Microbiology, Vancomycin, Staphylococcus aureus, Staphylococcal infections and Methicillin-resistant Staphylococcus aureus are his primary areas of study. His research investigates the connection between Microbiology and topics such as Virulence that intersect with issues in Clade and Clone. His Vancomycin study combines topics from a wide range of disciplines, such as Bacteremia, Internal medicine and Molecular epidemiology.
The Micrococcaceae research Benjamin P. Howden does as part of his general Staphylococcus aureus study is frequently linked to other disciplines of science, such as Cell wall thickening, therefore creating a link between diverse domains of science. His biological study spans a wide range of topics, including Rifampicin, Mutation, Immunology, Fusidic acid and rpoB. While the research belongs to areas of Methicillin-resistant Staphylococcus aureus, Benjamin P. Howden spends his time largely on the problem of Epidemiology, intersecting his research to questions surrounding Pneumonia and Standard treatment.
His main research concerns Microbiology, Staphylococcus aureus, Antibiotic resistance, Drug resistance and Internal medicine. His Microbiology study integrates concerns from other disciplines, such as Vancomycin, Methicillin-resistant Staphylococcus aureus and Virulence. Vancomycin connects with themes related to Virology in his study.
He works mostly in the field of Staphylococcus aureus, limiting it down to topics relating to Antibiotics and, in certain cases, Intensive care medicine. His Antibiotic resistance research integrates issues from Human pathogen, Multiple drug resistance and rpoB. His Internal medicine research includes themes of Bacteremia, Flucloxacillin and Surgery.
His primary scientific interests are in Internal medicine, Severe acute respiratory syndrome coronavirus 2, Microbiology, Antibiotic resistance and Public health. His Internal medicine study combines topics in areas such as Clinical microbiology and Typing. His research integrates issues of Chronic infection, Outbreak and Staphylococcus aureus in his study of Microbiology.
His primary area of study in Staphylococcus aureus is in the field of Staphylococcal infections. His work is dedicated to discovering how Antibiotic resistance, Multiple drug resistance are connected with Shigella sonnei, Fusidic acid, Skin infection, Mupirocin and Tetracycline and other disciplines. His Public health study also includes
Benjamin P. Howden focuses on Severe acute respiratory syndrome coronavirus 2, Internal medicine, Pandemic, Point-of-care testing and Serology. As part of his studies on Internal medicine, he often connects relevant areas like Flucloxacillin. His Pandemic investigation overlaps with other disciplines such as Environmental health, Genomics, Public health and Virology.
His work on Transmission as part of general Virology study is frequently linked to Candida auris, therefore connecting diverse disciplines of science. His Randomized controlled trial research is multidisciplinary, relying on both Bacteremia, Pharmacotherapy, Methicillin-resistant Staphylococcus aureus and Combination therapy. The concepts of his Combination therapy study are interwoven with issues in Vancomycin and Hemodialysis.
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Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications
Benjamin Peter Howden;John Keith Davies;Paul Donald Russell Johnson;Paul Donald Russell Johnson;Timothy Paul Stinear;Timothy Paul Stinear.
Clinical Microbiology Reviews (2010)
Treatment Outcomes for Serious Infections Caused by Methicillin-Resistant Staphylococcus aureus with Reduced Vancomycin Susceptibility
Benjamin P. Howden;Peter B. Ward;Patrick G. P. Charles;Tony M. Korman.
Clinical Infectious Diseases (2004)
Clinical Features Associated with Bacteremia Due to Heterogeneous Vancomycin-Intermediate Staphylococcus aureus
Patrick G. P. Charles;Peter B. Ward;Paul D. R. Johnson;Benjamin P. Howden.
Clinical Infectious Diseases (2004)
Evolution of multidrug resistance during Staphylococcus aureus infection involves mutation of the essential two component regulator WalKR.
Benjamin P Howden;Christopher R E McEvoy;David L Allen;Kyra Y L Chua;Kyra Y L Chua.
PLOS Pathogens (2011)
Antibiotic Choice May Not Explain Poorer Outcomes in Patients With Staphylococcus aureus Bacteremia and High Vancomycin Minimum Inhibitory Concentrations
Natasha E. Holmes;John D. Turnidge;John D. Turnidge;Wendy J. Munckhof;Wendy J. Munckhof;James O. Robinson.
The Journal of Infectious Diseases (2011)
Whole genome sequencing in clinical and public health microbiology
Jason C Kwong;N McCallum;N McCallum;Vitali Sintchenko;Vitali Sintchenko;Benjamin P Howden;Benjamin P Howden.
Isolates with low-level vancomycin resistance associated with persistent methicillin-resistant Staphylococcus aureus bacteremia
Benjamin Peter Howden;Paul D R Johnson;Peter Ward;Timothy Paul Stinear.
Antimicrobial Agents and Chemotherapy (2006)
Staphylococcus aureus bacteraemia: a major cause of mortality in Australia and New Zealand
John D Turnidge;Despina Kotsanas;Wendy Munckhof;Sally Roberts.
The Medical Journal of Australia (2009)
Two novel point mutations in clinical Staphylococcus aureus reduce linezolid susceptibility and switch on the stringent response to promote persistent infection.
Wei Gao;Kyra Chua;Kyra Chua;John Keith Davies;Hayley J Newton.
PLOS Pathogens (2010)
Not Community-Associated Methicillin-Resistant Staphylococcus aureus (CA-MRSA)! A Clinician's Guide to Community MRSA - Its Evolving Antimicrobial Resistance and Implications for Therapy
Kyra Chua;Frederic Laurent;Geoffrey Coombs;M Lindsay Lindsay Grayson.
Clinical Infectious Diseases (2011)
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