What is he best known for?
The fields of study he is best known for:
Steven B. Marston spends much of his time researching Actin, Troponin, Molecular biology, Tropomyosin and Mutant.
His study in Actin is interdisciplinary in nature, drawing from both Myosin, Cardiomyopathy, Troponin I and Skeletal muscle.
The concepts of his Troponin I study are interwoven with issues in Endocrinology and Troponin T.
As a part of the same scientific study, Steven B. Marston usually deals with the Troponin, concentrating on Microfilament and frequently concerns with Myosin binding, Gene isoform and Nebulin.
Steven B. Marston combines subjects such as Mutation, Myopathy, Calcium and Congenital myopathy with his study of Tropomyosin.
His studies deal with areas such as Hypertrophic cardiomyopathy and ATPase as well as Mutant.
His most cited work include:
- The molecular anatomy of caldesmon. (188 citations)
- Troponin phosphorylation and regulatory function in human heart muscle: Dephosphorylation of Ser23/24 on troponin I could account for the contractile defect in end-stage heart failure (148 citations)
- Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype. (129 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Actin, Internal medicine, Tropomyosin, Biochemistry and Troponin.
His research in Actin intersects with topics in Caldesmon, Biophysics, Myosin, Molecular biology and Skeletal muscle.
Steven B. Marston usually deals with Molecular biology and limits it to topics linked to Mutant and Mutation and Heat shock protein.
His Internal medicine research integrates issues from Endocrinology and Cardiology.
His research integrates issues of Actin-binding protein, Calcium, Nemaline myopathy and Microfilament in his study of Tropomyosin.
In Troponin, he works on issues like Troponin I, which are connected to Phosphorylation, Troponin T and Myofilament.
He most often published in these fields:
- Actin (51.69%)
- Internal medicine (30.93%)
- Tropomyosin (27.54%)
What were the highlights of his more recent work (between 2011-2021)?
- Actin (51.69%)
- Troponin I (24.15%)
- Phosphorylation (23.73%)
In recent papers he was focusing on the following fields of study:
Actin, Troponin I, Phosphorylation, Internal medicine and Troponin are his primary areas of study.
His Actin research is multidisciplinary, relying on both Mutation, Molecular biology and Skeletal muscle.
His study looks at the intersection of Phosphorylation and topics like Biophysics with Peptide, Crystallography and Muscle contraction.
His Internal medicine study combines topics from a wide range of disciplines, such as Endocrinology and Cardiology.
As part of the same scientific family, he usually focuses on Troponin, concentrating on Cardiac muscle and intersecting with Myosin.
His Tropomyosin study incorporates themes from Actin cytoskeleton and Nemaline myopathy.
Between 2011 and 2021, his most popular works were:
- Atomic model of the human cardiac muscle myosin filament (93 citations)
- How do MYBPC3 mutations cause hypertrophic cardiomyopathy (72 citations)
- Mutation Update and Genotype-Phenotype Correlations of Novel and Previously Described Mutations in TPM2 and TPM3 Causing Congenital Myopathies (70 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
His primary areas of investigation include Actin, Tropomyosin, Internal medicine, Phosphorylation and Endocrinology.
His Actin research is multidisciplinary, incorporating elements of Troponin I, Mutation, Troponin, Myopathy and Muscle contraction.
As a member of one scientific family, Steven B. Marston mostly works in the field of Troponin, focusing on Troponin T and, on occasion, Actin cytoskeleton.
His Tropomyosin study is related to the wider topic of Biochemistry.
Specifically, his work in Internal medicine is concerned with the study of Myofibril.
His work in Endocrinology tackles topics such as Hypertrophic cardiomyopathy which are related to areas like Energy metabolism, Mutation, Papillary muscle and Human heart.
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