D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 56 Citations 13,254 117 World Ranking 2541 National Ranking 90

Overview

What is he best known for?

The fields of study he is best known for:

  • Enzyme
  • Gene
  • Immune system

Ben J. Appelmelk focuses on Microbiology, Helicobacter pylori, Antigen, Immunology and Glycan. Ben J. Appelmelk interconnects Secretion, Mutant, Bacteria, Mycobacterium tuberculosis and Virulence in the investigation of issues within Microbiology. Lipopolysaccharide is closely connected to Embryo in his research, which is encompassed under the umbrella topic of Mutant.

His Helicobacter pylori study integrates concerns from other disciplines, such as Epitope, Molecular mimicry, Antibody and Monoclonal antibody. Ben J. Appelmelk focuses mostly in the field of Immunology, narrowing it down to matters related to Gastritis and, in some cases, Autoantibody, Gastric mucosa and Parietal cell. His Glycan research incorporates themes from Glycolipid, Lectin and Glycoconjugate.

His most cited work include:

  • Mycobacteria Target DC-SIGN to Suppress Dendritic Cell Function (916 citations)
  • Type VII secretion — mycobacteria show the way (525 citations)
  • Cutting Edge: Carbohydrate Profiling Identifies New Pathogens That Interact with Dendritic Cell-Specific ICAM-3-Grabbing Nonintegrin on Dendritic Cells (380 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Microbiology, Lipopolysaccharide, Epitope, Antigen and Immunology. His Microbiology research incorporates elements of Mutant, Enterobacteriaceae, Escherichia coli, Mycobacterium tuberculosis and Virulence. His Mycobacterium tuberculosis research is multidisciplinary, incorporating elements of DC-SIGN and Mycobacterium.

Ben J. Appelmelk combines subjects such as Virology, Neisseria, Antibody, Gram-negative bacteria and In vivo with his study of Lipopolysaccharide. His work deals with themes such as Lipid A, Biochemistry, Binding site, Molecular biology and Monoclonal antibody, which intersect with Epitope. In his work, Molecular mimicry, Gastric mucosa and Phase variation is strongly intertwined with Helicobacter pylori, which is a subfield of Immunology.

He most often published in these fields:

  • Microbiology (47.62%)
  • Lipopolysaccharide (26.98%)
  • Epitope (25.40%)

What were the highlights of his more recent work (between 2008-2021)?

  • Microbiology (47.62%)
  • Mycobacterium tuberculosis (12.70%)
  • Lipoarabinomannan (9.52%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Microbiology, Mycobacterium tuberculosis, Lipoarabinomannan, Mutant and Biochemistry. His research investigates the connection between Microbiology and topics such as C-type lectin that intersect with issues in Innate immune system. The study incorporates disciplines such as DC-SIGN, Mannose and Mycobacterium smegmatis in addition to Lipoarabinomannan.

His studies in Mutant integrate themes in fields like Antibody, Mycobacterium marinum and Biosynthesis. His Antibody study incorporates themes from Secretion and Protein family. His Biochemistry course of study focuses on Mycobacterium and Bacterial capsule.

Between 2008 and 2021, his most popular works were:

  • Lipoarabinomannan and related glycoconjugates: structure, biogenesis and role in Mycobacterium tuberculosis physiology and host–pathogen interaction (207 citations)
  • Direct visualization by cryo-EM of the mycobacterial capsular layer: a labile structure containing ESX-1-secreted proteins. (202 citations)
  • PPE and PE_PGRS proteins of Mycobacterium marinum are transported via the type VII secretion system ESX‐5 (202 citations)

In his most recent research, the most cited papers focused on:

  • Enzyme
  • Gene
  • Immune system

Ben J. Appelmelk mainly investigates Microbiology, Glycolipid, Mycobacterium tuberculosis, Biochemistry and Lipoarabinomannan. His biological study spans a wide range of topics, including Secretion and Mycobacterium marinum. His Glycolipid study combines topics from a wide range of disciplines, such as Protein purification, Bacterial capsule, Secretory protein, DC-SIGN and Cell envelope.

Ben J. Appelmelk has researched DC-SIGN in several fields, including Ligand binding assay and Mannose. His Mycobacterium tuberculosis research includes themes of Mutant, Protein family, Bacteria, Antibody and Virulence. His research integrates issues of Lipomannan and Tuberculosis, Mycobacterium in his study of Biochemistry.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mycobacteria Target DC-SIGN to Suppress Dendritic Cell Function

Teunis B.H. Geijtenbeek;Sandra J. van Vliet;Estella A. Koppel;Marta Sanchez-Hernandez.
Journal of Experimental Medicine (2003)

1366 Citations

Type VII secretion — mycobacteria show the way

Abdallah M Abdallah;Nicolaas C Gey van Pittius;Patricia A DiGiuseppe Champion;Jeffery Cox.
Nature Reviews Microbiology (2007)

836 Citations

Potential role of molecular mimicry between Helicobacter pylori lipopolysaccharide and host Lewis blood group antigens in autoimmunity.

B J Appelmelk;I Simoons-Smit;R Negrini;A P Moran.
Infection and Immunity (1996)

545 Citations

Cutting Edge: Carbohydrate Profiling Identifies New Pathogens That Interact with Dendritic Cell-Specific ICAM-3-Grabbing Nonintegrin on Dendritic Cells

Ben J. Appelmelk;Irma van Die;Sandra J. van Vliet;Christina M. J. E. Vandenbroucke-Grauls.
Journal of Immunology (2003)

537 Citations

Lactoferrin is a lipid A-binding protein.

B. J. Appelmelk;Yun-Qing An;M. Geerts;B. G. Thijs.
Infection and Immunity (1994)

469 Citations

The dendritic cell-specific C-type lectin DC-SIGN is a receptor for Schistosoma mansoni egg antigens and recognizes the glycan antigen Lewis x.

Irma van Die;Sandra J van Vliet;A Kwame Nyame;Richard D Cummings.
Glycobiology (2003)

419 Citations

Helicobacter pylori modulates the T helper cell 1/T helper cell 2 balance through phase-variable interaction between lipopolysaccharide and DC-SIGN.

Mathijs P. Bergman;Anneke Engering;Hermelijn H. Smits;Sandra J. van Vliet.
Journal of Experimental Medicine (2004)

403 Citations

Antigenic mimicry between Helicobacter pylori and gastric mucosa in the pathogenesis of body atrophic gastritis

R Negrini;A Savio;C Poiesi;BJ Appelmelk.
Gastroenterology (1996)

393 Citations

Molecular mimicry of host structures by bacterial lipopolysaccharides and its contribution to disease

Anthony P. Moran;Martina M. Prendergast;Ben J. Appelmelk.
Fems Immunology and Medical Microbiology (1996)

392 Citations

The gastric H+,K+-ATPase is a major autoantigen in chronic Helicobacter pylori gastritis with body mucosa atrophy ☆ ☆☆ ★ ★★

Dirk Claeys;Gerhard Faller;Ben J. Appelmelk;Riccardo Negrini.
Gastroenterology (1998)

324 Citations

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