Ben J. Appelmelk focuses on Microbiology, Helicobacter pylori, Antigen, Immunology and Glycan. Ben J. Appelmelk interconnects Secretion, Mutant, Bacteria, Mycobacterium tuberculosis and Virulence in the investigation of issues within Microbiology. Lipopolysaccharide is closely connected to Embryo in his research, which is encompassed under the umbrella topic of Mutant.
His Helicobacter pylori study integrates concerns from other disciplines, such as Epitope, Molecular mimicry, Antibody and Monoclonal antibody. Ben J. Appelmelk focuses mostly in the field of Immunology, narrowing it down to matters related to Gastritis and, in some cases, Autoantibody, Gastric mucosa and Parietal cell. His Glycan research incorporates themes from Glycolipid, Lectin and Glycoconjugate.
His primary scientific interests are in Microbiology, Lipopolysaccharide, Epitope, Antigen and Immunology. His Microbiology research incorporates elements of Mutant, Enterobacteriaceae, Escherichia coli, Mycobacterium tuberculosis and Virulence. His Mycobacterium tuberculosis research is multidisciplinary, incorporating elements of DC-SIGN and Mycobacterium.
Ben J. Appelmelk combines subjects such as Virology, Neisseria, Antibody, Gram-negative bacteria and In vivo with his study of Lipopolysaccharide. His work deals with themes such as Lipid A, Biochemistry, Binding site, Molecular biology and Monoclonal antibody, which intersect with Epitope. In his work, Molecular mimicry, Gastric mucosa and Phase variation is strongly intertwined with Helicobacter pylori, which is a subfield of Immunology.
The scientist’s investigation covers issues in Microbiology, Mycobacterium tuberculosis, Lipoarabinomannan, Mutant and Biochemistry. His research investigates the connection between Microbiology and topics such as C-type lectin that intersect with issues in Innate immune system. The study incorporates disciplines such as DC-SIGN, Mannose and Mycobacterium smegmatis in addition to Lipoarabinomannan.
His studies in Mutant integrate themes in fields like Antibody, Mycobacterium marinum and Biosynthesis. His Antibody study incorporates themes from Secretion and Protein family. His Biochemistry course of study focuses on Mycobacterium and Bacterial capsule.
Ben J. Appelmelk mainly investigates Microbiology, Glycolipid, Mycobacterium tuberculosis, Biochemistry and Lipoarabinomannan. His biological study spans a wide range of topics, including Secretion and Mycobacterium marinum. His Glycolipid study combines topics from a wide range of disciplines, such as Protein purification, Bacterial capsule, Secretory protein, DC-SIGN and Cell envelope.
Ben J. Appelmelk has researched DC-SIGN in several fields, including Ligand binding assay and Mannose. His Mycobacterium tuberculosis research includes themes of Mutant, Protein family, Bacteria, Antibody and Virulence. His research integrates issues of Lipomannan and Tuberculosis, Mycobacterium in his study of Biochemistry.
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Mycobacteria Target DC-SIGN to Suppress Dendritic Cell Function
Teunis B.H. Geijtenbeek;Sandra J. van Vliet;Estella A. Koppel;Marta Sanchez-Hernandez.
Journal of Experimental Medicine (2003)
Type VII secretion — mycobacteria show the way
Abdallah M Abdallah;Nicolaas C Gey van Pittius;Patricia A DiGiuseppe Champion;Jeffery Cox.
Nature Reviews Microbiology (2007)
Potential role of molecular mimicry between Helicobacter pylori lipopolysaccharide and host Lewis blood group antigens in autoimmunity.
B J Appelmelk;I Simoons-Smit;R Negrini;A P Moran.
Infection and Immunity (1996)
Cutting Edge: Carbohydrate Profiling Identifies New Pathogens That Interact with Dendritic Cell-Specific ICAM-3-Grabbing Nonintegrin on Dendritic Cells
Ben J. Appelmelk;Irma van Die;Sandra J. van Vliet;Christina M. J. E. Vandenbroucke-Grauls.
Journal of Immunology (2003)
Lactoferrin is a lipid A-binding protein.
B. J. Appelmelk;Yun-Qing An;M. Geerts;B. G. Thijs.
Infection and Immunity (1994)
The dendritic cell-specific C-type lectin DC-SIGN is a receptor for Schistosoma mansoni egg antigens and recognizes the glycan antigen Lewis x.
Irma van Die;Sandra J van Vliet;A Kwame Nyame;Richard D Cummings.
Helicobacter pylori modulates the T helper cell 1/T helper cell 2 balance through phase-variable interaction between lipopolysaccharide and DC-SIGN.
Mathijs P. Bergman;Anneke Engering;Hermelijn H. Smits;Sandra J. van Vliet.
Journal of Experimental Medicine (2004)
Antigenic mimicry between Helicobacter pylori and gastric mucosa in the pathogenesis of body atrophic gastritis
R Negrini;A Savio;C Poiesi;BJ Appelmelk.
Molecular mimicry of host structures by bacterial lipopolysaccharides and its contribution to disease
Anthony P. Moran;Martina M. Prendergast;Ben J. Appelmelk.
Fems Immunology and Medical Microbiology (1996)
The gastric H+,K+-ATPase is a major autoantigen in chronic Helicobacter pylori gastritis with body mucosa atrophy ☆ ☆☆ ★ ★★
Dirk Claeys;Gerhard Faller;Ben J. Appelmelk;Riccardo Negrini.
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