Her primary areas of investigation include Immunology, Genetics, Human leukocyte antigen, Genome-wide association study and Single-nucleotide polymorphism. Her biological study spans a wide range of topics, including PTPN22 and Internal medicine. Her Human leukocyte antigen research includes themes of Disease, Genetic predisposition, HLA Complex, Type 1 diabetes and Major histocompatibility complex.
Her research integrates issues of Multiple sclerosis and Genotyping in her study of Genome-wide association study. Her Single-nucleotide polymorphism research is multidisciplinary, relying on both IRF5, Locus and Candidate gene. The various areas that Benedicte A. Lie examines in her Allele study include Odds ratio and Genotype.
Benedicte A. Lie mainly focuses on Genetics, Immunology, Human leukocyte antigen, Allele and Single-nucleotide polymorphism. Haplotype, Genome-wide association study, Linkage disequilibrium, Gene and Major histocompatibility complex are among the areas of Genetics where the researcher is concentrating her efforts. Her Genome-wide association study research incorporates elements of Genetic Pleiotropy, CD247 and Bioinformatics.
Her research in Immunology intersects with topics in Odds ratio, Internal medicine, Genotyping and Primary sclerosing cholangitis. Her biological study deals with issues like HLA Complex, which deal with fields such as Genetic predisposition. Her Allele research is multidisciplinary, incorporating perspectives in Genotype and Locus.
The scientist’s investigation covers issues in Internal medicine, Human leukocyte antigen, microRNA, Chronic fatigue syndrome and Immunology. Her studies deal with areas such as Endocrinology, T lymphocyte and Allele as well as Internal medicine. The Human leukocyte antigen study combines topics in areas such as Haplotype, Genotyping, Allele frequency and Asymptomatic.
As a part of the same scientific study, she usually deals with the Allele frequency, concentrating on Mortality rate and frequently concerns with Genome-wide association study. Benedicte A. Lie works mostly in the field of Genome-wide association study, limiting it down to concerns involving Arthritis and, occasionally, Genetics and Candidate gene. Her Immunology research is multidisciplinary, incorporating perspectives in Enteropathy, Pregnancy and Odds ratio.
Her scientific interests lie mostly in Chronic fatigue syndrome, Severity of illness, Genotyping, Human leukocyte antigen and Linkage disequilibrium. To a larger extent, Benedicte A. Lie studies Internal medicine with the aim of understanding Chronic fatigue syndrome. Her work carried out in the field of Severity of illness brings together such families of science as Autoimmune disease, Case-control study, Immunology and Etiology.
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Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis
Ashley H. Beecham;Nikolaos A. Patsopoulos;Nikolaos A. Patsopoulos;Dionysia K. Xifara;Mary F. Davis.
Nature Genetics (2013)
Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci.
A Cortes;J Hadler;J P Pointon.
Nature Genetics (2013)
Fine mapping of type 1 diabetes susceptibility loci and evidence for colocalization of causal variants with lymphoid gene enhancers.
Suna Onengut-Gumuscu;Wei-Min Chen;Oliver Burren;Nick J Cooper.
Nature Genetics (2015)
Genome-wide association study of systemic sclerosis identifies CD247 as a new susceptibility locus
Timothy R.D.J. Radstake;Olga Y Gorlova;Blanca Rueda;Jose Ezequiel Martin.
Nature Genetics (2010)
Genome-Wide Association Analysis in Primary Sclerosing Cholangitis
Tom H. Karlsen;Andre Franke;Espen Melum;Arthur Kaser.
Celiac disease genetics: current concepts and practical applications.
Ludvig M. Sollid;Benedicte A. Lie.
Clinical Gastroenterology and Hepatology (2005)
HLA associated genetic predisposition to autoimmune diseases: Genes involved and possible mechanisms.
Erik Thorsby;Benedicte A. Lie.
Transplant Immunology (2005)
Identification of novel genetic markers associated with clinical phenotypes of systemic sclerosis through a genome-wide association strategy
Olga Gorlova;Jose Ezequiel Martin;Blanca Rueda;Bobby P C Koeleman.
PLOS Genetics (2011)
Major histocompatibility complex associations of ankylosing spondylitis are complex and involve further epistasis with ERAP1.
Adrian Cortes;Sara L. Pulit;Paul J. Leo;Jenny J. Pointon.
Nature Communications (2015)
Meta-analysis of shared genetic architecture across ten pediatric autoimmune diseases
Yun R Li;Jin Li;Sihai D Zhao;Sihai D Zhao;Jonathan P Bradfield.
Nature Medicine (2015)
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