John K. Davies mainly focuses on Microbiology, Genetics, Staphylococcus aureus, Genome and Vancomycin. John K. Davies is interested in Pathogen, which is a branch of Microbiology. His Gene and Mycobacterium marinum study in the realm of Genetics interacts with subjects such as Insertion sequence.
Gene and Leptospira interrogans are two areas of study in which he engages in interdisciplinary research. His works in Comparative genomics and Whole genome sequencing are all subjects of inquiry into Genome. As a part of the same scientific family, John K. Davies mostly works in the field of Vancomycin, focusing on Antibiotic resistance and, on occasion, Drug resistance.
John K. Davies mostly deals with Microbiology, Genetics, Gene, Neisseria gonorrhoeae and Molecular biology. John K. Davies combines subjects such as Staphylococcus aureus, Bacteria, Mycobacterium ulcerans and Virulence with his study of Microbiology. His biological study deals with issues like Buruli ulcer, which deal with fields such as Mycobacterium.
His studies in Neisseria gonorrhoeae integrate themes in fields like Bacterial outer membrane, Genetic variation, DNA and Pilin. His Molecular biology research is multidisciplinary, relying on both Gene expression and Mutant. His work on Comparative genomics as part of his general Genome study is frequently connected to Leptospira interrogans and Prophage, thereby bridging the divide between different branches of science.
Microbiology, Genetics, Gene, Virulence and Staphylococcus aureus are his primary areas of study. His research integrates issues of Mutant, Methicillin-resistant Staphylococcus aureus, Mycobacterium marinum and Whole genome sequencing in his study of Microbiology. His work on Genome, Neisseria, Mycobacterium and Inverted repeat as part of general Genetics study is frequently linked to Transposase, bridging the gap between disciplines.
His Gene research includes themes of Molecular biology and Neisseria gonorrhoeae. His research in Virulence focuses on subjects like Corynebacterium pseudotuberculosis, which are connected to Heat shock, CLPB, Caseous lymphadenitis and Downregulation and upregulation. His Staphylococcus aureus research is multidisciplinary, incorporating perspectives in Antibiotics and Clone.
His main research concerns Microbiology, Genetics, Genome, Gene and Staphylococcus aureus. His Microbiology study combines topics in areas such as Innate immune system, Methicillin-resistant Staphylococcus aureus and Virulence. His research related to Comparative genomics and Whole genome sequencing might be considered part of Genome.
His Comparative genomics study incorporates themes from Genome evolution, Gene density and Gene rearrangement. His research investigates the connection with Whole genome sequencing and areas like Mycobacterium marinum which intersect with concerns in Buruli ulcer, Mycolactone, Pseudogene and Mycobacterium ulcerans. His studies examine the connections between Staphylococcal infections and genetics, as well as such issues in Antibiotics, with regards to Pathogen.
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Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications
Benjamin Peter Howden;John Keith Davies;Paul Donald Russell Johnson;Paul Donald Russell Johnson;Timothy Paul Stinear;Timothy Paul Stinear.
Clinical Microbiology Reviews (2010)
Mechanisms involved in the resistance of Enterococcus faecalis to calcium hydroxide
Matthew Evans;John K Davies;Goran Sundqvist;David Figdor;David Figdor;David Figdor.
International Endodontic Journal (2002)
Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis
Timothy P. Stinear;Torsten Seemann;Paul F. Harrison;Grant A. Jenkin.
Genome Research (2008)
Giant plasmid-encoded polyketide synthases produce the macrolide toxin of Mycobacterium ulcerans.
Timothy Paul Stinear;Armand Mve-Obiang;Pamela L C Small;Wafa Frigui.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Reductive evolution and niche adaptation inferred from the genome of Mycobacterium ulcerans, the causative agent of Buruli ulcer
Timothy Paul Stinear;Torsten Seemann;Sacha James Pidot;Wafa Frigui.
Genome Research (2007)
Genome reduction in Leptospira borgpetersenii reflects limited transmission potential
Dieter Mark Bulach;Richard L Zuerner;Peter Wilson;Torsten Seemann.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Genome Sequence of the Saprophyte Leptospira biflexa Provides Insights into the Evolution of Leptospira and the Pathogenesis of Leptospirosis
Mathieu Picardeau;Dieter Mark Bulach;Dieter Mark Bulach;Christiane Bouchier;Richard L Zuerner.
PLOS ONE (2008)
Bacterial membrane vesicles deliver peptidoglycan to NOD1 in epithelial cells
Maria Kaparakis;Lynne Turnbull;Leticia Carneiro;Stephen Firth.
Cellular Microbiology (2010)
Evolution of multidrug resistance during Staphylococcus aureus infection involves mutation of the essential two component regulator WalKR.
Benjamin P Howden;Christopher R E McEvoy;David L Allen;Kyra Y L Chua;Kyra Y L Chua.
PLOS Pathogens (2011)
Isolates with low-level vancomycin resistance associated with persistent methicillin-resistant Staphylococcus aureus bacteremia
Benjamin Peter Howden;Paul D R Johnson;Peter Ward;Timothy Paul Stinear.
Antimicrobial Agents and Chemotherapy (2006)
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