium-dependent relaxation to acetylcholine and augments contractile reactivity in vitro.
K M VanBenthuysen;I F McMurtry;L D Horwitz.
Journal of Clinical Investigation (1987)
Inhibition of hypoxic pulmonary vasoconstriction by calcium antagonists in isolated rat lungs.
I F McMurtry;A B Davidson;J T Reeves;R F Grover.
Circulation Research (1976)
Granulocytes mediate acute edematous lung injury in rabbits and in isolated rabbit lungs perfused with phorbol myristate acetate: role of oxygen radicals.
D. Michael Shasby;Karyl M. Vanbenthuysen;Robert M. Tate;Sandra S. Shasby.
The American review of respiratory disease (2015)
Attenuation of acute hypoxic pulmonary vasoconstriction and hypoxic pulmonary hypertension in mice by inhibition of Rho-kinase.
Karen A. Fagan;Masahiko Oka;Natalie R. Bauer;Sarah A. Gebb.
American Journal of Physiology-lung Cellular and Molecular Physiology (2004)
The pulmonary circulation of homozygous or heterozygous eNOS-null mice is hyperresponsive to mild hypoxia
Karen A. Fagan;Brian W. Fouty;Robert C. Tyler;Kenneth G. Morris.
Journal of Clinical Investigation (1999)
Rho Kinase–Mediated Vasoconstriction Is Important in Severe Occlusive Pulmonary Arterial Hypertension in Rats
Masahiko Oka;Noriyuki Homma;Laimute Taraseviciene-Stewart;Kenneth G. Morris.
Circulation Research (2007)
Oxygen-Radical-Mediated Permeability Edema and Vasoconstriction in Isolated Perfused Rabbit Lungs1–4
Robert M. Tate;Karyl M. Vanbenthuysen;D. Michael Shasby;Ivan F. McMurtry.
The American review of respiratory disease (2015)
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