World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
48
Citations
8491
World Ranking
18381
National Ranking
1442

Charles Redwood publication distribution in Biology and Biochemistry in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Biology and Biochemistry in 2026. The highlighted bar marks where Charles Redwood sits on this spectrum.

47–56 publications: 8 scientists 57–66 publications: 35 scientists 67–76 publications: 106 scientists 77–86 publications: 231 scientists 87–96 publications: 414 scientists 97–106 publications: 546 scientists 107–116 publications: 704 scientists 117–126 publications: 849 scientists 127–136 publications: 980 scientists 137–146 publications: 942 scientists 147–156 publications: 969 scientists 157–166 publications: 950 scientists 167–176 publications: 951 scientists 177–186 publications: 915 scientists 187–196 publications: 787 scientists 197–206 publications: 841 scientists 207–216 publications: 735 scientists 217–226 publications: 709 scientists 227–236 publications: 651 scientists 237–246 publications: 605 scientists 247–256 publications: 510 scientists 257–266 publications: 524 scientists 267–276 publications: 434 scientists 277–286 publications: 418 scientists 287–296 publications: 350 scientists 297–306 publications: 363 scientists 307–316 publications: 315 scientists 317–326 publications: 296 scientists 327–336 publications: 261 scientists 337–346 publications: 240 scientists 347–356 publications: 219 scientists 357–366 publications: 197 scientists 367–376 publications: 154 scientists 377–386 publications: 161 scientists 387–396 publications: 155 scientists 397–406 publications: 145 scientists 407–416 publications: 124 scientists 417–426 publications: 112 scientists 427–436 publications: 132 scientists 437–446 publications: 116 scientists 447–456 publications: 99 scientists 457–466 publications: 81 scientists 467–476 publications: 91 scientists 477–486 publications: 80 scientists 487–496 publications: 80 scientists 497–506 publications: 60 scientists 507–516 publications: 36 scientists 517–526 publications: 46 scientists 527–536 publications: 54 scientists 537–546 publications: 44 scientists 547–556 publications: 43 scientists 557–566 publications: 43 scientists 567–576 publications: 42 scientists 577–586 publications: 25 scientists 587–596 publications: 34 scientists 597–606 publications: 23 scientists 607–616 publications: 33 scientists 617–626 publications: 31 scientists 627–636 publications: 27 scientists 637–646 publications: 25 scientists 647–656 publications: 28 scientists 657–666 publications: 34 scientists 667–676 publications: 18 scientists 677–686 publications: 16 scientists 687–696 publications: 10 scientists 697–706 publications: 12 scientists 707–716 publications: 21 scientists 717–726 publications: 12 scientists 727–736 publications: 12 scientists 737–746 publications: 10 scientists 747–756 publications: 7 scientists 757–766 publications: 13 scientists 767–776 publications: 15 scientists 777–786 publications: 13 scientists 787–796 publications: 9 scientists 797–806 publications: 9 scientists 807–816 publications: 7 scientists 817–826 publications: 4 scientists 827–836 publications: 9 scientists 837–846 publications: 7 scientists 847–856 publications: 3 scientists 857–866 publications: 5 scientists 867–876 publications: 5 scientists 877–886 publications: 11 scientists 887–896 publications: 3 scientists 897–906 publications: 4 scientists 907–916 publications: 7 scientists 917–926 publications: 5 scientists 927–936 publications: 6 scientists 937–946 publications: 6 scientists 947–956 publications: 3 scientists 957–966 publications: 7 scientists 967–976 publications: 2 scientists 977–986 publications: 2 scientists 987–996 publications: 1 scientists 997–1,006 publications: 5 scientists 1,007–1,016 publications: 2 scientists 1,017–1,026 publications: 2 scientists 1,027 publications: 1 scientists 1,028+ publications: 100 scientists
47 publications 1,028+

This scientist: 172 publications — 37th percentile

37% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 1,028 publications or more.

Charles Redwood D-index placement in Biology and Biochemistry in 2026

The chart shows the D-index (discipline H-index) distribution of Biology and Biochemistry scientists ranked by Research.com in 2026. The highlighted bar marks where Charles Redwood sits on this spectrum.

40–41 D-Index: 80 scientists 42–43 D-Index: 183 scientists 44–45 D-Index: 317 scientists 46–47 D-Index: 504 scientists 48–49 D-Index: 718 scientists 50–51 D-Index: 900 scientists 52–53 D-Index: 1,026 scientists 54–55 D-Index: 1,150 scientists 56–57 D-Index: 1,236 scientists 58–59 D-Index: 1,253 scientists 60–61 D-Index: 1,163 scientists 62–63 D-Index: 1,131 scientists 64–65 D-Index: 1,032 scientists 66–67 D-Index: 897 scientists 68–69 D-Index: 814 scientists 70–71 D-Index: 715 scientists 72–73 D-Index: 709 scientists 74–75 D-Index: 596 scientists 76–77 D-Index: 512 scientists 78–79 D-Index: 473 scientists 80–81 D-Index: 412 scientists 82–83 D-Index: 373 scientists 84–85 D-Index: 358 scientists 86–87 D-Index: 285 scientists 88–89 D-Index: 273 scientists 90–91 D-Index: 227 scientists 92–93 D-Index: 208 scientists 94–95 D-Index: 193 scientists 96–97 D-Index: 153 scientists 98–99 D-Index: 157 scientists 100–101 D-Index: 148 scientists 102–103 D-Index: 120 scientists 104–105 D-Index: 113 scientists 106–107 D-Index: 100 scientists 108–109 D-Index: 86 scientists 110–111 D-Index: 67 scientists 112–113 D-Index: 72 scientists 114–115 D-Index: 73 scientists 116–117 D-Index: 64 scientists 118–119 D-Index: 53 scientists 120–121 D-Index: 60 scientists 122–123 D-Index: 54 scientists 124–125 D-Index: 43 scientists 126–127 D-Index: 38 scientists 128–129 D-Index: 49 scientists 130–131 D-Index: 26 scientists 132–133 D-Index: 18 scientists 134–135 D-Index: 23 scientists 136–137 D-Index: 32 scientists 138–139 D-Index: 32 scientists 140–141 D-Index: 27 scientists 142–143 D-Index: 19 scientists 144–145 D-Index: 22 scientists 146–147 D-Index: 12 scientists 148–149 D-Index: 16 scientists 150–151 D-Index: 14 scientists 152–153 D-Index: 10 scientists 154–155 D-Index: 13 scientists 156–157 D-Index: 10 scientists 158–159 D-Index: 7 scientists 160–161 D-Index: 9 scientists 162–163 D-Index: 13 scientists 164–165 D-Index: 4 scientists 166 D-Index: 4 scientists 167+ D-Index: 98 scientists
40 D-Index 167+

This scientist: 48 D-Index — 7th percentile

7% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 167 D-Index or more.

Overview

Charles Redwood is affiliated with the University of Oxford in the United Kingdom. Their primary research spans several domains within medicine and molecular biology, with a focus on cardiology and cardiovascular medicine.

The main fields of study in Redwood's research include:

  • Medicine
  • Biochemistry, Genetics and Molecular Biology

Within these disciplines, Redwood's work concentrates on several subfields, notably:

  • Cardiology and Cardiovascular Medicine
  • Molecular Biology
  • Cellular and Molecular Neuroscience
  • Hematology
  • Plant Science

Redwood's research topics consistently address a range of specialized themes such as:

  • Cardiomyopathy and Myosin Studies
  • Cardiovascular Effects of Exercise
  • Muscle Physiology and Disorders
  • Viral Infections and Immunology Research
  • Cardiac Electrophysiology and Arrhythmias
  • Neuroscience and Neural Engineering
  • Ion Channel Regulation and Function

The scientist has authored multiple papers published in prominent journals. Recent highly cited publications include:

  • Myosin Sequestration Regulates Sarcomere Function, Cardiomyocyte Energetics, and Metabolism, Informing the Pathogenesis of Hypertrophic Cardiomyopathy (2020, Circulation)
  • Paracrine signalling by cardiac calcitonin controls atrial fibrogenesis and arrhythmia (2020, Nature)
  • Mavacamten rescues increased myofilament calcium sensitivity and dysregulation of Ca2+ flux caused by thin filament hypertrophic cardiomyopathy mutations (2020, American Journal of Physiology-Heart and Circulatory Physiology)
  • CalTrack: High-Throughput Automated Calcium Transient Analysis in Cardiomyocytes (2021, Circulation Research)
  • Comparing the effects of chemical Ca2+ dyes and R-GECO on contractility and Ca2+ transients in adult and human iPSC cardiomyocytes (2023, Journal of Molecular and Cellular Cardiology)

Redwood frequently publishes in leading journals such as:

  • International Journal of Molecular Sciences
  • bioRxiv (Cold Spring Harbor Laboratory)
  • American Journal of Physiology-Heart and Circulatory Physiology
  • Circulation
  • Nature

Collaboration is a significant aspect of Redwood's research activities. Frequent co-authors include:

  • Hugh Watkins
  • Alexander J. Sparrow
  • Paul Robinson
  • Yurii S. Borovikov
  • Olga E. Karpicheva

Best Publications

  • Mutations in the γ2 subunit of AMP-activated protein kinase cause familial hypertrophic cardiomyopathy: evidence for the central role of energy compromise in disease pathogenesis

    Edward Blair;Charles Redwood;Houman Ashrafian;Marisa Oliveira

  • Fumarate is cardioprotective via activation of the Nrf2 antioxidant pathway.

    Houman Ashrafian;Gabor Czibik;Mohamed Bellahcene;Dunja Aksentijević

  • The molecular anatomy of caldesmon.

    S B Marston;C S Redwood

  • Dilated and Hypertrophic Cardiomyopathy Mutations in Troponin and α-Tropomyosin Have Opposing Effects on the Calcium Affinity of Cardiac Thin Filaments

    Paul Robinson;Peter J. Griffiths;Hugh Watkins;Charles S. Redwood

  • Properties of mutant contractile proteins that cause hypertrophic cardiomyopathy

    Charles S. Redwood;Johanna C. Moolman-Smook;Johanna C. Moolman-Smook;Hugh Watkins

  • Physiological regulation of eukaryotic topoisomerase II.

    Richard J Isaacs;Sally L Davies;M.Ines Sandri;Charles Redwood

  • Myosin Sequestration Regulates Sarcomere Function, Cardiomyocyte Energetics, and Metabolism, Informing the Pathogenesis of Hypertrophic Cardiomyopathy

    Christopher N. Toepfer;Christopher N. Toepfer;Amanda C. Garfinkel;Gabriela Venturini;Gabriela Venturini;Hiroko Wakimoto

  • Altered regulatory properties of human cardiac troponin I mutants that cause hypertrophic cardiomyopathy.

    Kathryn Elliott;Hugh Watkins;Charles S. Redwood

  • Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype.

    Mahmooda Mirza;Steven Marston;Ruth Willott;Christopher Ashley

  • A Mutant Tropomyosin That Causes Hypertrophic Cardiomyopathy Is Expressed In Vivo and Associated With an Increased Calcium Sensitivity

    R. Bottinelli;D. A. Coviello;C. S. Redwood;M. A. Pellegrino

  • Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy.

    Paul Robinson;Mahmooda Mirza;Adam Knott;Hassan Abdulrazzak

  • Mutations in the gamma2 subunit of AMP-activated protein kinase cause familial hypertrophic cardiomyopathy: evidence for the central role of energy compromise in disease pathogenesis.

    Edward Blair;Charles Redwood;Houman Ashrafian;Marisa Oliveira

  • Hypertrophic cardiomyopathy mutations in MYBPC3 dysregulate myosin.

    Christopher N. Toepfer;Christopher N. Toepfer;Hiroko Wakimoto;Hiroko Wakimoto;Amanda C. Garfinkel;Barbara McDonough

  • Familial Dilated Cardiomyopathy Caused by an Alpha-Tropomyosin Mutation: The Distinctive Natural History of Sarcomeric Dilated Cardiomyopathy

    Neal K. Lakdawala;Lisa Dellefave;Charles S. Redwood;Elizabeth Sparks

  • Mutations in fast skeletal troponin I, troponin T, and beta-tropomyosin that cause distal arthrogryposis all increase contractile function.

    Paul Robinson;Simon Lipscomb;Laura C. Preston;Elissa Altin

  • Mutations of the light meromyosin domain of the beta-myosin heavy chain rod in hypertrophic cardiomyopathy.

    Edward Blair;Charles Redwood;Marisa de Jesus Oliveira;J.C. Moolman-Smook

  • The essential role of tropomyosin in cooperative regulation of smooth muscle thin filament activity by caldesmon.

    S.B. Marston;C.S. Redwood

  • Effects of two hypertrophic cardiomyopathy mutations in alpha-tropomyosin, Asp175Asn and Glu180Gly, on Ca2+ regulation of thin filament motility.

    Wu Bing;Charles S. Redwood;Charles S. Redwood;Ian F. Purcell;Ian F. Purcell;Giovanna Esposito;Giovanna Esposito

  • Alpha-tropomyosin mutations in inherited cardiomyopathies

    Charles Redwood;Paul Robinson

  • Identification and Functional Characterization of Cardiac Troponin I As a Novel Disease Gene in Autosomal Dominant Dilated Cardiomyopathy

    Sebastian Carballo;Paul Robinson;Robyn Otway;Diane Fatkin

Frequent Co-Authors

Hugh Watkins
Hugh Watkins University of Oxford
Barbara Casadei
Barbara Casadei University of Oxford
Steven B. Marston
Steven B. Marston National Institutes of Health
Christine E. Seidman
Christine E. Seidman Harvard University
David Carling
David Carling Imperial College London
Cristobal G. dos Remedios
Cristobal G. dos Remedios Victor Chang Cardiac Research Institute
Jonathan G. Seidman
Jonathan G. Seidman Harvard University
Dominic J. Wells
Dominic J. Wells Royal Veterinary College
William J. McKenna
William J. McKenna University College London
Thomas Eschenhagen
Thomas Eschenhagen Universität Hamburg

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