His primary areas of investigation include Cell biology, Protein kinase A, Molecular biology, Apoptosis and Cancer research. The Protein kinase A study combines topics in areas such as Protein phosphatase 2 and Signal transduction, Kinase activity. The concepts of his Kinase activity study are interwoven with issues in PRKCQ and Phorbol, Protein kinase C.
His research in Molecular biology intersects with topics in p38 mitogen-activated protein kinases, MAPK/ERK pathway, MAPK phosphatase, Tyrosine phosphorylation and GRB2. His studies in Cancer research integrate themes in fields like Muscle hypertrophy, Protein kinase B, P70-S6 Kinase 1 and Angiotensin receptor. His research in Proteasome inhibitor focuses on subjects like Cancer cell, which are connected to Transfection.
His main research concerns Cancer research, Kinase, Cell biology, Protein kinase A and Molecular biology. His Cancer research research incorporates elements of Cancer cell, Cancer, Prostate cancer, PIM1 and Signal transduction. His Kinase study combines topics from a wide range of disciplines, such as mTORC1, PI3K/AKT/mTOR pathway, T cell, Immunology and Phosphorylation.
His research integrates issues of Cyclin-dependent kinase 1, Interleukin 3 and Cell growth in his study of Cell biology. His research on Protein kinase A frequently connects to adjacent areas such as Kinase activity. His Molecular biology study integrates concerns from other disciplines, such as Phorbol, Transcription, Cellular differentiation and c-jun.
Andrew S. Kraft focuses on Cancer research, Kinase, PIM1, Cell biology and Prostate cancer. His biological study spans a wide range of topics, including Cancer cell, Cancer, T cell, Immunology and Signal transduction. His Kinase research is mostly focused on the topic Protein kinase A.
The various areas that he examines in his Protein kinase A study include Mitochondrion, Endocrinology, Knockout mouse and Senescence. His PIM1 research is multidisciplinary, incorporating perspectives in Haematopoiesis, Stem cell, Ponatinib, Tyrosine-kinase inhibitor and Leukemia. His work on Effector as part of general Cell biology study is frequently linked to IRS1, therefore connecting diverse disciplines of science.
Kinase, Cancer research, Cell biology, Internal medicine and Cell growth are his primary areas of study. In his research on the topic of Kinase, Endocrinology, Knockout mouse and Mitochondrion is strongly related with Cell aging. Andrew S. Kraft interconnects Protein kinase A, mTORC1, PIM1 and Signal transduction, Kinase activity in the investigation of issues within Cancer research.
As part of his studies on Cell biology, Andrew S. Kraft often connects relevant areas like T cell. His work deals with themes such as Gastroenterology, Sarcoma and Oncology, which intersect with Internal medicine. Andrew S. Kraft has researched Reactive oxygen species in several fields, including Tumor microenvironment, Cancer cell and Molecular biology.
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Phorbol esters increase the amount of Ca2+, phospholipid-dependent protein kinase associated with plasma membrane.
Andrew S. Kraft;Wayne B. Anderson.
Conditional Expression of the Mitogen-activated Protein Kinase (MAPK) Phosphatase MKP-1 Preferentially Inhibits p38 MAPK and Stress-activated Protein Kinase in U937 Cells
Christopher C. Franklin;Andrew S. Kraft.
Journal of Biological Chemistry (1997)
Phase II Study of Sorafenib in Patients With Metastatic or Recurrent Sarcomas
Robert G. Maki;David R. D'Adamo;Mary L. Keohan;Michael Saulle.
Journal of Clinical Oncology (2009)
Elevated Akt Activity Protects the Prostate Cancer Cell Line LNCaP from TRAIL-induced Apoptosis
Alexandre Nesterov;Xiaojun Lu;Michael Johnson;Gary J. Miller.
Journal of Biological Chemistry (2001)
Bryostatin, an activator of the calcium phospholipid-dependent protein kinase, blocks phorbol ester-induced differentiation of human promyelocytic leukemia cells HL-60
Andrew S. Kraft;Jeffrey B. Smith;Roger L. Berkow.
Proceedings of the National Academy of Sciences of the United States of America (1986)
The PIM-2 Kinase Phosphorylates BAD on Serine 112 and Reverses BAD-induced Cell Death
Bin Yan;Marina Zemskova;Sheldon Holder;Vernon Chin.
Journal of Biological Chemistry (2003)
The Role of the Transcription Factor Sp1 in Regulating the Expression of the WAF1/CIP1 Gene in U937 Leukemic Cells
Joseph R. Biggs;Jeffery E. Kudlow;Andrew S. Kraft.
Journal of Biological Chemistry (1996)
The 2014 Surgeon General's report: “The Health Consequences of Smoking–50 Years of Progress”: A paradigm shift in cancer care
Graham W. Warren;Anthony J. Alberg;Andrew S. Kraft;K. Michael Cummings.
The proteasome inhibitor PS-341 overcomes TRAIL resistance in Bax and caspase 9-negative or Bcl-xL overexpressing cells.
Thomas R. Johnson;Kimberley Stone;Malti Nikrad;Tammie Yeh.
Regions of the JAK2 tyrosine kinase required for coupling to the growth hormone receptor
Stuart J. Frank;Stuart J. Frank;Woelsung Yi;Yanming Zhao;Jeffrey F. Goldsmith.
Journal of Biological Chemistry (1995)
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