His main research concerns Coronavirus, Cell biology, Virology, Viral replication and Mouse hepatitis virus. His Cell biology research includes themes of Lipid bilayer fusion, Endocytosis, Cell fusion and Furin. His work carried out in the field of Lipid bilayer fusion brings together such families of science as Viral entry, Peptide sequence and Peptide.
His Furin research incorporates themes from Protein A and Fusion protein. His work in the fields of Virology, such as Virus, overlaps with other areas such as Nidovirales. His Viral replication study combines topics from a wide range of disciplines, such as Downregulation and upregulation, Point mutation, Molecular biology, Endoplasmic reticulum and Secretory pathway.
Cornelis A. M. de Haan mostly deals with Virology, Virus, Coronavirus, Cell biology and Viral replication. His Virology study incorporates themes from Epitope, Receptor and Immune system. His work deals with themes such as Innate immune system, Antibody and Vaccination, which intersect with Virus.
His Cell biology research includes elements of Viral envelope, Lipid bilayer fusion and Molecular biology. As a part of the same scientific study, Cornelis A. M. de Haan usually deals with the Lipid bilayer fusion, concentrating on Fusion protein and frequently concerns with Heptad repeat. His studies deal with areas such as RNA virus, Downregulation and upregulation, Transcription and Live cell imaging as well as Viral replication.
His primary scientific interests are in Influenza A virus, Neuraminidase, Virus, Virology and Hemagglutinin. His Influenza A virus study frequently involves adjacent topics like Biochemistry. His Neuraminidase research is multidisciplinary, incorporating elements of Sialic acid, Sialic acid binding and Microbiology.
His Virus research focuses on subjects like Innate immune system, which are linked to Cell activation and Viral entry. Many of his research projects under Virology are closely connected to Hemagglutination assay with Hemagglutination assay, tying the diverse disciplines of science together. He performs multidisciplinary study in Viral replication and Coronavirus in his work.
Cornelis A. M. de Haan mainly investigates Influenza A virus, Neuraminidase, Sialic acid, Hemagglutinin-neuraminidase and Cell biology. Influenza A virus is a subfield of Virus that he tackles. He is interested in Viral replication, which is a field of Virus.
The various areas that Cornelis A. M. de Haan examines in his Neuraminidase study include Hemagglutinin, Tropism and Virus genetics. His work on Secretory pathway is typically connected to Small GTPase as part of general Cell biology study, connecting several disciplines of science. Cornelis A. M. de Haan combines Mouse hepatitis virus and Virology in his research.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)
The Coronavirus Spike Protein Is a Class I Virus Fusion Protein: Structural and Functional Characterization of the Fusion Core Complex
Berend Jan Bosch;Ruurd van der Zee;Cornelis A. M. de Haan;Peter J. M. Rottier.
Journal of Virology (2003)
Molecular interactions in the assembly of coronaviruses.
Cornelis A M de Haan;Peter J M Rottier.
Advances in Virus Research (2005)
Dissection of the Influenza A Virus Endocytic Routes Reveals Macropinocytosis as an Alternative Entry Pathway
Erik de Vries;Donna M. Tscherne;Marleen J. Wienholts;Viviana Cobos-Jiménez.
PLOS Pathogens (2011)
Coronaviruses Hijack the LC3-I-Positive EDEMosomes, ER-Derived Vesicles Exporting Short-Lived ERAD Regulators, for Replication
Fulvio Reggiori;Iryna Monastyrska;Monique H. Verheije;Tito Calì.
Cell Host & Microbe (2010)
Coronavirus cell entry occurs through the endo-/lysosomal pathway in a proteolysis-dependent manner.
Christine Burkard;Monique H. Verheije;Oliver Wicht;Sander I. van Kasteren.
PLOS Pathogens (2014)
The group-specific murine coronavirus genes are not essential, but their deletion, by reverse genetics, is attenuating in the natural host.
Cornelis A.M. de Haan;Paul S. Masters;Xiaolan Shen;Susan Weiss.
Virology (2002)
Assembly of the coronavirus envelope: homotypic interactions between the M proteins.
Cornelis A. M. de Haan;Harry Vennema;Peter J. M. Rottier.
Journal of Virology (2000)
Impaired Antibody-mediated Protection and Defective IgA B-Cell Memory in Experimental Infection of Adults with Respiratory Syncytial Virus.
Maximillian S. Habibi;Agnieszka Jozwik;Spyridon Makris;Jake Dunning.
American Journal of Respiratory and Critical Care Medicine (2015)
Cleavage inhibition of the murine coronavirus spike protein by a furin-like enzyme affects cell-cell but not virus-cell fusion.
Cornelis A. M. de Haan;Konrad Stadler;Gert-Jan Godeke;Berend Jan Bosch.
Journal of Virology (2004)
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