His primary areas of investigation include Microbiology, Mycobacterium ulcerans, Genetics, Buruli ulcer and Mycolactone. His study in Microbiology is interdisciplinary in nature, drawing from both Plasmid, Vancomycin, Staphylococcus aureus and Virulence. In Staphylococcus aureus, Timothy P. Stinear works on issues like Antibiotic resistance, which are connected to Drug resistance.
His Mycobacterium ulcerans research is multidisciplinary, relying on both Inverted repeat, Outbreak and Polymerase chain reaction. He works mostly in the field of Genetics, limiting it down to topics relating to Mycobacterium tuberculosis and, in certain cases, Bacilli, Phylogenetics and Tubercle, as a part of the same area of interest. His work carried out in the field of Mycolactone brings together such families of science as Transmission, Pathogen, Signal transduction and Mycobacterium marinum.
His primary scientific interests are in Microbiology, Mycobacterium ulcerans, Genetics, Buruli ulcer and Staphylococcus aureus. Timothy P. Stinear interconnects Vancomycin, Virology and Virulence in the investigation of issues within Microbiology. His studies in Vancomycin integrate themes in fields like Antibacterial agent, Enterococcus faecium and Molecular epidemiology.
His Mycobacterium ulcerans research incorporates themes from Mycobacterium marinum, Immunology, Polymerase chain reaction and Insertion sequence. Infection control is closely connected to Transmission in his research, which is encompassed under the umbrella topic of Buruli ulcer. The concepts of his Staphylococcus aureus study are interwoven with issues in Mutation and Antibiotics.
Timothy P. Stinear mainly focuses on Gene, Genetics, Staphylococcus aureus, Microbiology and Antibiotic resistance. His work in the fields of Staphylococcal infections overlaps with other areas such as Respiratory tract. His research investigates the connection with Staphylococcal infections and areas like Phagocytosis which intersect with concerns in Immune system.
As part of the same scientific family, Timothy P. Stinear usually focuses on Microbiology, concentrating on Mycobacterium tuberculosis and intersecting with Nitroimidazole. His studies deal with areas such as Microbiome, Enterococcus faecium, Drug resistance and Mobile genetic elements as well as Antibiotic resistance. His Gene expression research integrates issues from Skin infection and Virulence.
Timothy P. Stinear mainly investigates Genomics, Genetics, Antibiotic resistance, Gene and Environmental health. His research integrates issues of Plasmid maintenance, Enterococcus faecium and Drug resistance in his study of Antibiotic resistance. The study incorporates disciplines such as Staphylococcal infections and Staphylococcus aureus in addition to Gene.
His study looks at the intersection of Staphylococcus aureus and topics like Virulence with Microbiology. His work deals with themes such as Tuberculosis and Mycobacterium tuberculosis, which intersect with Microbiology. His biological study spans a wide range of topics, including Disease reservoir, Population genomics and Disease, Tropical disease.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications
Benjamin Peter Howden;John Keith Davies;Paul Donald Russell Johnson;Paul Donald Russell Johnson;Timothy Paul Stinear;Timothy Paul Stinear.
Clinical Microbiology Reviews (2010)
Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis
Timothy P. Stinear;Torsten Seemann;Paul F. Harrison;Grant A. Jenkin.
Genome Research (2008)
Giant plasmid-encoded polyketide synthases produce the macrolide toxin of Mycobacterium ulcerans.
Timothy Paul Stinear;Armand Mve-Obiang;Pamela L C Small;Wafa Frigui.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Reductive evolution and niche adaptation inferred from the genome of Mycobacterium ulcerans, the causative agent of Buruli ulcer
Timothy Paul Stinear;Torsten Seemann;Sacha James Pidot;Wafa Frigui.
Genome Research (2007)
Evolution of multidrug resistance during Staphylococcus aureus infection involves mutation of the essential two component regulator WalKR.
Benjamin P Howden;Christopher R E McEvoy;David L Allen;Kyra Y L Chua;Kyra Y L Chua.
PLOS Pathogens (2011)
Genomic analysis of smooth tubercle bacilli provides insights into ancestry and pathoadaptation of Mycobacterium tuberculosis
Philip Supply;Michael Marceau;Sophie Mangenot;David Roche;David Roche.
Nature Genetics (2013)
Buruli Ulcer (M. ulcerans Infection): New Insights, New Hope for Disease Control
Paul D R Johnson;Timothy P Stinear;Pamela L C Small;Gerd Pluschke.
PLOS Medicine (2005)
Mycobacterium ulcerans in mosquitoes captured during outbreak of Buruli ulcer, southeastern Australia.
Paul D R Johnson;Joseph Azuolas;Caroline J Lavender;Elwyn Wishart.
Emerging Infectious Diseases (2007)
Isolates with low-level vancomycin resistance associated with persistent methicillin-resistant Staphylococcus aureus bacteremia
Benjamin Peter Howden;Paul D R Johnson;Peter Ward;Timothy Paul Stinear.
Antimicrobial Agents and Chemotherapy (2006)
Buruli ulcer: reductive evolution enhances pathogenicity of Mycobacterium ulcerans.
Caroline Demangel;Timothy Paul Stinear;Stewart T Cole.
Nature Reviews Microbiology (2009)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Melbourne
University of Melbourne
Austin Health
Monash University
Fiona Stanley Hospital
Swiss Tropical and Public Health Institute
Institute of Tropical Medicine Antwerp
Monash University
Université Paris Cité
University of Cambridge
Tel Aviv University
University of Parma
Qatar Environment and Energy Research Institute
University of Pennsylvania
Chinese Academy of Sciences
United States Air Force Research Laboratory
Utsunomiya University
Rutgers, The State University of New Jersey
Spanish National Research Council
Public Health Agency of Canada
Institut Pasteur
GEOMAR Helmholtz Centre for Ocean Research Kiel
Commonwealth Scientific and Industrial Research Organisation
Harvard University
United States Department of the Army
The University of Texas MD Anderson Cancer Center