D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 55 Citations 10,169 252 World Ranking 2734 National Ranking 1103

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Disease

His primary scientific interests are in Phagocytosis, Immunology, Receptor, Microbiology and Protein kinase A. His Phagocytosis research also works with subjects such as

  • Alveolar macrophage, which have a strong connection to Endocrinology, Internal medicine, Pneumococcal pneumonia and Streptococcus pneumoniae,
  • Prostacyclin which intersects with area such as In vivo, Immunity and Transplantation. The various areas that he examines in his Immunology study include Vasopressin, Angiotensin II, Cell signaling and Lung.

His work deals with themes such as Tumor necrosis factor alpha, Prostaglandin E2, Prostaglandin E, Signal transduction and Systemic inflammation, which intersect with Receptor. His work on Clostridium difficile as part of his general Microbiology study is frequently connected to Macrophage receptor with collagenous structure, thereby bridging the divide between different branches of science. The concepts of his Protein kinase A study are interwoven with issues in NADPH oxidase and Lipid signaling.

His most cited work include:

  • Prostaglandin E2 Inhibits Alveolar Macrophage Phagocytosis through an E-Prostanoid 2 Receptor-Mediated Increase in Intracellular Cyclic AMP (278 citations)
  • Cyclic AMP: master regulator of innate immune cell function. (255 citations)
  • Fever and hypothermia in systemic inflammation: recent discoveries and revisions. (254 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Microbiology, Immunology, Internal medicine, Clostridium difficile and Receptor. His biological study spans a wide range of topics, including Streptococcus, Streptococcus agalactiae and Virulence. David M. Aronoff combines subjects such as Gastroenterology, Endocrinology and Surgery with his study of Internal medicine.

His research in Clostridium difficile intersects with topics in Gut flora, Colitis, Colectomy, Ribotyping and Diarrhea. In his research on the topic of Receptor, Biochemistry is strongly related with Cell biology. As a part of the same scientific study, David M. Aronoff usually deals with the Phagocytosis, concentrating on Alveolar macrophage and frequently concerns with Pneumococcal pneumonia.

He most often published in these fields:

  • Microbiology (24.07%)
  • Immunology (21.02%)
  • Internal medicine (16.95%)

What were the highlights of his more recent work (between 2018-2021)?

  • Microbiology (24.07%)
  • Innate immune system (8.14%)
  • Streptococcus (7.80%)

In recent papers he was focusing on the following fields of study:

His main research concerns Microbiology, Innate immune system, Streptococcus, Macrophage and Immunology. His Microbiology study combines topics from a wide range of disciplines, such as Chorioamnionitis and Neonatal sepsis. As a member of one scientific family, David M. Aronoff mostly works in the field of Innate immune system, focusing on Virulence and, on occasion, Genotype, Phagocytosis and Antibody.

His Macrophage research integrates issues from Proinflammatory cytokine, Pathogen and Streptococcus agalactiae. His research links Clostridium difficile with Immunology. His Clostridium difficile study combines topics in areas such as Cyclooxygenase and Colitis.

Between 2018 and 2021, his most popular works were:

  • Masks and Coronavirus Disease 2019 (COVID-19). (29 citations)
  • Sex modifies placental gene expression in response to metabolic and inflammatory stress. (21 citations)
  • Food Safety and COVID-19. (20 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • Disease

David M. Aronoff mainly investigates 2019-20 coronavirus outbreak, Betacoronavirus, Coronavirus disease 2019, Immunology and Antibiotics. The study incorporates disciplines such as Pandemic and Severe acute respiratory syndrome coronavirus 2 in addition to 2019-20 coronavirus outbreak. Immunology is often connected to Clostridium difficile in his work.

David M. Aronoff interconnects Cyclooxygenase, Infectious Colitis, Gastrointestinal tract and Intestinal permeability in the investigation of issues within Clostridium difficile. His Antibiotics research is multidisciplinary, incorporating perspectives in Diabetic foot infections and Intensive care medicine. His Microbiology study which covers Virulence that intersects with Immune system.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Prostaglandin E2 Inhibits Alveolar Macrophage Phagocytosis through an E-Prostanoid 2 Receptor-Mediated Increase in Intracellular Cyclic AMP

David M. Aronoff;Claudio Canetti;Marc Peters-Golden.
Journal of Immunology (2004)

401 Citations

Cyclic AMP: master regulator of innate immune cell function.

Carlos H. Serezani;Megan N. Ballinger;David M. Aronoff;Marc Peters-Golden.
American Journal of Respiratory Cell and Molecular Biology (2008)

397 Citations

Fever and hypothermia in systemic inflammation: recent discoveries and revisions.

Andrej A Romanovsky;Maria C Almeida;David M Aronoff;Andrei I Ivanov.
Frontiers in Bioscience (2005)

385 Citations

Antipyretics: mechanisms of action and clinical use in fever suppression

David M Aronoff;Eric G Neilson.
The American Journal of Medicine (2001)

373 Citations

Determinants of the cellular specificity of acetaminophen as an inhibitor of prostaglandin H(2) synthases.

Olivier Boutaud;David M. Aronoff;Jacob H. Richardson;Lawrence J. Marnett.
Proceedings of the National Academy of Sciences of the United States of America (2002)

276 Citations

Cutting edge: macrophage inhibition by cyclic AMP (cAMP): differential roles of protein kinase A and exchange protein directly activated by cAMP-1.

David M. Aronoff;Claudio Canetti;Carlos H. Serezani;Carlos H. Serezani;Ming Luo.
Journal of Immunology (2005)

272 Citations

Microbiome Data Distinguish Patients with Clostridium difficile Infection and Non-C. difficile-Associated Diarrhea from Healthy Controls

Alyxandria M. Schubert;Mary A. M. Rogers;Cathrin Ring;Jill Mogle.
Mbio (2014)

266 Citations

New insights into the mechanism of action of acetaminophen: Its clinical pharmacologic characteristics reflect its inhibition of the two prostaglandin H2 synthases

David M. Aronoff;David M. Aronoff;John A. Oates;John A. Oates;Olivier Boutaud;Olivier Boutaud.
Clinical Pharmacology & Therapeutics (2006)

264 Citations

The influence of non-steroidal anti-inflammatory drugs on the gut microbiome

Mary A.M. Rogers;David M. Aronoff.
Clinical Microbiology and Infection (2016)

254 Citations

Epidemiology of Clostridium difficile infection.

Daryl D. DePestel;David M. Aronoff.
Journal of Pharmacy Practice (2013)

251 Citations

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