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Medicine

D-Index
91
Citations
42952
World Ranking
11558
National Ranking
5931

Overview

Govind Bhagat is affiliated with Columbia University in the United States and conducts research primarily in the field of Medicine. Their work spans several interconnected subfields, including Pathology and Forensic Medicine, Oncology, Immunology, Molecular Biology, and Genetics. This multidisciplinary approach supports a focus on hematological malignancies and immune-related disorders.

The scientist has contributed extensively to topics related to Lymphoma Diagnosis and Treatment, Viral-associated cancers and disorders, Chronic Lymphocytic Leukemia Research, Celiac Disease Research and Management, CAR-T cell therapy research, CNS Lymphoma Diagnosis and Treatment, and Immune Cell Function and Interaction.

Govind Bhagat has published articles in a range of scientific journals, with frequent contributions to Blood, Hematological Oncology, UNC Libraries, Leukemia, and The American Journal of Surgical Pathology. The number of publications in these venues reflects their sustained engagement in hematology and oncology research.

Recent notable papers authored by Govind Bhagat include:

  • The 5th edition of the World Health Organization Classification of Haematolymphoid Tumours: Lymphoid Neoplasms, 2022, Leukemia
  • Combined oral 5-azacytidine and romidepsin are highly effective in patients with PTCL: a multicenter phase 2 study, 2020, Blood
  • EBV-associated primary CNS lymphoma occurring after immunosuppression is a distinct immunobiological entity, 2020, Blood
  • DNA-PKcs has KU-dependent function in rRNA processing and haematopoiesis, 2020, Nature
  • Genetic and phenotypic attributes of splenic marginal zone lymphoma, 2021, Blood

Their work involves collaboration with frequent coauthors, reflecting a network of research partnerships. Notable frequent coauthors include Eric D. Hsi, Maurilio Ponzoni, Alexandar Tzankov, Carlo Visco, and April Chiu.

Best Publications

  • Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene

    Xueping Qu;Jie Yu;Govind Bhagat;Norihiko Furuya

  • Mutations of multiple genes cause deregulation of NF-κB in diffuse large B-cell lymphoma

    Mara Compagno;Wei Keat Lim;Adina Grunn;Subhadra V. Nandula

  • Analysis of the coding genome of diffuse large B-cell lymphoma

    Laura Pasqualucci;Vladimir Trifonov;Giulia Fabbri;Jing Ma

  • Mutational loss of PTEN induces resistance to NOTCH1 inhibition in T-cell leukemia

    Teresa Palomero;Maria Luisa Sulis;Maria Cortina;Pedro J Real

  • The DLEU2/miR-15a/16-1 cluster controls B cell proliferation and its deletion leads to chronic lymphocytic leukemia

    Ulf Klein;Marie Lia;Marta Crespo;Rachael Siegel

  • Coordinated Induction by IL15 of a TCR-Independent NKG2D Signaling Pathway Converts CTL into Lymphokine-Activated Killer Cells in Celiac Disease

    Bertrand Meresse;Zhangguo Chen;Cezary Ciszewski;Maria Tretiakova

  • Overexpression of Interleukin-1β Induces Gastric Inflammation and Cancer and Mobilizes Myeloid-Derived Suppressor Cells in Mice

    Shuiping Tu;Govind Bhagat;Guanglin Cui;Shigeo Takaishi

  • Nomenclature of the finer branches of the biliary tree: canals, ductules, and ductular reactions in human livers.

    Tania A. Roskams;Neil D. Theise;Charles Balabaud;Govind Bhagat

  • Akt-Mediated Regulation of Autophagy and Tumorigenesis Through Beclin 1 Phosphorylation

    Richard C Wang;Yongjie Wei;Zhenyi An;Zhongju Zou

  • Recurrent mutations in epigenetic regulators, rhoa and fyn kinase in peripheral t-cell lymphomas

    Adolfo Ferrando;Raul Rabadan;Teresa Palomero

  • Genetics of Follicular Lymphoma Transformation

    Laura Pasqualucci;Hossein Khiabanian;Marco Fangazio;Mansi Vasishtha

  • MYC/BCL2 protein coexpression contributes to the inferior survival of activated B-cell subtype of diffuse large B-cell lymphoma and demonstrates high-risk gene expression signatures: a report from The International DLBCL Rituximab-CHOP Consortium Program

    Shimin Hu;Zijun Y. Xu-Monette;Alexander Tzankov;Tina Green

  • EGFR-Mediated Beclin 1 Phosphorylation in Autophagy Suppression, Tumor Progression, and Tumor Chemoresistance

    Yongjie Wei;Zhongju Zou;Nils Becker;Matthew Anderson

  • Leukaemogenesis induced by an activating β-catenin mutation in osteoblasts

    Aruna Kode;John S. Manavalan;Ioanna Mosialou;Govind Bhagat

  • Disruption of the beclin 1–BCL2 autophagy regulatory complex promotes longevity in mice

    Álvaro F. Fernández;Salwa Sebti;Yongjie Wei;Zhongju Zou

  • Bile Acid and Inflammation Activate Gastric Cardia Stem Cells in a Mouse Model of Barrett-Like Metaplasia

    Michael Quante;Michael Quante;Govind Bhagat;Julian A. Abrams;Frederic Marache

  • Risk of malignancy in patients with celiac disease

    Peter H.R Green;Aaron T Fleischauer;Govind Bhagat;Rishi Goyal

  • Combined Genetic Inactivation of β2-Microglobulin and CD58 Reveals Frequent Escape from Immune Recognition in Diffuse Large B Cell Lymphoma

    Madhavi Challa-Malladi;Yen K. Lieu;Olivia Califano;Antony B. Holmes

  • A Signaling Pathway Mediating Downregulation of BCL6 in Germinal Center B Cells Is Blocked by BCL6 Gene Alterations in B Cell Lymphoma

    Masumichi Saito;Jie Gao;Katia Basso;Yukiko Kitagawa

  • Breast Implant–Associated Anaplastic Large-Cell Lymphoma: Long-Term Follow-Up of 60 Patients

    Roberto N. Miranda;Tariq N. Aladily;H. Miles Prince;Rashmi Kanagal-Shamanna

Frequent Co-Authors

Peter H.R. Green
Peter H.R. Green Columbia University
Maurilio Ponzoni
Maurilio Ponzoni Vita-Salute San Raffaele University
Miguel A. Piris
Miguel A. Piris Instituto de Salud Carlos III
Eric D. Hsi
Eric D. Hsi Cleveland Clinic
Andrés J.M. Ferreri
Andrés J.M. Ferreri Vita-Salute San Raffaele University
L. Jeffrey Medeiros
L. Jeffrey Medeiros The University of Texas MD Anderson Cancer Center
Youli Zu
Youli Zu Houston Methodist
Vundavalli V. Murty
Vundavalli V. Murty Columbia University Medical Center
Santiago Montes-Moreno
Santiago Montes-Moreno Instituto de Investigación Marqués de Valdecilla
Yong Li
Yong Li Baylor College of Medicine

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