D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 65 Citations 13,845 225 World Ranking 1543 National Ranking 49

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Bacteria
  • DNA

His primary areas of investigation include Microbiology, Mycobacterium ulcerans, Genetics, Buruli ulcer and Mycolactone. His study in Microbiology is interdisciplinary in nature, drawing from both Plasmid, Vancomycin, Staphylococcus aureus and Virulence. In Staphylococcus aureus, Timothy P. Stinear works on issues like Antibiotic resistance, which are connected to Drug resistance.

His Mycobacterium ulcerans research is multidisciplinary, relying on both Inverted repeat, Outbreak and Polymerase chain reaction. He works mostly in the field of Genetics, limiting it down to topics relating to Mycobacterium tuberculosis and, in certain cases, Bacilli, Phylogenetics and Tubercle, as a part of the same area of interest. His work carried out in the field of Mycolactone brings together such families of science as Transmission, Pathogen, Signal transduction and Mycobacterium marinum.

His most cited work include:

  • Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications (656 citations)
  • Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications (656 citations)
  • Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis (414 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Microbiology, Mycobacterium ulcerans, Genetics, Buruli ulcer and Staphylococcus aureus. Timothy P. Stinear interconnects Vancomycin, Virology and Virulence in the investigation of issues within Microbiology. His studies in Vancomycin integrate themes in fields like Antibacterial agent, Enterococcus faecium and Molecular epidemiology.

His Mycobacterium ulcerans research incorporates themes from Mycobacterium marinum, Immunology, Polymerase chain reaction and Insertion sequence. Infection control is closely connected to Transmission in his research, which is encompassed under the umbrella topic of Buruli ulcer. The concepts of his Staphylococcus aureus study are interwoven with issues in Mutation and Antibiotics.

He most often published in these fields:

  • Microbiology (65.03%)
  • Mycobacterium ulcerans (31.82%)
  • Genetics (31.82%)

What were the highlights of his more recent work (between 2018-2021)?

  • Gene (15.73%)
  • Genetics (31.82%)
  • Staphylococcus aureus (24.83%)

In recent papers he was focusing on the following fields of study:

Timothy P. Stinear mainly focuses on Gene, Genetics, Staphylococcus aureus, Microbiology and Antibiotic resistance. His work in the fields of Staphylococcal infections overlaps with other areas such as Respiratory tract. His research investigates the connection with Staphylococcal infections and areas like Phagocytosis which intersect with concerns in Immune system.

As part of the same scientific family, Timothy P. Stinear usually focuses on Microbiology, concentrating on Mycobacterium tuberculosis and intersecting with Nitroimidazole. His studies deal with areas such as Microbiome, Enterococcus faecium, Drug resistance and Mobile genetic elements as well as Antibiotic resistance. His Gene expression research integrates issues from Skin infection and Virulence.

Between 2018 and 2021, his most popular works were:

  • Tracking the COVID-19 pandemic in Australia using genomics. (57 citations)
  • Unstable chromosome rearrangements in Staphylococcus aureus cause phenotype switching associated with persistent infections (24 citations)
  • Genomics for Molecular Epidemiology and Detecting Transmission of Carbapenemase-Producing Enterobacterales in Victoria, Australia, 2012 to 2016 (20 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Bacteria
  • DNA

Timothy P. Stinear mainly investigates Genomics, Genetics, Antibiotic resistance, Gene and Environmental health. His research integrates issues of Plasmid maintenance, Enterococcus faecium and Drug resistance in his study of Antibiotic resistance. The study incorporates disciplines such as Staphylococcal infections and Staphylococcus aureus in addition to Gene.

His study looks at the intersection of Staphylococcus aureus and topics like Virulence with Microbiology. His work deals with themes such as Tuberculosis and Mycobacterium tuberculosis, which intersect with Microbiology. His biological study spans a wide range of topics, including Disease reservoir, Population genomics and Disease, Tropical disease.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications

Benjamin Peter Howden;John Keith Davies;Paul Donald Russell Johnson;Paul Donald Russell Johnson;Timothy Paul Stinear;Timothy Paul Stinear.
Clinical Microbiology Reviews (2010)

1098 Citations

Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis

Timothy P. Stinear;Torsten Seemann;Paul F. Harrison;Grant A. Jenkin.
Genome Research (2008)

576 Citations

Giant plasmid-encoded polyketide synthases produce the macrolide toxin of Mycobacterium ulcerans.

Timothy Paul Stinear;Armand Mve-Obiang;Pamela L C Small;Wafa Frigui.
Proceedings of the National Academy of Sciences of the United States of America (2004)

425 Citations

Reductive evolution and niche adaptation inferred from the genome of Mycobacterium ulcerans, the causative agent of Buruli ulcer

Timothy Paul Stinear;Torsten Seemann;Sacha James Pidot;Wafa Frigui.
Genome Research (2007)

417 Citations

Evolution of multidrug resistance during Staphylococcus aureus infection involves mutation of the essential two component regulator WalKR.

Benjamin P Howden;Christopher R E McEvoy;David L Allen;Kyra Y L Chua;Kyra Y L Chua.
PLOS Pathogens (2011)

306 Citations

Genomic analysis of smooth tubercle bacilli provides insights into ancestry and pathoadaptation of Mycobacterium tuberculosis

Philip Supply;Michael Marceau;Sophie Mangenot;David Roche;David Roche.
Nature Genetics (2013)

291 Citations

Buruli Ulcer (M. ulcerans Infection): New Insights, New Hope for Disease Control

Paul D R Johnson;Timothy P Stinear;Pamela L C Small;Gerd Pluschke.
PLOS Medicine (2005)

279 Citations

Mycobacterium ulcerans in mosquitoes captured during outbreak of Buruli ulcer, southeastern Australia.

Paul D R Johnson;Joseph Azuolas;Caroline J Lavender;Elwyn Wishart.
Emerging Infectious Diseases (2007)

269 Citations

Isolates with low-level vancomycin resistance associated with persistent methicillin-resistant Staphylococcus aureus bacteremia

Benjamin Peter Howden;Paul D R Johnson;Peter Ward;Timothy Paul Stinear.
Antimicrobial Agents and Chemotherapy (2006)

262 Citations

Buruli ulcer: reductive evolution enhances pathogenicity of Mycobacterium ulcerans.

Caroline Demangel;Timothy Paul Stinear;Stewart T Cole.
Nature Reviews Microbiology (2009)

226 Citations

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