D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 46 Citations 9,375 104 World Ranking 15091 National Ranking 426

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Bacteria
  • DNA

John K. Davies mainly focuses on Microbiology, Genetics, Staphylococcus aureus, Genome and Vancomycin. John K. Davies is interested in Pathogen, which is a branch of Microbiology. His Gene and Mycobacterium marinum study in the realm of Genetics interacts with subjects such as Insertion sequence.

Gene and Leptospira interrogans are two areas of study in which he engages in interdisciplinary research. His works in Comparative genomics and Whole genome sequencing are all subjects of inquiry into Genome. As a part of the same scientific family, John K. Davies mostly works in the field of Vancomycin, focusing on Antibiotic resistance and, on occasion, Drug resistance.

His most cited work include:

  • Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications (656 citations)
  • Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis (414 citations)
  • Mechanisms involved in the resistance of Enterococcus faecalis to calcium hydroxide (384 citations)

What are the main themes of his work throughout his whole career to date?

John K. Davies mostly deals with Microbiology, Genetics, Gene, Neisseria gonorrhoeae and Molecular biology. John K. Davies combines subjects such as Staphylococcus aureus, Bacteria, Mycobacterium ulcerans and Virulence with his study of Microbiology. His biological study deals with issues like Buruli ulcer, which deal with fields such as Mycobacterium.

His studies in Neisseria gonorrhoeae integrate themes in fields like Bacterial outer membrane, Genetic variation, DNA and Pilin. His Molecular biology research is multidisciplinary, relying on both Gene expression and Mutant. His work on Comparative genomics as part of his general Genome study is frequently connected to Leptospira interrogans and Prophage, thereby bridging the divide between different branches of science.

He most often published in these fields:

  • Microbiology (53.21%)
  • Genetics (42.20%)
  • Gene (37.61%)

What were the highlights of his more recent work (between 2006-2020)?

  • Microbiology (53.21%)
  • Genetics (42.20%)
  • Gene (37.61%)

In recent papers he was focusing on the following fields of study:

Microbiology, Genetics, Gene, Virulence and Staphylococcus aureus are his primary areas of study. His research integrates issues of Mutant, Methicillin-resistant Staphylococcus aureus, Mycobacterium marinum and Whole genome sequencing in his study of Microbiology. His work on Genome, Neisseria, Mycobacterium and Inverted repeat as part of general Genetics study is frequently linked to Transposase, bridging the gap between disciplines.

His Gene research includes themes of Molecular biology and Neisseria gonorrhoeae. His research in Virulence focuses on subjects like Corynebacterium pseudotuberculosis, which are connected to Heat shock, CLPB, Caseous lymphadenitis and Downregulation and upregulation. His Staphylococcus aureus research is multidisciplinary, incorporating perspectives in Antibiotics and Clone.

Between 2006 and 2020, his most popular works were:

  • Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications (656 citations)
  • Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis (414 citations)
  • Reductive evolution and niche adaptation inferred from the genome of Mycobacterium ulcerans, the causative agent of Buruli ulcer (311 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Bacteria
  • DNA

His main research concerns Microbiology, Genetics, Genome, Gene and Staphylococcus aureus. His Microbiology study combines topics in areas such as Innate immune system, Methicillin-resistant Staphylococcus aureus and Virulence. His research related to Comparative genomics and Whole genome sequencing might be considered part of Genome.

His Comparative genomics study incorporates themes from Genome evolution, Gene density and Gene rearrangement. His research investigates the connection with Whole genome sequencing and areas like Mycobacterium marinum which intersect with concerns in Buruli ulcer, Mycolactone, Pseudogene and Mycobacterium ulcerans. His studies examine the connections between Staphylococcal infections and genetics, as well as such issues in Antibiotics, with regards to Pathogen.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications

Benjamin Peter Howden;John Keith Davies;Paul Donald Russell Johnson;Paul Donald Russell Johnson;Timothy Paul Stinear;Timothy Paul Stinear.
Clinical Microbiology Reviews (2010)

1098 Citations

Mechanisms involved in the resistance of Enterococcus faecalis to calcium hydroxide

Matthew Evans;John K Davies;Goran Sundqvist;David Figdor;David Figdor;David Figdor.
International Endodontic Journal (2002)

823 Citations

Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis

Timothy P. Stinear;Torsten Seemann;Paul F. Harrison;Grant A. Jenkin.
Genome Research (2008)

576 Citations

Giant plasmid-encoded polyketide synthases produce the macrolide toxin of Mycobacterium ulcerans.

Timothy Paul Stinear;Armand Mve-Obiang;Pamela L C Small;Wafa Frigui.
Proceedings of the National Academy of Sciences of the United States of America (2004)

425 Citations

Reductive evolution and niche adaptation inferred from the genome of Mycobacterium ulcerans, the causative agent of Buruli ulcer

Timothy Paul Stinear;Torsten Seemann;Sacha James Pidot;Wafa Frigui.
Genome Research (2007)

417 Citations

Genome reduction in Leptospira borgpetersenii reflects limited transmission potential

Dieter Mark Bulach;Richard L Zuerner;Peter Wilson;Torsten Seemann.
Proceedings of the National Academy of Sciences of the United States of America (2006)

408 Citations

Genome Sequence of the Saprophyte Leptospira biflexa Provides Insights into the Evolution of Leptospira and the Pathogenesis of Leptospirosis

Mathieu Picardeau;Dieter Mark Bulach;Dieter Mark Bulach;Christiane Bouchier;Richard L Zuerner.
PLOS ONE (2008)

389 Citations

Bacterial membrane vesicles deliver peptidoglycan to NOD1 in epithelial cells

Maria Kaparakis;Lynne Turnbull;Leticia Carneiro;Stephen Firth.
Cellular Microbiology (2010)

381 Citations

Evolution of multidrug resistance during Staphylococcus aureus infection involves mutation of the essential two component regulator WalKR.

Benjamin P Howden;Christopher R E McEvoy;David L Allen;Kyra Y L Chua;Kyra Y L Chua.
PLOS Pathogens (2011)

306 Citations

Isolates with low-level vancomycin resistance associated with persistent methicillin-resistant Staphylococcus aureus bacteremia

Benjamin Peter Howden;Paul D R Johnson;Peter Ward;Timothy Paul Stinear.
Antimicrobial Agents and Chemotherapy (2006)

262 Citations

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