Steven B. Marston spends much of his time researching Actin, Troponin, Molecular biology, Tropomyosin and Mutant. His study in Actin is interdisciplinary in nature, drawing from both Myosin, Cardiomyopathy, Troponin I and Skeletal muscle. The concepts of his Troponin I study are interwoven with issues in Endocrinology and Troponin T.
As a part of the same scientific study, Steven B. Marston usually deals with the Troponin, concentrating on Microfilament and frequently concerns with Myosin binding, Gene isoform and Nebulin. Steven B. Marston combines subjects such as Mutation, Myopathy, Calcium and Congenital myopathy with his study of Tropomyosin. His studies deal with areas such as Hypertrophic cardiomyopathy and ATPase as well as Mutant.
His primary areas of study are Actin, Internal medicine, Tropomyosin, Biochemistry and Troponin. His research in Actin intersects with topics in Caldesmon, Biophysics, Myosin, Molecular biology and Skeletal muscle. Steven B. Marston usually deals with Molecular biology and limits it to topics linked to Mutant and Mutation and Heat shock protein.
His Internal medicine research integrates issues from Endocrinology and Cardiology. His research integrates issues of Actin-binding protein, Calcium, Nemaline myopathy and Microfilament in his study of Tropomyosin. In Troponin, he works on issues like Troponin I, which are connected to Phosphorylation, Troponin T and Myofilament.
Actin, Troponin I, Phosphorylation, Internal medicine and Troponin are his primary areas of study. His Actin research is multidisciplinary, relying on both Mutation, Molecular biology and Skeletal muscle. His study looks at the intersection of Phosphorylation and topics like Biophysics with Peptide, Crystallography and Muscle contraction.
His Internal medicine study combines topics from a wide range of disciplines, such as Endocrinology and Cardiology. As part of the same scientific family, he usually focuses on Troponin, concentrating on Cardiac muscle and intersecting with Myosin. His Tropomyosin study incorporates themes from Actin cytoskeleton and Nemaline myopathy.
His primary areas of investigation include Actin, Tropomyosin, Internal medicine, Phosphorylation and Endocrinology. His Actin research is multidisciplinary, incorporating elements of Troponin I, Mutation, Troponin, Myopathy and Muscle contraction. As a member of one scientific family, Steven B. Marston mostly works in the field of Troponin, focusing on Troponin T and, on occasion, Actin cytoskeleton.
His Tropomyosin study is related to the wider topic of Biochemistry. Specifically, his work in Internal medicine is concerned with the study of Myofibril. His work in Endocrinology tackles topics such as Hypertrophic cardiomyopathy which are related to areas like Energy metabolism, Mutation, Papillary muscle and Human heart.
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The molecular anatomy of caldesmon.
S B Marston;C S Redwood.
Biochemical Journal (1991)
Troponin phosphorylation and regulatory function in human heart muscle: Dephosphorylation of Ser23/24 on troponin I could account for the contractile defect in end-stage heart failure
Andrew E. Messer;Adam M. Jacques;Steven B. Marston.
Journal of Molecular and Cellular Cardiology (2007)
Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype.
Mahmooda Mirza;Steven Marston;Ruth Willott;Christopher Ashley.
Journal of Biological Chemistry (2005)
Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy.
Paul Robinson;Mahmooda Mirza;Adam Knott;Hassan Abdulrazzak.
Journal of Biological Chemistry (2002)
In Vitro Motility Analysis of Actin-Tropomyosin Regulation by Troponin and Calcium THE THIN FILAMENT IS SWITCHED AS A SINGLE COOPERATIVE UNIT
Iain D. C. Fraser;Steven B. Marston.
Journal of Biological Chemistry (1995)
Alpha-cardiac actin mutations produce atrial septal defects
Hans Matsson;Jacqueline Eason;Carol S. Bookwalter;Joakim Klar.
Human Molecular Genetics (2008)
How Do Mutations in Contractile Proteins Cause the Primary Familial Cardiomyopathies
Steven B. Marston.
Journal of Cardiovascular Translational Research (2011)
Purification and properties of Ca2+-regulated thin filaments and F-actin from sheep aorta smooth muscle
Steven B. Marston;Christopher W. J. Smith.
Journal of Muscle Research and Cell Motility (1984)
Some Properties of Human Small Heat Shock Protein Hsp20 (HspB6)
Olesya V. Bukach;Alim S. Seit-Nebi;Steven B. Marston;Nikolai B. Gusev.
FEBS Journal (2004)
Calcium ion-regulated thin filaments from vascular smooth muscle
S B Marston;R M Trevett;M Walters.
Biochemical Journal (1980)
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