Gerd R. Pape mainly investigates Immunology, Virology, Hepatitis C virus, Virus and Immune system. He studies Viral disease, a branch of Immunology. His Viral disease research incorporates themes from Interferon and Peripheral blood mononuclear cell.
The study incorporates disciplines such as Cytotoxic T cell and Antigen in addition to Virology. His research integrates issues of Hepatitis C, CD8 and Interferon gamma in his study of Hepatitis C virus. His Virus study often links to related topics such as Chronic infection.
The scientist’s investigation covers issues in Virology, Immunology, Hepatitis C virus, Virus and Internal medicine. Gerd R. Pape combines subjects such as Epitope, CD8 and Antigen with his study of Virology. His Immunology study typically links adjacent topics like Cytotoxic T cell.
His Hepatitis C virus study combines topics in areas such as Interferon gamma, Cd4 t cell and Viral load. His Virus research includes themes of Chronic infection and Peripheral blood mononuclear cell. He has included themes like Gastroenterology, Ribavirin and Surgery in his Internal medicine study.
Gerd R. Pape spends much of his time researching Virology, Hepatitis C virus, Immunology, Ribavirin and Virus. Gerd R. Pape has researched Virology in several fields, including Epitope, Antigen and Receptor. His Hepatitis C virus study focuses on Hepacivirus in particular.
His Immunology study frequently draws connections to other fields, such as Presentation. His Ribavirin study also includes
Gerd R. Pape mainly focuses on Hepatitis C virus, Immunology, Hepacivirus, Virus and Virology. Gerd R. Pape is interested in Ribavirin, which is a branch of Hepatitis C virus. His study in Hepatitis C and Immune system is carried out as part of his Immunology studies.
His study explores the link between Hepacivirus and topics such as Epitope that cross with problems in NS2-3 protease, Viral hepatitis and Cytotoxic T cell. In most of his Virus studies, his work intersects topics such as Interferon. In his study, Antigen presentation and Cell biology is strongly linked to Antigen, which falls under the umbrella field of Virology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Memory CD8+ T cells vary in differentiation phenotype in different persistent virus infections.
Victor Appay;P. Rod Dunbar;Margaret Callan;Paul Klenerman.
Nature Medicine (2002)
Recurrence of hepatitis C virus after loss of virus-specific CD4(+) T-cell response in acute hepatitis C.
J.Tilman Gerlach;Helmut M. Diepolder;‡ Maria–Christina Jung;Norbert H. Gruener.
Possible mechanism involving T-lymphocyte response to non-structural protein 3 in viral clearance in acute hepatitis C virus infection
H.M. Diepolder;R. Zachoval;R.M. Hoffmann;M-C. Jung.
The Lancet (1995)
Polymorphic structure of the tumor necrosis factor (TNF) locus: an NcoI polymorphism in the first intron of the human TNF-beta gene correlates with a variant amino acid in position 26 and a reduced level of TNF-beta production.
Gerald Messer;Ulrich Spengler;Maria C. Jung;G. Honold.
Journal of Experimental Medicine (1991)
Acute Hepatitis C: High Rate of Both Spontaneous and Treatment-Induced Viral Clearance
J.Tilman Gerlach;Helmut M Diepolder;Reinhart Zachoval;Norbert H Gruener.
Extended treatment duration for hepatitis C virus type 1: comparing 48 versus 72 weeks of peginterferon-alfa-2a plus ribavirin.
Thomas Berg;Michael von Wagner;Samer Nasser;Christoph Sarrazin.
Sustained Dysfunction of Antiviral CD8+ T Lymphocytes after Infection with Hepatitis C Virus
Norbert H. Gruener;Franziska Lechner;Maria-Christina Jung;Helmut Diepolder.
Journal of Virology (2001)
Ursodeoxycholic acid for treatment of primary sclerosing cholangitis: A placebo‐controlled trial
Ulrich Beuers;Ulrich Spengler;Wolfgang Kruis;ülker Aydemir.
Association of Hepatitis C Virus—Specific CD8+ T Cells with Viral Clearance in Acute Hepatitis C
Norbert H. Grüner;Tilman J. Gerlach;Maria-Christina Jung;Helmut M. Diepolder.
The Journal of Infectious Diseases (2000)
Immunodominant CD4+ T-cell epitope within nonstructural protein 3 in acute hepatitis C virus infection.
H M Diepolder;J T Gerlach;R Zachoval;R M Hoffmann.
Journal of Virology (1997)
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